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Genetic analysis of glutamate receptors in Drosophila reveals a retrograde signal regulating presynaptic transmitter release.
Petersen SA, Fetter RD, Noordermeer JN, Goodman CS, DiAntonio A. Petersen SA, et al. Among authors: diantonio a. Neuron. 1997 Dec;19(6):1237-48. doi: 10.1016/s0896-6273(00)80415-8. Neuron. 1997. PMID: 9427247
We cloned the gene encoding a second muscle-specific glutamate receptor, DGluRIIB, which is closely related to the previously identified DGluRIIA and located adjacent to it in the genome. ...Thus, a decrease in postsynaptic receptors leads to an increase in presynap …
We cloned the gene encoding a second muscle-specific glutamate receptor, DGluRIIB, which is closely related to the previously identif …
Genetic interactions with integrins during wing morphogenesis in Drosophila.
Wessendorf LH, Wehrli M, DiAntonio A, Wilcox M. Wessendorf LH, et al. Among authors: diantonio a. Cold Spring Harb Symp Quant Biol. 1992;57:241-8. doi: 10.1101/sqb.1992.057.01.029. Cold Spring Harb Symp Quant Biol. 1992. PMID: 1339662 Review. No abstract available.
The effect on synaptic physiology of synaptotagmin mutations in Drosophila.
DiAntonio A, Schwarz TL. DiAntonio A, et al. Neuron. 1994 Apr;12(4):909-20. doi: 10.1016/0896-6273(94)90342-5. Neuron. 1994. PMID: 7909234
Reduced levels of synaptotagmin resulted in a substantial alteration in synaptic function in the eye and at larval neuromuscular junctions. ...These data are discussed in the context of models in which synaptotagmin's function includes a role in vesicle docking....
Reduced levels of synaptotagmin resulted in a substantial alteration in synaptic function in the eye and at larval neuromuscular junc …
Synaptic transmission persists in synaptotagmin mutants of Drosophila.
DiAntonio A, Parfitt KD, Schwarz TL. DiAntonio A, et al. Cell. 1993 Jul 2;73(7):1281-90. doi: 10.1016/0092-8674(93)90356-u. Cell. 1993. PMID: 8100740
It has been postulated to dock vesicles to their release sites, to act as the Ca2+ sensor for the release process, and to be a fusion protein during exocytosis. ...The lethality of synaptotagmin in late first instar larvae is probably due to a perturbation of transm …
It has been postulated to dock vesicles to their release sites, to act as the Ca2+ sensor for the release process, and to be a fusion …
Identification and characterization of Drosophila genes for synaptic vesicle proteins.
DiAntonio A, Burgess RW, Chin AC, Deitcher DL, Scheller RH, Schwarz TL. DiAntonio A, et al. J Neurosci. 1993 Nov;13(11):4924-35. doi: 10.1523/JNEUROSCI.13-11-04924.1993. J Neurosci. 1993. PMID: 8229205 Free PMC article.
This investigation describes Drosophila homologs of vamp, synaptotagmin, and rab3 that are expressed in a pattern consistent with a function in Drosophila neurotransmission. ...Because of their distribution in the nervous system and because n-syb, synaptotagmin, and …
This investigation describes Drosophila homologs of vamp, synaptotagmin, and rab3 that are expressed in a pattern consistent with …
Cloning and characterization of alpha PS1, a novel Drosophila melanogaster integrin.
Wehrli M, DiAntonio A, Fearnley IM, Smith RJ, Wilcox M. Wehrli M, et al. Among authors: diantonio a. Mech Dev. 1993 Sep;43(1):21-36. doi: 10.1016/0925-4773(93)90020-x. Mech Dev. 1993. PMID: 8240969
We find several positions and phases of introns conserved which, supported by conservation also in the amino acid sequence, indicates that they all derive from a common ancestral gene....
We find several positions and phases of introns conserved which, supported by conservation also in the amino acid sequence, indicates that t …
Drosophila genetics and the functions of synaptic proteins.
Parfitt K, Reist N, Li J, Burgess R, Deitcher D, DiAntonio A, Schwarz TL. Parfitt K, et al. Among authors: diantonio a. Cold Spring Harb Symp Quant Biol. 1995;60:371-7. doi: 10.1101/sqb.1995.060.01.041. Cold Spring Harb Symp Quant Biol. 1995. PMID: 8824410 Review. No abstract available.
Postsynaptic PKA controls quantal size and reveals a retrograde signal that regulates presynaptic transmitter release in Drosophila.
Davis GW, DiAntonio A, Petersen SA, Goodman CS. Davis GW, et al. Among authors: diantonio a. Neuron. 1998 Feb;20(2):305-15. doi: 10.1016/s0896-6273(00)80458-4. Neuron. 1998. PMID: 9491991
Two distinct mechanisms regulate synaptic efficacy at the Drosophila neuromuscular junction (NMJ): a PKA-dependent modulation of quantal size and a retrograde regulation of presynaptic release. Postsynaptic expression of a constitutively active PKA catalytic …
Two distinct mechanisms regulate synaptic efficacy at the Drosophila neuromuscular junction (NMJ): a PKA-dependent modulation of quan …
Glutamate receptor expression regulates quantal size and quantal content at the Drosophila neuromuscular junction.
DiAntonio A, Petersen SA, Heckmann M, Goodman CS. DiAntonio A, et al. J Neurosci. 1999 Apr 15;19(8):3023-32. doi: 10.1523/JNEUROSCI.19-08-03023.1999. J Neurosci. 1999. PMID: 10191319 Free PMC article.
At the Drosophila glutamatergic neuromuscular junction, the postsynaptic cell can regulate synaptic strength by both changing its sensitivity to neurotransmitter and generating a retrograde signal that regulates presynaptic transmitter release. To investigate the molecular …
At the Drosophila glutamatergic neuromuscular junction, the postsynaptic cell can regulate synaptic strength by both changing its sensitivit …
Learning something ORIGINal at the Drosophila neuromuscular junction.
DiAntonio A. DiAntonio A. Neuron. 1999 May;23(1):1-2. doi: 10.1016/s0896-6273(00)80743-6. Neuron. 1999. PMID: 10402184 Review. No abstract available.
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