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Transgenic-mouse model of amyotrophic lateral sclerosis.
Gurney ME. Gurney ME. N Engl J Med. 1994 Dec 22;331(25):1721-2. doi: 10.1056/NEJM199412223312516. N Engl J Med. 1994. PMID: 7832899 No abstract available.
The copper chelator d-penicillamine delays onset of disease and extends survival in a transgenic mouse model of familial amyotrophic lateral sclerosis.
Hottinger AF, Fine EG, Gurney ME, Zurn AD, Aebischer P. Hottinger AF, et al. Eur J Neurosci. 1997 Jul;9(7):1548-51. doi: 10.1111/j.1460-9568.1997.tb01511.x. Eur J Neurosci. 1997. PMID: 9240414
A low expressor line of transgenic mice carrying a mutant human Cu,Zn superoxide dismutase (SOD1) gene develops pathological changes that most closely resemble those in human amyotrophic lateral sclerosis.
Dal Canto MC, Gurney ME. Dal Canto MC, et al. Acta Neuropathol. 1997 Jun;93(6):537-50. doi: 10.1007/s004010050650. Acta Neuropathol. 1997. PMID: 9194892
Transgenic animal models of familial amyotrophic lateral sclerosis.
Gurney ME. Gurney ME. J Neurol. 1997 May;244 Suppl 2:S15-20. doi: 10.1007/BF03160575. J Neurol. 1997. PMID: 9178166
Transgenic mice carrying a human mutant superoxide dismutase transgene develop neuronal cytoskeletal pathology resembling human amyotrophic lateral sclerosis lesions.
Tu PH, Raju P, Robinson KA, Gurney ME, Trojanowski JQ, Lee VM. Tu PH, et al. Proc Natl Acad Sci U S A. 1996 Apr 2;93(7):3155-60. doi: 10.1073/pnas.93.7.3155. Proc Natl Acad Sci U S A. 1996. PMID: 8610185 Free PMC article.
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