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Age-related impairment of synaptic transmission but normal long-term potentiation in transgenic mice that overexpress the human APP695SWE mutant form of amyloid precursor protein.
Fitzjohn SM, Morton RA, Kuenzi F, Rosahl TW, Shearman M, Lewis H, Smith D, Reynolds DS, Davies CH, Collingridge GL, Seabrook GR. Fitzjohn SM, et al. J Neurosci. 2001 Jul 1;21(13):4691-8. doi: 10.1523/JNEUROSCI.21-13-04691.2001. J Neurosci. 2001. PMID: 11425896 Free PMC article.
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