Hancock WW - Search Results

1

The NOD Idd9 genetic interval influences the pathogenicity of insulitis and contains molecular variants of Cd30, Tnfr2, and Cd137.

Lyons PA, Hancock WW, Denny P, Lord CJ, Hill NJ, Armitage N, Siegmund T, Todd JA, Phillips MS, Hess JF, Chen SL, Fischer PA, Peterson LB, Wicker LS. Lyons PA, et al. Among authors: hancock ww. Immunity. 2000 Jul;13(1):107-15. doi: 10.1016/s1074-7613(00)00012-1. Immunity. 2000. PMID: 10933399 Free article.

2

Suppression of insulitis in non-obese diabetic (NOD) mice by oral insulin administration is associated with selective expression of interleukin-4 and -10, transforming growth factor-beta, and prostaglandin-E.

Hancock WW, Polanski M, Zhang J, Blogg N, Weiner HL. Hancock WW, et al. Am J Pathol. 1995 Nov;147(5):1193-9. Am J Pathol. 1995. PMID: 7485382 Free PMC article.

3

TRAF6 is a critical factor for dendritic cell maturation and development.

Kobayashi T, Walsh PT, Walsh MC, Speirs KM, Chiffoleau E, King CG, Hancock WW, Caamano JH, Hunter CA, Scott P, Turka LA, Choi Y. Kobayashi T, et al. Among authors: hancock ww. Immunity. 2003 Sep;19(3):353-63. doi: 10.1016/s1074-7613(03)00230-9. Immunity. 2003. PMID: 14499111 Free article.

4

Modulation of LIGHT-HVEM costimulation prolongs cardiac allograft survival.

Ye Q, Fraser CC, Gao W, Wang L, Busfield SJ, Wang C, Qiu Y, Coyle AJ, Gutierrez-Ramos JC, Hancock WW. Ye Q, et al. Among authors: hancock ww. J Exp Med. 2002 Mar 18;195(6):795-800. doi: 10.1084/jem.20012088. J Exp Med. 2002. PMID: 11901205 Free PMC article.

5

Islet mass plays a critical role in initiation, but not progression, of the diabetogenic process in NOD mice.

Kimura T, Ito A, Hancock WW, Maki T. Kimura T, et al. Among authors: hancock ww. J Autoimmun. 1999 Jun;12(4):243-9. doi: 10.1006/jaut.1999.0283. J Autoimmun. 1999. PMID: 10330295

6

Oral tolerance to myelin basic protein and natural recovery from experimental autoimmune encephalomyelitis are associated with downregulation of inflammatory cytokines and differential upregulation of transforming growth factor beta, interleukin 4, and prostaglandin E expression in the brain.

Khoury SJ, Hancock WW, Weiner HL. Khoury SJ, et al. Among authors: hancock ww. J Exp Med. 1992 Nov 1;176(5):1355-64. doi: 10.1084/jem.176.5.1355. J Exp Med. 1992. PMID: 1383385 Free PMC article.

7

Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory cytokines.

Maron R, Hancock WW, Slavin A, Hattori M, Kuchroo V, Weiner HL. Maron R, et al. Among authors: hancock ww. Int Immunol. 1999 Sep;11(9):1573-80. doi: 10.1093/intimm/11.9.1573. Int Immunol. 1999. PMID: 10464178

8

Nasal administration of amyloid-beta peptide decreases cerebral amyloid burden in a mouse model of Alzheimer's disease.

Weiner HL, Lemere CA, Maron R, Spooner ET, Grenfell TJ, Mori C, Issazadeh S, Hancock WW, Selkoe DJ. Weiner HL, et al. Among authors: hancock ww. Ann Neurol. 2000 Oct;48(4):567-79. Ann Neurol. 2000. PMID: 11026440

9

Oral, but not intravenous, alloantigen prevents accelerated allograft rejection by selective intragraft Th2 cell activation.

Hancock WW, Sayegh MH, Kwok CA, Weiner HL, Carpenter CB. Hancock WW, et al. Transplantation. 1993 May;55(5):1112-8. doi: 10.1097/00007890-199305000-00034. Transplantation. 1993. PMID: 8497891

10

Mechanisms of oral tolerance by MHC peptides.

Sayegh MH, Khoury SJ, Hancock WW, Weiner HL, Carpenter CB. Sayegh MH, et al. Among authors: hancock ww. Ann N Y Acad Sci. 1996 Feb 13;778:338-45. doi: 10.1111/j.1749-6632.1996.tb21141.x. Ann N Y Acad Sci. 1996. PMID: 8610987 Review.

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