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Doxorubicin induces cardiotoxicity through upregulation of death receptors mediated apoptosis in cardiomyocytes.
Zhao L, Zhang B. Zhao L, et al. Sci Rep. 2017 Mar 16;7:44735. doi: 10.1038/srep44735. Sci Rep. 2017. PMID: 28300219 Free PMC article.
The resulting iPS-CMs cells underwent spontaneous apoptosis which was further enhanced by physiologically relevant death ligands including TNF-related apoptosis inducing ligand (TRAIL). Furthermore, TRAIL potentiated doxorubicin-induced d …
The resulting iPS-CMs cells underwent spontaneous apoptosis which was further enhanced by physiologically relevant death ligan …
Acylated Ghrelin Protects the Hearts of Rats from Doxorubicin-Induced Fas/FasL Apoptosis by Stimulating SERCA2a Mediated by Activation of PKA and Akt.
Shati AA, Dallak M. Shati AA, et al. Cardiovasc Toxicol. 2019 Dec;19(6):529-547. doi: 10.1007/s12012-019-09527-8. Cardiovasc Toxicol. 2019. PMID: 31093930
This study investigated if the cardioprotective effect of acylated ghrelin (AG) against doxorubicin (DOX)-induced cardiac toxicity in rats involves inhibition of Fas/FasL-mediated cell death. It also investigated if such an effect is mediated
This study investigated if the cardioprotective effect of acylated ghrelin (AG) against doxorubicin (DOX)-induced cardiac
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