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Hydrogen sulfide inhibits Cav3.2 T-type Ca2+ channels.
Elies J, Scragg JL, Huang S, Dallas ML, Huang D, MacDougall D, Boyle JP, Gamper N, Peers C. Elies J, et al. FASEB J. 2014 Dec;28(12):5376-87. doi: 10.1096/fj.14-257113. Epub 2014 Sep 2. FASEB J. 2014. PMID: 25183670
Inhibition of native T-type channels in 3 cell lines correlated with expression of Cav3.2 and not Cav3.1 channels. Notably, H2S also inhibited native T-type (primarily Cav3.2) channels
Inhibition of native T-type channels in 3 cell lines correlated with expression of Cav3.2 and not
Inhibition of T-type Ca2+ Channels by Hydrogen Sulfide.
Elies J, Scragg JL, Dallas ML, Huang D, Huang S, Boyle JP, Gamper N, Peers C. Elies J, et al. Adv Exp Med Biol. 2015;860:353-60. doi: 10.1007/978-3-319-18440-1_40. Adv Exp Med Biol. 2015. PMID: 26303500
Using patch-clamp electrophysiology, we demonstrate that the H(2)S donor, NaHS, selectively inhibits Cav3.2 T-type Ca(2+) channels heterologously expressed in HEK293 cells, whilst Cav3.1 and Cav3.3 ch
Using patch-clamp electrophysiology, we demonstrate that the H(2)S donor, NaHS, selectively inhibits Cav3.2
H2S does not regulate proliferation via T-type Ca2+ channels.
Elies J, Johnson E, Boyle JP, Scragg JL, Peers C. Elies J, et al. Biochem Biophys Res Commun. 2015 Jun 12;461(4):659-64. doi: 10.1016/j.bbrc.2015.04.087. Epub 2015 Apr 24. Biochem Biophys Res Commun. 2015. PMID: 25918023 Free article.
We have also shown that the T-type Ca(2+) channel Cav3.2 is selectively inhibited by hydrogen sulfide (H2S) whilst the other channel isoforms (Cav3.1 and Cav3.3) are unaffected. ...Our data indica …
We have also shown that the T-type Ca(2+) channel Cav3.2 is selectively inhibited by hyd
Regulation of the T-type Ca(2+) channel Cav3.2 by hydrogen sulfide: emerging controversies concerning the role of H2 S in nociception.
Elies J, Scragg JL, Boyle JP, Gamper N, Peers C. Elies J, et al. J Physiol. 2016 Aug 1;594(15):4119-29. doi: 10.1113/JP270963. Epub 2016 Feb 25. J Physiol. 2016. PMID: 26804000 Free PMC article. Review.
Indeed, many of the biological actions of these gases can be accounted for by their ability to modulate ion channel activity. Here, we report recent evidence that H2 S is a modulator of low voltage-activated T-type Ca(2+) channels, and di …
Indeed, many of the biological actions of these gases can be accounted for by their ability to modulate ion channel activity. Here, w …
Endogenous and exogenous hydrogen sulfide facilitates T-type calcium channel currents in Cav3.2-expressing HEK293 cells.
Sekiguchi F, Miyamoto Y, Kanaoka D, Ide H, Yoshida S, Ohkubo T, Kawabata A. Sekiguchi F, et al. Biochem Biophys Res Commun. 2014 Feb 28;445(1):225-9. doi: 10.1016/j.bbrc.2014.01.185. Epub 2014 Feb 6. Biochem Biophys Res Commun. 2014. PMID: 24508802
We have shown that an H2S donor, NaHS, causes hyperalgesia in rodents, an effect inhibited by knockdown of Cav3.2 T-type Ca(2+) channels (T-channels), and that NaHS facilitates T-channel-dependent curren …
We have shown that an H2S donor, NaHS, causes hyperalgesia in rodents, an effect inhibited by knockdown of Cav3.2 T
Hyperalgesia induced by spinal and peripheral hydrogen sulfide: evidence for involvement of Cav3.2 T-type calcium channels.
