Kahn SE - Search Results

1

Factors affecting the decline in incidence of diabetes in the Diabetes Prevention Program Outcomes Study (DPPOS).

Diabetes Prevention Program (DPP) Research Group; Hamman RF, Horton E, Barrett-Connor E, Bray GA, Christophi CA, Crandall J, Florez JC, Fowler S, Goldberg R, Kahn SE, Knowler WC, Lachin JM, Murphy MB, Venditti E. Diabetes Prevention Program (DPP) Research Group, et al. Among authors: kahn se. Diabetes. 2015 Mar;64(3):989-98. doi: 10.2337/db14-0333. Epub 2014 Oct 2. Diabetes. 2015. PMID: 25277389 Free PMC article.

2

Nonprogression of subclinical beta-cell dysfunction among first-degree relatives of IDDM patients. 5-yr follow-up of the Seattle Family Study.

McCulloch DK, Klaff LJ, Kahn SE, Schoenfeld SL, Greenbaum CJ, Mauseth RS, Benson EA, Nepom GT, Shewey L, Palmer JP. McCulloch DK, et al. Among authors: kahn se. Diabetes. 1990 May;39(5):549-56. doi: 10.2337/diab.39.5.549. Diabetes. 1990. PMID: 2185106

3

Disproportionately elevated proinsulin in Pima Indians with noninsulin-dependent diabetes mellitus.

Saad MF, Kahn SE, Nelson RG, Pettitt DJ, Knowler WC, Schwartz MW, Kowalyk S, Bennett PH, Porte D Jr. Saad MF, et al. Among authors: kahn se. J Clin Endocrinol Metab. 1990 May;70(5):1247-53. doi: 10.1210/jcem-70-5-1247. J Clin Endocrinol Metab. 1990. PMID: 2186054

4

Hyperproinsulinemia and amyloid in NIDDM. Clues to etiology of islet beta-cell dysfunction?

Porte D Jr, Kahn SE. Porte D Jr, et al. Among authors: kahn se. Diabetes. 1989 Nov;38(11):1333-6. doi: 10.2337/diab.38.11.1333. Diabetes. 1989. PMID: 2695369 Review.

5

Proinsulin as a marker for the development of NIDDM in Japanese-American men.

Kahn SE, Leonetti DL, Prigeon RL, Boyko EJ, Bergstrom RW, Fujimoto WY. Kahn SE, et al. Diabetes. 1995 Feb;44(2):173-9. doi: 10.2337/diab.44.2.173. Diabetes. 1995. PMID: 7859937

6

Glucokinase gene variations in Japanese-Americans with a family history of NIDDM.

Stone LM, Kahn SE, Deeb SS, Fujimoto WY, Porte D Jr. Stone LM, et al. Among authors: kahn se. Diabetes Care. 1994 Dec;17(12):1480-3. doi: 10.2337/diacare.17.12.1480. Diabetes Care. 1994. PMID: 7882822

7

Nicotinamide's effects on glucose metabolism in subjects at risk for IDDM.

Greenbaum CJ, Kahn SE, Palmer JP. Greenbaum CJ, et al. Among authors: kahn se. Diabetes. 1996 Nov;45(11):1631-4. doi: 10.2337/diab.45.11.1631. Diabetes. 1996. PMID: 8866571 Clinical Trial.

8

Release of incompletely processed proinsulin is the cause of the disproportionate proinsulinemia of NIDDM.

Kahn SE, Halban PA. Kahn SE, et al. Diabetes. 1997 Nov;46(11):1725-32. doi: 10.2337/diab.46.11.1725. Diabetes. 1997. PMID: 9356018

9

Reduced amylin release is a characteristic of impaired glucose tolerance and type 2 diabetes in Japanese Americans.

Kahn SE, Verchere CB, Andrikopoulos S, Asberry PJ, Leonetti DL, Wahl PW, Boyko EJ, Schwartz RS, Newell-Morris L, Fujimoto WY. Kahn SE, et al. Diabetes. 1998 Apr;47(4):640-5. doi: 10.2337/diabetes.47.4.640. Diabetes. 1998. PMID: 9568698

10

Relationship of beta-cell function and autoantibodies to progression and nonprogression of subclinical type 1 diabetes: follow-up of the Seattle Family Study.

Greenbaum CJ, Sears KL, Kahn SE, Palmer JP. Greenbaum CJ, et al. Among authors: kahn se. Diabetes. 1999 Jan;48(1):170-5. doi: 10.2337/diabetes.48.1.170. Diabetes. 1999. PMID: 9892239

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