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A novel tool for the assessment of pain: validation in low back pain.
Scholz J, Mannion RJ, Hord DE, Griffin RS, Rawal B, Zheng H, Scoffings D, Phillips A, Guo J, Laing RJ, Abdi S, Decosterd I, Woolf CJ. Scholz J, et al. PLoS Med. 2009 Apr 7;6(4):e1000047. doi: 10.1371/journal.pmed.1000047. Epub 2009 Apr 7. PLoS Med. 2009. PMID: 19360087 Free PMC article.
Assessment and analysis of mechanical allodynia-like behavior induced by spared nerve injury (SNI) in the mouse.
Bourquin AF, Süveges M, Pertin M, Gilliard N, Sardy S, Davison AC, Spahn DR, Decosterd I. Bourquin AF, et al. Pain. 2006 May;122(1-2):14.e1-14. doi: 10.1016/j.pain.2005.10.036. Epub 2006 Mar 20. Pain. 2006. PMID: 16542774
Blocking caspase activity prevents transsynaptic neuronal apoptosis and the loss of inhibition in lamina II of the dorsal horn after peripheral nerve injury.
Scholz J, Broom DC, Youn DH, Mills CD, Kohno T, Suter MR, Moore KA, Decosterd I, Coggeshall RE, Woolf CJ. Scholz J, et al. J Neurosci. 2005 Aug 10;25(32):7317-23. doi: 10.1523/JNEUROSCI.1526-05.2005. J Neurosci. 2005. PMID: 16093381 Free PMC article.
We show that transsynaptic apoptosis is induced in the superficial dorsal horn (laminas I-III) of the spinal cord by three distinct partial peripheral nerve lesions: spared nerve injury, chronic constriction, and spinal nerve ligation. ...
We show that transsynaptic apoptosis is induced in the superficial dorsal horn (laminas I-III) of the spinal cord by three distinct p …
Development of neuropathic pain in the rat spared nerve injury model is not prevented by a peripheral nerve block.
Suter MR, Papaloïzos M, Berde CB, Woolf CJ, Gilliard N, Spahn DR, Decosterd I. Suter MR, et al. Anesthesiology. 2003 Dec;99(6):1402-8. doi: 10.1097/00000542-200312000-00025. Anesthesiology. 2003. PMID: 14639156
Development of neuropathic pain behavior was assessed for 4 weeks by withdrawal responses to stimulation (i.e., von Frey filaments, acetone, pinprick, radiant heat) in bupivacaine microspheres-treated animals (n = 12) and in controls (n = 11). ...
Development of neuropathic pain behavior was assessed for 4 weeks by withdrawal responses to stimulation (i.e., von Frey filaments, a …
Selective inhibition of JNK with a peptide inhibitor attenuates pain hypersensitivity and tumor growth in a mouse skin cancer pain model.
Gao YJ, Cheng JK, Zeng Q, Xu ZZ, Decosterd I, Xu X, Ji RR. Gao YJ, et al. Exp Neurol. 2009 Sep;219(1):146-55. doi: 10.1016/j.expneurol.2009.05.006. Epub 2009 May 13. Exp Neurol. 2009. PMID: 19445931 Free PMC article.
Repeated systemic injections of D-JNKI-1 (6 mg/kg, i.p.), a selective and cell-permeable peptide inhibitor of JNK, produced an accumulative inhibition of mechanical allodynia and heat hyperalgesia. ...
Repeated systemic injections of D-JNKI-1 (6 mg/kg, i.p.), a selective and cell-permeable peptide inhibitor of JNK, produced an accumu …
The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition.
Gao YJ, Xu ZZ, Liu YC, Wen YR, Decosterd I, Ji RR. Gao YJ, et al. Pain. 2010 Feb;148(2):309-19. doi: 10.1016/j.pain.2009.11.017. Pain. 2010. PMID: 20022176 Free PMC article.
Nerve conduction blockade in the sciatic nerve prevents but does not reverse the activation of p38 mitogen-activated protein kinase in spinal microglia in the rat spared nerve injury model.
Wen YR, Suter MR, Kawasaki Y, Huang J, Pertin M, Kohno T, Berde CB, Decosterd I, Ji RR. Wen YR, et al. Anesthesiology. 2007 Aug;107(2):312-21. doi: 10.1097/01.anes.0000270759.11086.e7. Anesthesiology. 2007. PMID: 17667577
A peptide c-Jun N-terminal kinase (JNK) inhibitor blocks mechanical allodynia after spinal nerve ligation: respective roles of JNK activation in primary sensory neurons and spinal astrocytes for neuropathic pain development and maintenance.
Zhuang ZY, Wen YR, Zhang DR, Borsello T, Bonny C, Strichartz GR, Decosterd I, Ji RR. Zhuang ZY, et al. J Neurosci. 2006 Mar 29;26(13):3551-60. doi: 10.1523/JNEUROSCI.5290-05.2006. J Neurosci. 2006. PMID: 16571763 Free PMC article.
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