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A novel P/V/C gene in a new member of the Paramyxoviridae family, which causes lethal infection in humans, horses, and other animals.
Wang LF, Michalski WP, Yu M, Pritchard LI, Crameri G, Shiell B, Eaton BT. Wang LF, et al. Among authors: Crameri G. J Virol. 1998 Feb;72(2):1482-90. J Virol. 1998. PMID: 9445051 Free PMC article.
The P and C proteins are larger and smaller, respectively, than are cognate proteins in members of the subfamily, and the V protein is made as a result of a single G insertion during transcription. ...
The P and C proteins are larger and smaller, respectively, than are cognate proteins in members of the subfamily, and the V protein is made …
RNA synthesis during infection by Hendra virus: an examination by quantitative real-time PCR of RNA accumulation, the effect of ribavirin and the attenuation of transcription.
Wright PJ, Crameri G, Eaton BT. Wright PJ, et al. Among authors: Crameri G. Arch Virol. 2005 Mar;150(3):521-32. doi: 10.1007/s00705-004-0417-5. Epub 2004 Oct 29. Arch Virol. 2005. PMID: 15526144
It was also used to determine the points of attenuation during transcription of the six viral genes N, P, M, F, G and L by targeting amplicons located towards the 3' end of each gene. ...There was significant attenuation at the M-F and G-L junctions, more closely resembling Sendai virus (genus Respirovirus) than measles virus (genus Morbillivirus)....
It was also used to determine the points of attenuation during transcription of the six viral genes N, P, M, F, G and L by targeting …
Receptor binding, fusion inhibition, and induction of cross-reactive neutralizing antibodies by a soluble G glycoprotein of Hendra virus.
Bossart KN, Crameri G, Dimitrov AS, Mungall BA, Feng YR, Patch JR, Choudhary A, Wang LF, Eaton BT, Broder CC. Bossart KN, et al. Among authors: Crameri G. J Virol. 2005 Jun;79(11):6690-702. doi: 10.1128/JVI.79.11.6690-6702.2005. J Virol. 2005. PMID: 15890907 Free PMC article.
These viruses infect cells by a pH-independent membrane fusion event mediated by their attachment (G) and fusion (F) glycoproteins. ...To further characterize the G glycoprotein and its unknown receptor, soluble forms of HeV G (sG) were constructed by replacing its cytoplasmic tail and transmembrane domains with an immunoglobulin kappa leader sequence coupled to either an S-peptide tag (sG(S-tag)) or myc-epitope tag (sG(myc-tag)) to facilitate purification and detection. ...
These viruses infect cells by a pH-independent membrane fusion event mediated by their attachment (G) and fusion (F) glycoproteins. . …
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