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A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde manner.
Rassadi S, Krishnaswamy A, Pié B, McConnell R, Jacob MH, Cooper E. Rassadi S, et al. Among authors: cooper e. J Neurosci. 2005 Sep 14;25(37):8555-66. doi: 10.1523/JNEUROSCI.1983-05.2005. J Neurosci. 2005. PMID: 16162937 Free PMC article.
Removing 4E-BP Enables Synapses to Refine without Postsynaptic Activity.
Chong Y, Saviuk N, Pie B, Basisty N, Quinn RK, Schilling B, Sonenberg N, Cooper E, Haghighi AP. Chong Y, et al. Among authors: cooper e. Cell Rep. 2018 Apr 3;23(1):11-22. doi: 10.1016/j.celrep.2018.03.040. Cell Rep. 2018. PMID: 29617653 Free PMC article.
1,733 results