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1975 1
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Page 1
Butyrylcholinesterase deficiency.
Delacour H, Dedome E, Courcelle S, Hary B, Ceppa F. Delacour H, et al. Ann Biol Clin (Paris). 2016 Jun 1;74(3):279-85. doi: 10.1684/abc.2016.1141. Ann Biol Clin (Paris). 2016. PMID: 27237801 Free article. Review. English.
Butyrylcholinesterase (EC 3.1.1.8; BChE) is a sister enzyme of acetylcholinesterase. ...Although many acquired conditions may affect BChE activity, BChE deficiency is mainly due to mutations in the BCHE gene (OMIM 177400). ...
Butyrylcholinesterase (EC 3.1.1.8; BChE) is a sister enzyme of acetylcholinesterase. ...Although many acquired conditions may affect
Status of acetylcholinesterase and butyrylcholinesterase in Alzheimer's disease and type 2 diabetes mellitus.
Mushtaq G, Greig NH, Khan JA, Kamal MA. Mushtaq G, et al. CNS Neurol Disord Drug Targets. 2014;13(8):1432-9. doi: 10.2174/1871527313666141023141545. CNS Neurol Disord Drug Targets. 2014. PMID: 25345511 Free PMC article. Review.
Both Alzheimer's disease (AD) and Type 2 diabetes mellitus (T2DM) share the presence of systemic and neuro-inflammation, enhanced production and accumulation of beta -amyloid peptide and abnormal levels of the enzymes acetylcholinesterase (AChE) and butyrylcholinesterase ( …
Both Alzheimer's disease (AD) and Type 2 diabetes mellitus (T2DM) share the presence of systemic and neuro-inflammation, enhanced production …
Pseudocholinesterase deficiency: a comprehensive review of genetic, acquired, and drug influences.
Soliday FK, Conley YP, Henker R. Soliday FK, et al. AANA J. 2010 Aug;78(4):313-20. AANA J. 2010. PMID: 20879632 Review.
Pseudocholinesterase deficiency is an inherited or acquired condition in which the metabolism of succinylcholine, mivacurium, or ester local anesthetics is potentially impaired. ...
Pseudocholinesterase deficiency is an inherited or acquired condition in which the metabolism of succinylcholine, mivacurium, or este …
A Current Perspective on the Inhibition of Cholinesterase by Natural and Synthetic Inhibitors.
Shah AA, Dar TA, Dar PA, Ganie SA, Kamal MA. Shah AA, et al. Curr Drug Metab. 2017;18(2):96-111. doi: 10.2174/1389200218666161123122734. Curr Drug Metab. 2017. PMID: 27890007 Review.
Majority of the observed cognitive and behavioral changes in Alzheimer's disease are postulated to be due to the deficiencies in cholinergic pathways of the brain. Enhancement of cholinergic transmission may thus stimulate the cholinergic receptors or prolong the availabil …
Majority of the observed cognitive and behavioral changes in Alzheimer's disease are postulated to be due to the deficiencies in chol …
The origin of the molecular diversity and functional anchoring of cholinesterases.
Massoulié J. Massoulié J. Neurosignals. 2002 May-Jun;11(3):130-43. doi: 10.1159/000065054. Neurosignals. 2002. PMID: 12138250 Review.
Vertebrates possess two cholinesterases, acetylcholinesterase (AChE) and butyrylcholinesterase (BChE) which both hydrolyze acetylcholine, but differ in their specificity towards other substrates, and in their sensitivity to inhibitors. ...
Vertebrates possess two cholinesterases, acetylcholinesterase (AChE) and butyrylcholinesterase (BChE) which both hydrolyze acetylchol …
Rivastigmine in the treatment of Alzheimer's disease: an update.
Onor ML, Trevisiol M, Aguglia E. Onor ML, et al. Clin Interv Aging. 2007;2(1):17-32. doi: 10.2147/ciia.2007.2.1.17. Clin Interv Aging. 2007. PMID: 18044073 Free PMC article. Review.
The condition is an age-related neurodegenerative disorder characterized by multiple cognitive deficiencies, including loss of memory, judgment, and comprehension. ...Rivastigmine (Exelon, Novartis Basel-Switzerland) is a slowly reversible inhibitor of acetylcholinesterase …
The condition is an age-related neurodegenerative disorder characterized by multiple cognitive deficiencies, including loss of memory …
Serine hydrolase targets of organophosphorus toxicants.
Casida JE, Quistad GB. Casida JE, et al. Chem Biol Interact. 2005 Dec 15;157-158:277-83. doi: 10.1016/j.cbi.2005.10.036. Epub 2005 Oct 21. Chem Biol Interact. 2005. PMID: 16243304 Review.
The toxicological relevance of known secondary OP targets is established mainly from observations with humans (butyrylcholinesterase and neuropathy target esterase-lysophospholipase) and studies with mice (cannabinoid CB1 receptor, carboxylesterase, lysophospholipase and p …
The toxicological relevance of known secondary OP targets is established mainly from observations with humans (butyrylcholinesterase
Pharmacogenetic screening and therapeutic drugs.
Steimer W, Potter JM. Steimer W, et al. Clin Chim Acta. 2002 Jan;315(1-2):137-55. doi: 10.1016/s0009-8981(01)00713-6. Clin Chim Acta. 2002. PMID: 11728416 Review.
27 results