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Cohesin gene mutations in tumorigenesis: from discovery to clinical significance.
Solomon DA, Kim JS, Waldman T. Solomon DA, et al. BMB Rep. 2014 Jun;47(6):299-310. doi: 10.5483/bmbrep.2014.47.6.092. BMB Rep. 2014. PMID: 24856830 Free PMC article. Review.
Recent cancer genomics analyses have discovered a high frequency of somatic mutations in the genes encoding the core cohesin subunits as well as cohesin regulatory factors (e.g. NIPBL, PDS5B, ESPL1) in a select subset of human tumors including glioblastoma, Ewing sarcoma, …
Recent cancer genomics analyses have discovered a high frequency of somatic mutations in the genes encoding the core cohesin subunits as wel …
A NUP98-positive acute myeloid leukemia with a t(11;12)(p15;q13) without HOXC cluster gene involvement.
La Starza R, Brandimarte L, Pierini V, Nofrini V, Gorello P, Crescenzi B, Berchicci L, Matteucci C, Romoli S, Beacci D, Rosati R, Martelli MF, Mecucci C. La Starza R, et al. Cancer Genet Cytogenet. 2009 Sep;193(2):109-11. doi: 10.1016/j.cancergencyto.2009.04.015. Cancer Genet Cytogenet. 2009. PMID: 19665072
Our fluorescence in situ hybridization (FISH) showed that putative candidate partners mapping 600 kilobases centromeric to HOXC were RARG (retinoic acid receptor gamma), MFSD5 (major facilitator superfamily domain containing 5), and ESPL1 (extra spindle pole bodies homolog …
Our fluorescence in situ hybridization (FISH) showed that putative candidate partners mapping 600 kilobases centromeric to HOXC were RARG (r …