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Mechanisms of resistance to TRAIL-induced apoptosis in cancer.
Zhang L, Fang B. Zhang L, et al. Among authors: fang b. Cancer Gene Ther. 2005 Mar;12(3):228-37. doi: 10.1038/sj.cgt.7700792. Cancer Gene Ther. 2005. PMID: 15550937 Review.
Finally, activation of different subunits of mitogen-activated protein kinases or nuclear factor-kappa B can lead to development of either TRAIL resistance or apoptosis in certain types of cancer cells....
Finally, activation of different subunits of mitogen-activated protein kinases or nuclear factor-kappa B can lead to development of e …
Gene therapy for hemophilia B: host immunosuppression prolongs the therapeutic effect of adenovirus-mediated factor IX expression.
Fang B, Eisensmith RC, Wang H, Kay MA, Cross RE, Landen CN, Gordon G, Bellinger DA, Read MS, Hu PC, et al. Fang B, et al. Hum Gene Ther. 1995 Aug;6(8):1039-44. doi: 10.1089/hum.1995.6.8-1039. Hum Gene Ther. 1995. PMID: 7578416
Hemophilia B is caused by a deficiency of blood clotting factor IX (FIX). Previous studies have shown that the delivery of a recombinant adenoviral vector expressing canine FIX (cFIX) resulted in a complete correction of hemophilia B in FIX-deficient dogs, but that …
Hemophilia B is caused by a deficiency of blood clotting factor IX (FIX). Previous studies have shown that the delivery of a recombin …
Lack of persistence of E1- recombinant adenoviral vectors containing a temperature-sensitive E2A mutation in immunocompetent mice and hemophilia B dogs.
Fang B, Wang H, Gordon G, Bellinger DA, Read MS, Brinkhous KM, Woo SL, Eisensmith RC. Fang B, et al. Gene Ther. 1996 Mar;3(3):217-22. Gene Ther. 1996. PMID: 8646552
The effects of the inclusion of the ts125 mutation on transgene expression in vivo were evaluated in Balb/c mice and hemophilia B dogs by comparison with adenoviral vectors containing the same transgene but lacking the ts125 mutation. ...
The effects of the inclusion of the ts125 mutation on transgene expression in vivo were evaluated in Balb/c mice and hemophilia B dog …
Suppression of the immune response to an adenovirus vector and enhancement of intratumoral transgene expression by low-dose etoposide.
Bouvet M, Fang B, Ekmekcioglu S, Ji L, Bucana CD, Hamada K, Grimm EA, Roth JA. Bouvet M, et al. Among authors: fang b. Gene Ther. 1998 Feb;5(2):189-95. doi: 10.1038/sj.gt.3300564. Gene Ther. 1998. PMID: 9578838
Adenovirus-mediated wild-type p53 gene transfer down-regulates vascular endothelial growth factor expression and inhibits angiogenesis in human colon cancer.
Bouvet M, Ellis LM, Nishizaki M, Fujiwara T, Liu W, Bucana CD, Fang B, Lee JJ, Roth JA. Bouvet M, et al. Among authors: fang b. Cancer Res. 1998 Jun 1;58(11):2288-92. Cancer Res. 1998. PMID: 9622060
Adenovirus-mediated wild-type p53 tumor suppressor gene therapy induces apoptosis and suppresses growth of human pancreatic cancer [seecomments].
Bouvet M, Bold RJ, Lee J, Evans DB, Abbruzzese JL, Chiao PJ, McConkey DJ, Chandra J, Chada S, Fang B, Roth JA. Bouvet M, et al. Among authors: fang b. Ann Surg Oncol. 1998 Dec;5(8):681-8. doi: 10.1007/BF02303477. Ann Surg Oncol. 1998. PMID: 9869513
Adenovirus-mediated delivery of antisense gene to urokinase-type plasminogen activator receptor suppresses glioma invasion and tumor growth.
Mohan PM, Chintala SK, Mohanam S, Gladson CL, Kim ES, Gokaslan ZL, Lakka SS, Roth JA, Fang B, Sawaya R, Kyritsis AP, Rao JS. Mohan PM, et al. Among authors: fang b. Cancer Res. 1999 Jul 15;59(14):3369-73. Cancer Res. 1999. PMID: 10416596
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