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Temporal characterization of optic fissure basement membrane composition suggests nidogen may be an initial target of remodeling.
Carrara N, Weaver M, Piedade WP, Vöcking O, Famulski JK. Carrara N, et al. Dev Biol. 2019 Aug 1;452(1):43-54. doi: 10.1016/j.ydbio.2019.04.012. Epub 2019 Apr 26. Dev Biol. 2019. PMID: 31034836 Free article.
Zebrafish optic fissure BMs were found to express laminin a1, a4, b1a, c1 and c3, nidogen 1a, 1b and 2a, collagen IV a1 and a2 as well as perlecan. ...
Zebrafish optic fissure BMs were found to express laminin a1, a4, b1a, c1 and c3, nidogen 1a, 1b and 2a, collagen IV a1 and a2 …
Activation of B-1 Cells Promotes Tumor Cell Killing in the Peritoneal Cavity.
Haro MA, Dyevoich AM, Phipps JP, Haas KM. Haro MA, et al. Cancer Res. 2019 Jan 1;79(1):159-170. doi: 10.1158/0008-5472.CAN-18-0981. Epub 2018 Sep 17. Cancer Res. 2019. PMID: 30224373 Free PMC article.
Consistent with this, we found significant deposition of IgM and C3 on peritoneal tumor cells as early as 5 days post-treatment. ...
Consistent with this, we found significant deposition of IgM and C3 on peritoneal tumor cells as early as 5 days post-treatment. ...
BAFF-driven autoimmunity requires CD19 expression.
Fairfax KA, Tsantikos E, Figgett WA, Vincent FB, Quah PS, LePage M, Hibbs ML, Mackay F. Fairfax KA, et al. J Autoimmun. 2015 Aug;62:1-10. doi: 10.1016/j.jaut.2015.06.001. Epub 2015 Jun 20. J Autoimmun. 2015. PMID: 26103922
These cells infiltrate the salivary glands and kidneys of diseased BAFF-Tg mice. However, loss of B1a or MZ B cells does not protect BAFF-Tg mice against disease, suggesting that B1b B cells might be the important pathogenic B cell subset. To test this hypothesis, we have …
These cells infiltrate the salivary glands and kidneys of diseased BAFF-Tg mice. However, loss of B1a or MZ B cells does not protect …
Sca-1 influences the innate immune response during skeletal muscle regeneration.
Long KK, Pavlath GK, Montano M. Long KK, et al. Am J Physiol Cell Physiol. 2011 Feb;300(2):C287-94. doi: 10.1152/ajpcell.00319.2010. Epub 2010 Dec 1. Am J Physiol Cell Physiol. 2011. PMID: 21123737 Free PMC article.
In the present study we demonstrate that Sca-1(-/-) mice have a defect in their capacity to recruit soluble IgM, and subsequently C3 complement, to damaged muscle. We hypothesize that this defect in recruitment delays or decreases phagocytosis by macrophages, contributing …
In the present study we demonstrate that Sca-1(-/-) mice have a defect in their capacity to recruit soluble IgM, and subsequently C3 …
Paucity of clinical disease despite serological autoimmunity and kidney pathology in lupus-prone New Zealand mixed 2328 mice deficient in BAFF.
Jacob CO, Pricop L, Putterman C, Koss MN, Liu Y, Kollaros M, Bixler SA, Ambrose CM, Scott ML, Stohl W. Jacob CO, et al. J Immunol. 2006 Aug 15;177(4):2671-80. doi: 10.4049/jimmunol.177.4.2671. J Immunol. 2006. PMID: 16888029 Free PMC article.
To determine whether SLE can develop de novo in BAFF-deficient hosts, BAFF-deficient New Zealand Mixed (NZM) 2328 (NZM.Baff(-/-)) mice were generated. In NZM.Baff(-/-) mice, spleen B cells (including CD5(+) B1a and CD5(-) B1b B cells), germinal centers, Ig-secreting cells, …
To determine whether SLE can develop de novo in BAFF-deficient hosts, BAFF-deficient New Zealand Mixed (NZM) 2328 (NZM.Baff(-/-)) mice were …
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