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Ontogenesis of dopamine receptor binding in the corpus striatum of the rat.
Pardo JV, Creese I, Burt DR, Snyder SH. Pardo JV, et al. Brain Res. 1977 Apr 15;125(2):376-82. doi: 10.1016/0006-8993(77)90633-3. Brain Res. 1977. PMID: 851885 No abstract available.
Amphetamine response in rat after dopamine neurone destruction.
Creese I, Iversen SD. Creese I, et al. Nat New Biol. 1972 Aug 23;238(86):247-8. doi: 10.1038/newbio238247a0. Nat New Biol. 1972. PMID: 4404010 No abstract available.
Systemic 3-nitropropionic acid: long-term effects on locomotor behavior.
Koutouzis TK, Borlongan CV, Scorcia T, Creese I, Cahill DW, Freeman TB, Sanberg PR. Koutouzis TK, et al. Brain Res. 1994 May 23;646(2):242-6. doi: 10.1016/0006-8993(94)90085-x. Brain Res. 1994. PMID: 8069670
Restricted eating with weight loss selectively decreases extracellular dopamine in the nucleus accumbens and alters dopamine response to amphetamine, morphine, and food intake.
Pothos EN, Creese I, Hoebel BG. Pothos EN, et al. J Neurosci. 1995 Oct;15(10):6640-50. doi: 10.1523/JNEUROSCI.15-10-06640.1995. J Neurosci. 1995. PMID: 7472425 Free PMC article.
In underweight rats, systemic amphetamine (1.5 mg/kg i.p.) transiently restored extracellular DA, but only to basal normal levels. Morphine (20 mg/kg i.p.) or a meal also increased DA, but the percent increase was significantly smaller in underweight than normal wei …
In underweight rats, systemic amphetamine (1.5 mg/kg i.p.) transiently restored extracellular DA, but only to basal normal levels. Mo …
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