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The transformation of primary and established mouse mammary epithelial cells by p21-ras is concentration dependent.
Redmond SM, Reichmann E, Müller RG, Friis RR, Groner B, Hynes NE. Redmond SM, et al. Among authors: hynes ne. Oncogene. 1988 Mar;2(3):259-65. Oncogene. 1988. PMID: 3127778
Essential functions of p21-activated kinase 1 in morphogenesis and differentiation of mammary glands.
Wang RA, Vadlamudi RK, Bagheri-Yarmand R, Beuvink I, Hynes NE, Kumar R. Wang RA, et al. Among authors: hynes ne. J Cell Biol. 2003 May 12;161(3):583-92. doi: 10.1083/jcb.200212066. Epub 2003 May 5. J Cell Biol. 2003. PMID: 12732616 Free PMC article.
Chronic expression of wild-type Ret receptor in the mammary gland induces luminal tumors that are sensitive to Ret inhibition.
Gattelli A, García Solá ME, Roloff TC, Cardiff RD, Kordon EC, Chodosh LA, Hynes NE. Gattelli A, et al. Among authors: hynes ne. Oncogene. 2018 Jul;37(29):4046-4054. doi: 10.1038/s41388-018-0235-y. Epub 2018 Apr 26. Oncogene. 2018. PMID: 29695833
Tenascin-C blocks cell-cycle progression of anchorage-dependent fibroblasts on fibronectin through inhibition of syndecan-4.
Orend G, Huang W, Olayioye MA, Hynes NE, Chiquet-Ehrismann R. Orend G, et al. Among authors: hynes ne. Oncogene. 2003 Jun 19;22(25):3917-26. doi: 10.1038/sj.onc.1206618. Oncogene. 2003. PMID: 12813465
Blocking of FGFR signaling inhibits breast cancer cell proliferation through downregulation of D-type cyclins.
Koziczak M, Holbro T, Hynes NE. Koziczak M, et al. Among authors: hynes ne. Oncogene. 2004 Apr 29;23(20):3501-8. doi: 10.1038/sj.onc.1207331. Oncogene. 2004. PMID: 15116089
Beta1 integrins regulate mammary gland proliferation and maintain the integrity of mammary alveoli.
Li N, Zhang Y, Naylor MJ, Schatzmann F, Maurer F, Wintermantel T, Schuetz G, Mueller U, Streuli CH, Hynes NE. Li N, et al. Among authors: hynes ne. EMBO J. 2005 Jun 1;24(11):1942-53. doi: 10.1038/sj.emboj.7600674. Epub 2005 May 12. EMBO J. 2005. PMID: 15889143 Free PMC article.
Mammary gland development and lactation are controlled by different glucocorticoid receptor activities.
Reichardt HM, Horsch K, Gröne HJ, Kolbus A, Beug H, Hynes N, Schütz G. Reichardt HM, et al. Eur J Endocrinol. 2001 Oct;145(4):519-27. doi: 10.1530/eje.0.1450519. Eur J Endocrinol. 2001. PMID: 11581013
Induction of transforming growth factor alpha expression in mouse mammary epithelial cells after transformation with a point-mutated c-Ha-ras protooncogene.
Ciardiello F, Kim N, Hynes N, Jaggi R, Redmond S, Liscia DS, Sanfilippo B, Merlo G, Callahan R, Kidwell WR, et al. Ciardiello F, et al. Mol Endocrinol. 1988 Dec;2(12):1202-16. doi: 10.1210/mend-2-12-1202. Mol Endocrinol. 1988. PMID: 3063955
Transforming growth factor-alpha expression is enhanced in human mammary epithelial cells transformed by an activated c-Ha-ras protooncogene but not by the c-neu protooncogene, and overexpression of the transforming growth factor-alpha complementary DNA leads to transformation.
Ciardiello F, McGeady ML, Kim N, Basolo F, Hynes N, Langton BC, Yokozaki H, Saeki T, Elliott JW, Masui H, et al. Ciardiello F, et al. Cell Growth Differ. 1990 Sep;1(9):407-20. Cell Growth Differ. 1990. PMID: 1981145
Transformation of mouse mammary epithelial cells with the Ha-ras but not with the neu oncogene results in a gene dosage-dependent increase in transforming growth factor-alpha production.
Ciardiello F, Hynes N, Kim N, Valverius EM, Lippman ME, Salomon DS. Ciardiello F, et al. FEBS Lett. 1989 Jul 3;250(2):474-8. doi: 10.1016/0014-5793(89)80779-3. FEBS Lett. 1989. PMID: 2568949
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