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BCR/ABL regulates mammalian RecA homologs, resulting in drug resistance.
Slupianek A, Schmutte C, Tombline G, Nieborowska-Skorska M, Hoser G, Nowicki MO, Pierce AJ, Fishel R, Skorski T. Slupianek A, et al. Among authors: Fishel R. Mol Cell. 2001 Oct;8(4):795-806. doi: 10.1016/s1097-2765(01)00357-4. Mol Cell. 2001. PMID: 11684015
hXRCC2 enhances ADP/ATP processing and strand exchange by hRAD51.
Shim KS, Schmutte C, Tombline G, Heinen CD, Fishel R. Shim KS, et al. Among authors: Fishel R. J Biol Chem. 2004 Jul 16;279(29):30385-94. doi: 10.1074/jbc.M306066200. Epub 2004 May 3. J Biol Chem. 2004. PMID: 15123651
Fusion tyrosine kinases induce drug resistance by stimulation of homology-dependent recombination repair, prolongation of G(2)/M phase, and protection from apoptosis.
Slupianek A, Hoser G, Majsterek I, Bronisz A, Malecki M, Blasiak J, Fishel R, Skorski T. Slupianek A, et al. Among authors: Fishel R. Mol Cell Biol. 2002 Jun;22(12):4189-201. doi: 10.1128/mcb.22.12.4189-4201.2002. Mol Cell Biol. 2002. PMID: 12024032 Free PMC article.
Fusion tyrosine kinases (FTKs) such as BCR/ABL, TEL/ABL, TEL/JAK2, TEL/PDGF beta R, TEL/TRKC(L), and NPM/ALK arise from reciprocal chromosomal translocations and cause acute and chronic leukemias and non-Hodgkin's lymphoma. ...
Fusion tyrosine kinases (FTKs) such as BCR/ABL, TEL/ABL, TEL/JAK2, TEL/PDGF beta R, TEL/TRKC(L), and NPM/ALK arise from reciprocal ch …
BRCA1 and cell signaling.
Wang Q, Zhang H, Fishel R, Greene MI. Wang Q, et al. Among authors: Fishel R. Oncogene. 2000 Dec 11;19(53):6152-8. doi: 10.1038/sj.onc.1203974. Oncogene. 2000. PMID: 11156529 Review.
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