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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
2001 1
2006 1
2008 2
2009 2
2010 2
2011 2
2013 1
2014 2
2015 1
2016 1
2017 1
2018 2
2019 2
2023 0

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19 results

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Page 1
The cancer driver genes IDH1/2, JARID1C/ KDM5C, and UTX/ KDM6A: crosstalk between histone demethylation and hypoxic reprogramming in cancer metabolism.
Chang S, Yim S, Park H. Chang S, et al. Exp Mol Med. 2019 Jun 20;51(6):1-17. doi: 10.1038/s12276-019-0230-6. Exp Mol Med. 2019. PMID: 31221981 Free PMC article. Review.
Recent studies on mutations in cancer genomes have distinguished driver mutations from passenger mutations, which occur as byproducts of cancer development. ...We have also discussed mutations in other isoforms such as the JARID1A, 1B, 1D of KDM5 subfamilies and the …
Recent studies on mutations in cancer genomes have distinguished driver mutations from passenger mutations, which occur as byproducts …
Hereditary and familial thyroid tumours.
Guilmette J, Nosé V. Guilmette J, et al. Histopathology. 2018 Jan;72(1):70-81. doi: 10.1111/his.13373. Histopathology. 2018. PMID: 29239041 Review.
Hereditary thyroid neoplasms arising from calcitonin-producing C cells are known as familial medullary thyroid carcinomas (FMTCs), and include well-documented syndromes such as multiple endocrine neoplasia IIA or IIB, and pure familial me …
Hereditary thyroid neoplasms arising from calcitonin-producing C cells are known as familial medullary thyroid c …
Familial thyroid cancer: a review.
Nosé V. Nosé V. Mod Pathol. 2011 Apr;24 Suppl 2:S19-33. doi: 10.1038/modpathol.2010.147. Mod Pathol. 2011. PMID: 21455198 Free article. Review.
Thyroid carcinomas can be sporadic or familial. Familial syndromes are classified into familial medullary thyroid carcinoma (FMTC), derived from calcitonin-producing C cells, and familial non-medullary thyroid carcinoma
Thyroid carcinomas can be sporadic or familial. Familial syndromes are classified into familial medullary thy
Familial thyroid carcinoma: a diagnostic algorithm.
Dotto J, Nosé V. Dotto J, et al. Adv Anat Pathol. 2008 Nov;15(6):332-49. doi: 10.1097/PAP.0b013e31818a64af. Adv Anat Pathol. 2008. PMID: 18948764 Review.
Familial syndromes are classified into familial medullary thyroid carcinoma and familial nonmedullary thyroid carcinoma. Multifocal papillary carcinoma is the most frequent presentation of familial nonmedullary
Familial syndromes are classified into familial medullary thyroid carcinoma and familial nonmedullary
Familial nonmedullary thyroid cancer: screening, clinical, molecular and genetic findings.
Navas-Carrillo D, Ríos A, Rodríguez JM, Parrilla P, Orenes-Piñero E. Navas-Carrillo D, et al. Biochim Biophys Acta. 2014 Dec;1846(2):468-76. doi: 10.1016/j.bbcan.2014.09.002. Epub 2014 Sep 11. Biochim Biophys Acta. 2014. PMID: 25218916 Review.
Familial thyroid cancer has become a well-recognized, unique, clinical entity in patients with thyroid cancer originating from follicular cells, that is, nonmedullary thyroid carcinoma. Hereditary nonmedullary thyroid can
Familial thyroid cancer has become a well-recognized, unique, clinical entity in patients with thyroid cancer
Familial non-medullary thyroid carcinoma: an update.
Nosé V. Nosé V. Endocr Pathol. 2008 Winter;19(4):226-40. doi: 10.1007/s12022-008-9045-z. Endocr Pathol. 2008. PMID: 18931957 Review.
Familial thyroid cancer can arise from follicular cells (familial non-medullary thyroid carcinoma (FNMTC)) or from the calcitonin-producing C-cell (familial medullary thyroid carcinoma). This is usually a component of
Familial thyroid cancer can arise from follicular cells (familial non-medullary thyroid carcinoma
Next generation immunohistochemistry: Emerging substitutes to genetic testing?
Andrici J, Gill AJ, Hornick JL. Andrici J, et al. Semin Diagn Pathol. 2018 May;35(3):161-169. doi: 10.1053/j.semdp.2017.05.004. Epub 2017 Jun 27. Semin Diagn Pathol. 2018. PMID: 28662997 Review.
The identification of at-risk kindreds facilitates screening and risk reduction strategies for patients with hereditary cancer predisposition syndromes. Recently, immunohistochemistry (IHC) has emerged as a cost-effective strategy for detecting or inferring the presence of …
The identification of at-risk kindreds facilitates screening and risk reduction strategies for patients with hereditary cancer predis …
[A Familial Non Medullary Thyroid Carcinoma (FNMTC) : a clinical and genetic update].
Valdes-Socin H, Palmeira L, Burlacu MC, Daly AF, Bours V, Beckers A. Valdes-Socin H, et al. Rev Med Liege. 2016 Dec;71(12):557-561. Rev Med Liege. 2016. PMID: 28387096 Free article. Review. French.
The syndrome of Familial Non Medullary Thyroid Carcinoma (FNMTC) includes two or more patients with an isolated non-medullary thyroid cancer (papillary, follicular, anaplastic) within the same family. ...Several candidate genes of …
The syndrome of Familial Non Medullary Thyroid Carcinoma (FNMTC) includes two or more patients with an isolated non-med …
The evolving classification of renal cell neoplasia.
Delahunt B, Srigley JR. Delahunt B, et al. Semin Diagn Pathol. 2015 Mar;32(2):90-102. doi: 10.1053/j.semdp.2015.02.002. Epub 2015 Feb 4. Semin Diagn Pathol. 2015. PMID: 25753529 Review.
The classification of renal cell neoplasia is morphologically based; however, this has evolved over the last 35 years with the incorporation of genetic characteristics into the diagnostic features of some tumors. The 2013 Vancouver classification recognized 17 morph …
The classification of renal cell neoplasia is morphologically based; however, this has evolved over the last 35 years with the …
RET and neuroendocrine tumors.
Murakumo Y, Jijiwa M, Asai N, Ichihara M, Takahashi M. Murakumo Y, et al. Pituitary. 2006;9(3):179-92. doi: 10.1007/s11102-006-0263-4. Pituitary. 2006. PMID: 17036197 Review.
The RET proto-oncogene encodes a receptor tyrosine kinase that is a main component of the signaling pathway activated by the glial cell line-derived neurotrophic factor family ligands. Gene targeting studies revealed that signaling through RET plays a crucial role in neuro …
The RET proto-oncogene encodes a receptor tyrosine kinase that is a main component of the signaling pathway activated by the glial cell line …
19 results