Maeda Y, Aoki Y, Sekiguchi F, Matsunami M, Takahashi T, Nishikawa H, Kawabata A. Maeda Y, et al. Pain. 2009 Mar;142(1-2):127-32. doi: 10.1016/j.pain.2008.12.021. Epub 2009 Jan 23. Pain. 2009. PMID: 19167819
NaHS in rats, and then examined if Cav3.2 isoform of T-type Ca2+ channels contributed to the pro-nociceptive effects of i.t. and i.pl. ...The hyperalgesia caused by i.t. and i.pl. NaHS was abolished by co-administration of m …
NaHS in rats, and then examined if Cav3.2 isoform of T-type Ca2+ channels contributed to the pro-n …
Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis.
Terada Y, Fujimura M, Nishimura S, Tsubota M, Sekiguchi F, Kawabata A. Terada Y, et al. J Neurosci Res. 2015 Feb;93(2):361-9. doi: 10.1002/jnr.23490. Epub 2014 Sep 30. J Neurosci Res. 2015. PMID: 25267397
Hydrogen sulfide (H(2)S), formed by multiple enzymes, including cystathionine-gamma-lyase (CSE), targets Ca(v)3.2 T-type Ca(2+) channels (T channels) and transient receptor potential ankyrin-1 (TRPA1), f
Hydrogen sulfide (H(2)S), formed by multiple enzymes, including cystathionine-gamma-lyase (CSE), targets Ca(v)3.
Effects of AP39, a novel triphenylphosphonium derivatised anethole dithiolethione hydrogen sulfide donor, on rat haemodynamic parameters and chloride and calcium Cav3 and RyR2 channels.
Tomasova L, Pavlovicova M, Malekova L, Misak A, Kristek F, Grman M, Cacanyiova S, Tomasek M, Tomaskova Z, Perry A, Wood ME, Lacinova L, Ondrias K, Whiteman M. Tomasova L, et al. Nitric Oxide. 2015 Apr 30;46:131-44. doi: 10.1016/j.niox.2014.12.012. Epub 2014 Dec 30. Nitric Oxide. 2015. PMID: 25555533
The involvement of membrane channels T-type Ca(2+) channels CaV3.1, CaV3.2 and CaV3.3 as well as Ca(2+) ryanodine (RyR2) and Cl(-) single channels derived from rat heart sarcoplasmic reticulum were also …
The involvement of membrane channels T-type Ca(2+) channels CaV3.1, CaV3.2 and Ca
H2S and Pain: A Novel Aspect for Processing of Somatic, Visceral and Neuropathic Pain Signals.
Terada Y, Kawabata A. Terada Y, et al. Handb Exp Pharmacol. 2015;230:217-30. doi: 10.1007/978-3-319-18144-8_11. Handb Exp Pharmacol. 2015. PMID: 26162837 Review.
Hydrogen sulfide (H2S) formed by multiple enzymes including cystathionine-gamma-lyase (CSE) targets Cav3.2 T-type Ca2+ channels (T-channels) and transient receptor potential ankyrin-1 (TRPA1). ...Electrophysiol
Hydrogen sulfide (H2S) formed by multiple enzymes including cystathionine-gamma-lyase (CSE) targets Cav3.2 T
[Role of hydrogen sulfide, a gasotransmitter, in colonic pain and inflammation].
Tsubota M, Kawabata A. Tsubota M, et al. Yakugaku Zasshi. 2014;134(12):1245-52. doi: 10.1248/yakushi.14-00209-2. Yakugaku Zasshi. 2014. PMID: 25452234 Free article. Review. Japanese.
We previously demonstrated that H2S activates Cav3.2 T-type Ca(2+) channels expressed on sensory neurons, leading to hyperalgesia and facilitation of inflammation. ...In rats with colitis induced by 2,4,6-trinitrobenzenesulfonic ac …
We previously demonstrated that H2S activates Cav3.2 T-type Ca(2+) channels expressed on sensory n …
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