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2012 1
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Systemic elevation of PTEN induces a tumor-suppressive metabolic state.
Garcia-Cao I, Song MS, Hobbs RM, Laurent G, Giorgi C, de Boer VC, Anastasiou D, Ito K, Sasaki AT, Rameh L, Carracedo A, Vander Heiden MG, Cantley LC, Pinton P, Haigis MC, Pandolfi PP. Garcia-Cao I, et al. Cell. 2012 Mar 30;149(1):49-62. doi: 10.1016/j.cell.2012.02.030. Epub 2012 Mar 6. Cell. 2012. PMID: 22401813 Free PMC article.
Decremental loss of PTEN results in cancer susceptibility and tumor progression. PTEN elevation might therefore be an attractive option for cancer prevention and therapy. ...Unexpectedly, PTEN elevation at the organism level results in he …
Decremental loss of PTEN results in cancer susceptibility and tumor progression. PTEN elevation might therefore …
SENP1 regulates PTEN stability to dictate prostate cancer development.
Bawa-Khalfe T, Yang FM, Ritho J, Lin HK, Cheng J, Yeh ET. Bawa-Khalfe T, et al. Oncotarget. 2017 Mar 14;8(11):17651-17664. doi: 10.18632/oncotarget.13283. Oncotarget. 2017. PMID: 27852060 Free PMC article.
However, mice expressing an androgen-responsive promoter driven SENP1-transgene (SENP1-Tg) develop high-grade prostatic intraepithelial neoplasia, but not carcinoma. We now show that tumor suppressive PTEN signaling is induced in SENP1-Tg to enhance pr …
However, mice expressing an androgen-responsive promoter driven SENP1-transgene (SENP1-Tg) develop high-grade prostatic intraepithelial neop …
MMP7 interacts with ARF in nucleus to potentiate tumor microenvironments for prostate cancer progression in vivo.
Xie Y, Lu W, Liu S, Yang Q, Goodwin JS, Sathyanarayana SA, Pratap S, Chen Z. Xie Y, et al. Oncotarget. 2016 Jul 26;7(30):47609-47619. doi: 10.18632/oncotarget.10251. Oncotarget. 2016. PMID: 27356744 Free PMC article.
ARF couples with TP53 in a canonical signaling pathway to activate cellular senescence for tumor suppressive function under oncogenic insults. However, the mechanisms on aberrant elevation of ARF in cancers are still poorly understood. We previously showed th …
ARF couples with TP53 in a canonical signaling pathway to activate cellular senescence for tumor suppressive function under on …
Transcription factor KLLN inhibits tumor growth by AR suppression, induces apoptosis by TP53/TP73 stimulation in prostate carcinomas, and correlates with cellular differentiation.
Wang Y, Radhakrishnan D, He X, Peehl DM, Eng C. Wang Y, et al. J Clin Endocrinol Metab. 2013 Mar;98(3):E586-94. doi: 10.1210/jc.2012-3490. Epub 2013 Feb 5. J Clin Endocrinol Metab. 2013. PMID: 23386643 Free PMC article.
CONTEXT: KLLN is a newly identified gene with unknown function and shares a bidirectional promoter with PTEN. OBJECTIVE: The objective of the study was to analyze the relationship between KILLIN (KLLN) expression and prostate cancer and the potential tumor suppre
CONTEXT: KLLN is a newly identified gene with unknown function and shares a bidirectional promoter with PTEN. OBJECTIVE: The objectiv …
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