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1996 1
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2000 4
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Insulin attenuates apoptosis in neuronal cells by an integrin-linked kinase-dependent mechanism.
Tan J, Digicaylioglu M, Wang SXJ, Dresselhuis J, Dedhar S, Mills J. Tan J, et al. Heliyon. 2019 Aug 16;5(8):e02294. doi: 10.1016/j.heliyon.2019.e02294. eCollection 2019 Aug. Heliyon. 2019. PMID: 31463398 Free PMC article.
Insulin promotes neuronal survival by activating a phosphatidylinositol 3-kinase (PI 3-kinase)/AKT-dependent signaling pathway and reducing caspase activation. ...Insulin and IGF-1 decreased ca
Insulin promotes neuronal survival by activating a phosphatidylinositol 3-kinase (PI 3-kinase
Insulin activates caspase-3 by a phosphatidylinositol 3'-kinase-dependent pathway.
Godbout JP, Cengel KA, Cheng SL, Minshall C, Kelley KW, Freund GG. Godbout JP, et al. Cell Signal. 1999 Jan;11(1):15-23. doi: 10.1016/s0898-6568(98)00024-2. Cell Signal. 1999. PMID: 10206340
Here we report that insulin activates caspase-3 by a pathway requiring phosphatidylinositol 3'-kinase (PI3-kinase). ...Taken together, these findings indicate that insulin activates caspase-3
Here we report that insulin activates caspase-3 by a pathway requiring phosphatidylinositol 3
Combined effects of 17β-estradiol and exercise training on cardiac apoptosis in ovariectomized rats.
Lin YY, Chen JS, Wu XB, Shyu WC, Chaunchaiyakul R, Zhao XL, Kuo CH, Cheng YJ, Yang AL, Lee SD. Lin YY, et al. PLoS One. 2018 Dec 20;13(12):e0208633. doi: 10.1371/journal.pone.0208633. eCollection 2018. PLoS One. 2018. PMID: 30571718 Free PMC article.
The protein levels of truncation of Bid (t-Bid), Bcl-2-associated death promotor (Bad), Bcl-2-associated X protein (Bax), Cytochrome c, caspases-9, and caspases-3 (mitochondria-dependent apoptotic pathway), as well as the protein levels of tumor necrosis fact …
The protein levels of truncation of Bid (t-Bid), Bcl-2-associated death promotor (Bad), Bcl-2-associated X protein (Bax), Cytochrome c, casp …
Insulin rescues retinal neurons from apoptosis by a phosphatidylinositol 3-kinase/Akt-mediated mechanism that reduces the activation of caspase-3.
Barber AJ, Nakamura M, Wolpert EB, Reiter CE, Seigel GM, Antonetti DA, Gardner TW. Barber AJ, et al. J Biol Chem. 2001 Aug 31;276(35):32814-21. doi: 10.1074/jbc.M104738200. Epub 2001 Jul 6. J Biol Chem. 2001. PMID: 11443130 Free article.
Insulin induced serine phosphorylation of Akt, through the phosphatidylinositol (PI) 3-kinase pathway. ...These results suggest that insulin is a survival factor for retinal neurons by activating the PI 3-kinase/Akt
Insulin induced serine phosphorylation of Akt, through the phosphatidylinositol (PI) 3-kinase pathway. ..
IGF-1 protects SH-SY5Y cells against MPP(+)-induced apoptosis via PI3K/PDK-1/Akt pathway.
Kim C, Park S. Kim C, et al. Endocr Connect. 2018 Mar;7(3):443-455. doi: 10.1530/EC-17-0350. Epub 2018 Feb 19. Endocr Connect. 2018. PMID: 29459421 Free PMC article.
Insulin-like growth factor (IGF)-1 is a well-known anti-apoptotic pro-survival factor and phosphatidylinositol-3-kinase (PI3K)/Akt pathway is linked to cell survival induced by IGF-1. It is also reported that Akt signaling is modulated by 3
Insulin-like growth factor (IGF)-1 is a well-known anti-apoptotic pro-survival factor and phosphatidylinositol-3-kin
Autophagy in the physiological endometrium and cancer.
