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A homeotic gene cluster patterns the anteroposterior body axis of C. elegans.
Wang BB, Müller-Immergluck MM, Austin J, Robinson NT, Chisholm A, Kenyon C. Wang BB, et al. Cell. 1993 Jul 16;74(1):29-42. doi: 10.1016/0092-8674(93)90292-x. Cell. 1993. PMID: 8101474
The nematode C. elegans also contains a small cluster of HOM-C genes, one of which has been shown to specify positional identity. ...Each C. elegans HOM-C gene is responsible for a distinct body region; however, where their domains overlap, two HOM-C genes can act t …
The nematode C. elegans also contains a small cluster of HOM-C genes, one of which has been shown to specify positional identity. ... …
Control of cell fate in the tail region of C. elegans by the gene egl-5.
Chisholm A. Chisholm A. Development. 1991 Apr;111(4):921-32. Development. 1991. PMID: 1879361
The tail region of C. elegans contains a number of blast cell and neuron types that either are found only in the tail, or are different from more anterior homologues. ...By genetic mapping, egl-5 lies very close to mab-5, a gene with an analogous role in the immedia …
The tail region of C. elegans contains a number of blast cell and neuron types that either are found only in the tail, or are differe …
Patterning of the Caenorhabditis elegans head region by the Pax-6 family member vab-3.
Chisholm AD, Horvitz HR. Chisholm AD, et al. Nature. 1995 Sep 7;377(6544):52-5. doi: 10.1038/377052a0. Nature. 1995. PMID: 7659159
Mutations in the human PAX6 gene are found in patients with a variety of eye disorders, including aniridia and Peters' anomaly, and mutations in the Drosophila Pax-6 homologue cause the eyeless phenotype. ...Here we show that vab-3 is a member of the paired-domain-c …
Mutations in the human PAX6 gene are found in patients with a variety of eye disorders, including aniridia and Peters' anomaly, and m …
Control of cell fates in the central body region of C. elegans by the homeobox gene lin-39.
Clark SG, Chisholm AD, Horvitz HR. Clark SG, et al. Cell. 1993 Jul 16;74(1):43-55. doi: 10.1016/0092-8674(93)90293-y. Cell. 1993. PMID: 8101475
In lin-39 mutants, mid-body cells express fates characteristic of more anterior or posterior homologs, and the migration of a neuroblast through the mid-body is defective. lin-39 acts cell autonomously in these mid-body cells and in the migrating neuroblast. lin-39 encodes …
In lin-39 mutants, mid-body cells express fates characteristic of more anterior or posterior homologs, and the migration of a neurobl …
The VAB-1 Eph receptor tyrosine kinase functions in neural and epithelial morphogenesis in C. elegans.
George SE, Simokat K, Hardin J, Chisholm AD. George SE, et al. Cell. 1998 Mar 6;92(5):633-43. doi: 10.1016/s0092-8674(00)81131-9. Cell. 1998. PMID: 9506518
We show that vab-1 encodes a receptor tyrosine kinase of the Eph family. Disruption of the kinase domain of VAB-1 causes weak mutant phenotypes, indicating that VAB-1 may have both kinase-dependent and kinase-independent activities. ...
We show that vab-1 encodes a receptor tyrosine kinase of the Eph family. Disruption of the kinase domain of VAB-1 causes weak mutant …
The inositol trisphosphate receptor regulates a 50-second behavioral rhythm in C. elegans.
Dal Santo P, Logan MA, Chisholm AD, Jorgensen EM. Dal Santo P, et al. Cell. 1999 Sep 17;98(6):757-67. doi: 10.1016/s0092-8674(00)81510-x. Cell. 1999. PMID: 10499793
Thus, the IP3 receptor is an essential component of the timekeeper for this cycle and represents a novel mechanism for the control of behavioral rhythms....
Thus, the IP3 receptor is an essential component of the timekeeper for this cycle and represents a novel mechanism for the control of …
The ephrin VAB-2/EFN-1 functions in neuronal signaling to regulate epidermal morphogenesis in C. elegans.
Chin-Sang ID, George SE, Ding M, Moseley SL, Lynch AS, Chisholm AD. Chin-Sang ID, et al. Cell. 1999 Dec 23;99(7):781-90. doi: 10.1016/s0092-8674(00)81675-x. Cell. 1999. PMID: 10619431
We show that the ephrin VAB-2 (also known as EFN-1) is a ligand for VAB-1 and can function in neurons to regulate epidermal morphogenesis. In the absence of VAB-1 signaling, ephrin-expressing neurons are disorganized. vab-2/efn-1 mutations synergize with vab-1 kinase allel …
We show that the ephrin VAB-2 (also known as EFN-1) is a ligand for VAB-1 and can function in neurons to regulate epidermal morphogen …
Form of the worm: genetics of epidermal morphogenesis in C. elegans.
Chin-Sang ID, Chisholm AD. Chin-Sang ID, et al. Trends Genet. 2000 Dec;16(12):544-51. doi: 10.1016/s0168-9525(00)02143-0. Trends Genet. 2000. PMID: 11102704 Review.
The divergent C. elegans ephrin EFN-4 functions inembryonic morphogenesis in a pathway independent of the VAB-1 Eph receptor.
Chin-Sang ID, Moseley SL, Ding M, Harrington RJ, George SE, Chisholm AD. Chin-Sang ID, et al. Development. 2002 Dec;129(23):5499-510. doi: 10.1242/dev.00122. Development. 2002. PMID: 12403719
The C. elegans genome encodes a single Eph receptor tyrosine kinase, VAB-1, which functions in neurons to control epidermal morphogenesis. ...Mutations in the LAR-like receptor tyrosine phosphatase PTP-3 and in the Semaphorin-2A homolog MAB-20 disrupt embryonic neural morp …
The C. elegans genome encodes a single Eph receptor tyrosine kinase, VAB-1, which functions in neurons to control epidermal morphogen …
The Caenorhabditis elegans vab-10 spectraplakin isoforms protect the epidermis against internal and external forces.
Bosher JM, Hahn BS, Legouis R, Sookhareea S, Weimer RM, Gansmuller A, Chisholm AD, Rose AM, Bessereau JL, Labouesse M. Bosher JM, et al. J Cell Biol. 2003 May 26;161(4):757-68. doi: 10.1083/jcb.200302151. Epub 2003 May 19. J Cell Biol. 2003. PMID: 12756232 Free PMC article.
Here, we report the identification of new mutations in a gene known as vab-10, which lead to severe morphogenesis defects, and show that vab-10 corresponds to the C. elegans spectraplakin locus. ...
Here, we report the identification of new mutations in a gene known as vab-10, which lead to severe morphogenesis defects, and show t …
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