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Glucocorticoid-induced apoptosis of human leukemic cells is caused by the repressive function of the glucocorticoid receptor.
Helmberg A, Auphan N, Caelles C, Karin M. Helmberg A, et al. EMBO J. 1995 Feb 1;14(3):452-60. EMBO J. 1995. PMID: 7859735 Free PMC article.
A prevailing hypothesis postulates induction of so-called 'lysis genes' by the activated receptor. In this study, we show that an activation-deficient glucocorticoid receptor mutant is as effective as the wild-type receptor in repression of AP-1 activity, inhibition of int
A prevailing hypothesis postulates induction of so-called 'lysis genes' by the activated receptor. In this study, we show that an act
c-Myc does not prevent glucocorticoid-induced apoptosis of human leukemic lymphoblasts.
Löffler M, Ausserlechner MJ, Tonko M, Hartmann BL, Bernhard D, Geley S, Helmberg A, Kofler R. Löffler M, et al. Among authors: helmberg a. Oncogene. 1999 Aug 12;18(32):4626-31. doi: 10.1038/sj.onc.1202820. Oncogene. 1999. PMID: 10467407 Free article.
Gene expression profiles of proliferating vs. G1/G0 arrested human leukemia cells suggest a mechanism for glucocorticoid-induced apoptosis.
Tonko M, Ausserlechner MJ, Bernhard D, Helmberg A, Kofler R. Tonko M, et al. Among authors: helmberg a. FASEB J. 2001 Mar;15(3):693-9. doi: 10.1096/fj.00-0327com. FASEB J. 2001. PMID: 11259387
Immunoregulatory genes and immunosuppression by glucocorticoids.
Auphan N, Didonato JA, Helmberg A, Rosette C, Karin M. Auphan N, et al. Among authors: helmberg a. Arch Toxicol Suppl. 1997;19:87-95. doi: 10.1007/978-3-642-60682-3_8. Arch Toxicol Suppl. 1997. PMID: 9079197 No abstract available.
p53-dependent apoptosis in the absence of transcriptional activation of p53-target genes.
Caelles C, Helmberg A, Karin M. Caelles C, et al. Nature. 1994 Jul 21;370(6486):220-3. doi: 10.1038/370220a0. Nature. 1994. PMID: 8028670
As p53 is a transcriptional activator that mediates gene induction after DNA damage, it has been proposed to be a genetic switch that activates apoptosis-mediator genes. ...A shift to the permissive temperature triggers apoptosis following DNA damage, but thi …
As p53 is a transcriptional activator that mediates gene induction after DNA damage, it has been proposed to be a genetic swit …
Expression profiling of glucocorticoid-treated T-ALL cell lines: rapid repression of multiple genes involved in RNA-, protein- and nucleotide synthesis.
Obexer P, Certa U, Kofler R, Helmberg A. Obexer P, et al. Among authors: helmberg a. Oncogene. 2001 Jul 19;20(32):4324-36. doi: 10.1038/sj.onc.1204573. Oncogene. 2001. PMID: 11466613 Free article.
A RAS recruitment screen identifies ZKSCAN4 as a glucocorticoid receptor-interacting protein.
Ecker K, Lorenz A, Wolf F, Ploner C, Böck G, Duncan T, Geley S, Helmberg A. Ecker K, et al. Among authors: helmberg a. J Mol Endocrinol. 2009 Feb;42(2):105-17. doi: 10.1677/JME-08-0087. Epub 2008 Nov 13. J Mol Endocrinol. 2009. PMID: 19008334
Gene expression profiling of human endothelial cells exposed to 50-Hz magnetic fields fails to produce regulated candidate genes.
Henderson B, Kind M, Boeck G, Helmberg A, Wick G. Henderson B, et al. Among authors: helmberg a. Cell Stress Chaperones. 2006 Autumn;11(3):227-32. doi: 10.1379/csc-196.1. Cell Stress Chaperones. 2006. PMID: 17009595 Free PMC article.
Identification, tissue expression, and glucocorticoid responsiveness of alternative first exons of the human glucocorticoid receptor.
Presul E, Schmidt S, Kofler R, Helmberg A. Presul E, et al. Among authors: helmberg a. J Mol Endocrinol. 2007 Feb;38(1-2):79-90. doi: 10.1677/jme.1.02183. J Mol Endocrinol. 2007. PMID: 17242171
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