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Platelet-binding domains in 2 fibrinogen-binding proteins of Staphylococcus aureus identified by phage display.
Heilmann C, Herrmann M, Kehrel BE, Peters G. Heilmann C, et al. J Infect Dis. 2002 Jul 1;186(1):32-9. doi: 10.1086/341081. Epub 2002 Jun 10. J Infect Dis. 2002. PMID: 12089659
The surface-associated fraction of the C-terminal domain of coagulase or the N-terminal domain of Efb may be involved in bacterial adherence to immobilized platelets, and fibrinogen may act as a bridging molecule in that interaction....
The surface-associated fraction of the C-terminal domain of coagulase or the N-terminal domain of Efb may be involved in bacterial ad …
Human platelets attenuate Aspergillus species via granule-dependent mechanisms.
Perkhofer S, Kehrel BE, Dierich MP, Donnelly JP, Nussbaumer W, Hofmann J, von Eiff C, Lass-Flörl C. Perkhofer S, et al. J Infect Dis. 2008 Oct 15;198(8):1243-6. doi: 10.1086/591458. J Infect Dis. 2008. PMID: 18752432 Free PMC article.
Potential antifungal effects of human platelets against zygomycetes in vitro.
Perkhofer S, Kainzner B, Kehrel BE, Dierich MP, Nussbaumer W, Lass-Flörl C. Perkhofer S, et al. J Infect Dis. 2009 Oct 1;200(7):1176-9. doi: 10.1086/605607. J Infect Dis. 2009. PMID: 19698079 Free PMC article.
Glycoprotein VI is a major collagen receptor for platelet activation: it recognizes the platelet-activating quaternary structure of collagen, whereas CD36, glycoprotein IIb/IIIa, and von Willebrand factor do not.
Kehrel B, Wierwille S, Clemetson KJ, Anders O, Steiner M, Knight CG, Farndale RW, Okuma M, Barnes MJ. Kehrel B, et al. Blood. 1998 Jan 15;91(2):491-9. Blood. 1998. PMID: 9427702
Platelet-collagen interactions.
Kehrel B. Kehrel B. Semin Thromb Hemost. 1995;21(2):123-9. doi: 10.1055/s-2007-1000386. Semin Thromb Hemost. 1995. PMID: 7660134 Review.
Interaction of von Willebrand factor with Staphylococcus aureus.
Herrmann M, Hartleib J, Kehrel B, Montgomery RR, Sixma JJ, Peters G. Herrmann M, et al. J Infect Dis. 1997 Oct;176(4):984-91. doi: 10.1086/516502. J Infect Dis. 1997. PMID: 9333157
HDL3-mediated inhibition of thrombin-induced platelet aggregation and fibrinogen binding occurs via decreased production of phosphoinositide-derived second messengers 1,2-diacylglycerol and inositol 1,4,5-tris-phosphate.
Nofer JR, Walter M, Kehrel B, Wierwille S, Tepel M, Seedorf U, Assmann G. Nofer JR, et al. Arterioscler Thromb Vasc Biol. 1998 Jun;18(6):861-9. doi: 10.1161/01.atv.18.6.861. Arterioscler Thromb Vasc Biol. 1998. PMID: 9633924
Furthermore, they could be blocked by calphostin C and bis-indolylmaleimide, 2 highly selective and structurally unrelated protein kinase C inhibitors. ...Protein kinase C may be involved in this process....
Furthermore, they could be blocked by calphostin C and bis-indolylmaleimide, 2 highly selective and structurally unrelated protein ki …
Human platelet CD36 (GPIIIb, GPIV) binds to cholesteryl-hemisuccinate and can be purified by a simple two-step method making use of this property.
Kronenberg A, Grahl H, Kehrel B. Kronenberg A, et al. Thromb Haemost. 1998 May;79(5):1021-4. Thromb Haemost. 1998. PMID: 9609240
This binding may be of physiologic relevance since CD36 has a long extracellular hydrophobic region at its N-terminus and belongs to a family of proteins involved in lipid metabolism. ...
This binding may be of physiologic relevance since CD36 has a long extracellular hydrophobic region at its N-terminus and belongs to …
Pneumococcal association to platelets is mediated by soluble fibrin and supported by thrombospondin-1.
Niemann S, Kehrel BE, Heilmann C, Rennemeier C, Peters G, Hammerschmidt S. Niemann S, et al. Thromb Haemost. 2009 Oct;102(4):735-42. doi: 10.1160/TH09-01-0049. Thromb Haemost. 2009. PMID: 19806260
In addition to be a platelet agonist, thrombin cleaves fibrinogen, which results in the generation of fibrin. The simultaneous formation of fibrin and activation of platelets was shown to be a prerequisite for a high number of platelet-pneumococci associates. ...
In addition to be a platelet agonist, thrombin cleaves fibrinogen, which results in the generation of fibrin. The simultaneous format …
Role for the fibrinogen-binding proteins coagulase and Efb in the Staphylococcus aureus-Candida interaction.
Fehrmann C, Jurk K, Bertling A, Seidel G, Fegeler W, Kehrel BE, Peters G, Becker K, Heilmann C. Fehrmann C, et al. Int J Med Microbiol. 2013 Jul;303(5):230-8. doi: 10.1016/j.ijmm.2013.02.011. Epub 2013 Mar 13. Int J Med Microbiol. 2013. PMID: 23684234
In conclusion, the inhibition of phagocytosis of Candida cells by coagulase and Efb via two distinct mechanisms suggests that S. aureus might be beneficial for Candida to persist as it helps Candida to circumvent the host immune system....
In conclusion, the inhibition of phagocytosis of Candida cells by coagulase and Efb via two distinct mechanisms suggests that S. aureus migh …
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