Devis-Jauregui L, Eritja N, Davis ML, Matias-Guiu X, Llobet-Navàs D. Devis-Jauregui L, et al. Autophagy. 2020 May 13:1-19. doi: 10.1080/15548627.2020.1752548. Online ahead of print. Autophagy. 2020. PMID: 32401642
Autophagy is a highly conserved catabolic process and a major cellular pathway for the degradation of long-lived proteins and cytoplasmic organelles. ...Current research efforts aiming to therapeutically exploit autophagy and the methodological approaches used are discusse …
Autophagy is a highly conserved catabolic process and a major cellular pathway for the degradation of long-lived proteins and cytopla …
Baicalin prevents tumor necrosis factor-α-induced apoptosis and dysfunction of pancreatic β-cell line Min6 via upregulation of miR-205.
You W, Wang K, Yu C, Song L. You W, et al. J Cell Biochem. 2018 Nov;119(10):8547-8554. doi: 10.1002/jcb.27095. Epub 2018 Jul 30. J Cell Biochem. 2018. PMID: 30058243
Expression of apoptosis-associated proteins and inducible nitric oxide synthase (iNOS), and activation of phosphatidylinositol 3'-kinase/protein kinase B (PI3K/AKT) and nuclear factor kappaB (NF-kappaB) pathways were analyzed by Western b …
Expression of apoptosis-associated proteins and inducible nitric oxide synthase (iNOS), and activation of phosphatidylinositol
Suppression of transforming growth factor-beta-induced apoptosis through a phosphatidylinositol 3-kinase/Akt-dependent pathway.
Chen RH, Su YH, Chuang RL, Chang TY. Chen RH, et al. Oncogene. 1998 Oct 15;17(15):1959-68. doi: 10.1038/sj.onc.1202111. Oncogene. 1998. PMID: 9788439 Free article.
The Ras/mitogen-activated protein kinase (MAP kinase) and the phosphatidylinositol 3-kinase (PI 3-kinase)/Akt pathways are both activated upon insulin stimulation and can protect against apoptosis under …
The Ras/mitogen-activated protein kinase (MAP kinase) and the phosphatidylinositol 3-kinase (PI …
Insulin-like growth factor 1 inhibits extracellular signal-regulated kinase to promote neuronal survival via the phosphatidylinositol 3-kinase/protein kinase A/c-Raf pathway.
Subramaniam S, Shahani N, Strelau J, Laliberté C, Brandt R, Kaplan D, Unsicker K. Subramaniam S, et al. J Neurosci. 2005 Mar 16;25(11):2838-52. doi: 10.1523/JNEUROSCI.5060-04.2005. J Neurosci. 2005. PMID: 15772344 Free PMC article.
Phosphatidylinositol 3-kinase (PI3K) or PKA inhibitors, but not a specific Akt inhibitor, abrogated PKA signaling. ...Activation of caspase-3 promoted DNA damage. Its inhibition by IGF-1 was both PI3K and Akt dependent but PKA inde
Phosphatidylinositol 3-kinase (PI3K) or PKA inhibitors, but not a specific Akt inhibitor, abrogated PKA signaling. ...
Insulin can block apoptosis by decreasing oxidative stress via phosphatidylinositol 3-kinase- and extracellular signal-regulated protein kinase-dependent signaling pathways in HepG2 cells.
Kang S, Song J, Kang H, Kim S, Lee Y, Park D. Kang S, et al. Eur J Endocrinol. 2003 Jan;148(1):147-55. doi: 10.1530/eje.0.1480147. Eur J Endocrinol. 2003. PMID: 12534368
Western blot analysis was performed to identify signaling molecules activated in response to insulin. RESULTS: Serum starvation resulted in a marked accumulation of ROS, activation of caspase-3, and subsequent apoptotic cell death which were, in …
Western blot analysis was performed to identify signaling molecules activated in response to insulin. RESULTS: Serum starvatio …
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