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Differential susceptibility following beta-amyloid peptide-(1-40) administration in C57BL/6 and Swiss albino mice: Evidence for a dissociation between cognitive deficits and the glutathione system response.
Prediger RD, Franco JL, Pandolfo P, Medeiros R, Duarte FS, Di Giunta G, Figueiredo CP, Farina M, Calixto JB, Takahashi RN, Dafre AL. Prediger RD, et al. Among authors: figueiredo cp. Behav Brain Res. 2007 Feb 27;177(2):205-13. doi: 10.1016/j.bbr.2006.11.032. Epub 2006 Dec 27. Behav Brain Res. 2007. PMID: 17194489
Connecting TNF-alpha signaling pathways to iNOS expression in a mouse model of Alzheimer's disease: relevance for the behavioral and synaptic deficits induced by amyloid beta protein.
Medeiros R, Prediger RD, Passos GF, Pandolfo P, Duarte FS, Franco JL, Dafre AL, Di Giunta G, Figueiredo CP, Takahashi RN, Campos MM, Calixto JB. Medeiros R, et al. Among authors: figueiredo cp. J Neurosci. 2007 May 16;27(20):5394-404. doi: 10.1523/JNEUROSCI.5047-06.2007. J Neurosci. 2007. PMID: 17507561 Free PMC article.
Role of the macrophage inflammatory protein-1alpha/CC chemokine receptor 5 signaling pathway in the neuroinflammatory response and cognitive deficits induced by beta-amyloid peptide.
Passos GF, Figueiredo CP, Prediger RD, Pandolfo P, Duarte FS, Medeiros R, Calixto JB. Passos GF, et al. Among authors: figueiredo cp. Am J Pathol. 2009 Oct;175(4):1586-97. doi: 10.2353/ajpath.2009.081113. Epub 2009 Sep 3. Am J Pathol. 2009. PMID: 19729478 Free PMC article.
136 results