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Intestinal permeation and gastrointestinal disease.
DeMeo MT, Mutlu EA, Keshavarzian A, Tobin MC. DeMeo MT, et al. Among authors: keshavarzian a. J Clin Gastroenterol. 2002 Apr;34(4):385-96. doi: 10.1097/00004836-200204000-00003. J Clin Gastroenterol. 2002. PMID: 11907349 Review.
A variety of specific and nonspecific mechanisms are in operation to establish the host barrier; these include luminal mechanisms and digestive enzymes, the epithelial cells together with tight junctions in between them, and the gut immune system. ...An overview of the evi
A variety of specific and nonspecific mechanisms are in operation to establish the host barrier; these include luminal mechanisms and
Gastrointestinal involvement in insulin-requiring diabetes mellitus.
Keshavarzian A, Iber FL. Keshavarzian A, et al. J Clin Gastroenterol. 1987 Dec;9(6):685-92. doi: 10.1097/00004836-198712000-00014. J Clin Gastroenterol. 1987. PMID: 3443733
Gastrointestinal (GI) dysfunction in diabetes mellitus has never been evaluated systematically in all parts of the digestive system in a group of diabetics. ...
Gastrointestinal (GI) dysfunction in diabetes mellitus has never been evaluated systematically in all parts of the digestive system in a
Hepatic abscesses associated with Crohn's disease detected by 111Indium leucocyte scanning.
Saverymuttu SH, Keshavarzian A, Gibson R, Chadwick VS, Hodgson HJ. Saverymuttu SH, et al. Among authors: keshavarzian a. J Clin Gastroenterol. 1985 Jun;7(3):273-6. doi: 10.1097/00004836-198506000-00018. J Clin Gastroenterol. 1985. PMID: 4020088
Reactive oxygen and nitrogen metabolites as mediators of secretory diarrhea.
Gaginella TS, Kachur JF, Tamai H, Keshavarzian A. Gaginella TS, et al. Among authors: keshavarzian a. Gastroenterology. 1995 Dec;109(6):2019-28. doi: 10.1016/0016-5085(95)90772-6. Gastroenterology. 1995. PMID: 7498670 Review. No abstract available.
Increased gastrointestinal symptoms in chronic alcoholics.
Fields JZ, Turk A, Durkin M, Ravi NV, Keshavarzian A. Fields JZ, et al. Among authors: keshavarzian a. Am J Gastroenterol. 1994 Mar;89(3):382-6. Am J Gastroenterol. 1994. PMID: 8122650
A standardized questionnaire was administered on two separate occasions to all subjects, and 14 GI symptoms were evaluated for three periods (during active drinking, early withdrawal, and sobriety). Symptom severity was assessed with a visual analog scale (1-10). Bo
A standardized questionnaire was administered on two separate occasions to all subjects, and 14 GI symptoms were evaluated for three
Erythromycin accelerates gastric emptying of indigestible solids and transpyloric migration of the tip of an enteral feeding tube in fasting and fed states.
Keshavarzian A, Isaac RM. Keshavarzian A, et al. Am J Gastroenterol. 1993 Feb;88(2):193-7. Am J Gastroenterol. 1993. PMID: 8424419 Clinical Trial.
For fasting studies, the subjects remained fasted; in the fed studies, the subjects were fed a cheeseburger and fries after placement of the feeding tube. ...Hence, erythromycin can be beneficial when placement of a feeding tube in the small intestine is clinically …
For fasting studies, the subjects remained fasted; in the fed studies, the subjects were fed a cheeseburger and fries after placement …
MPTP-induced duodenal ulcers in rat. Prevention by reuptake blockers for serotonin and norepinephrine, but not dopamine.
Keshavarzian A, Wibowo A, Gordon JH, Fields JZ. Keshavarzian A, et al. Gastroenterology. 1990 Mar;98(3):554-60. Gastroenterology. 1990. PMID: 2298362
Duodenal ulcers were produced in rats by 12 subcutaneous injections of a neurotoxin, MPTP, over 4 days. At an MPTP dose of 20 mg.kg. injection, duodenal ulcers developed in 91% (43 of 47) of animals with low mortality. ...MAO-B inhibitors (pargyline [55%], deprenyl [43%]) …
Duodenal ulcers were produced in rats by 12 subcutaneous injections of a neurotoxin, MPTP, over 4 days. At an MPTP dose of 20 mg.kg. …
Gastric emptying in patients with severe reflux esophagitis.
Keshavarzian A, Bushnell DL, Sontag S, Yegelwel EJ, Smid K. Keshavarzian A, et al. Am J Gastroenterol. 1991 Jun;86(6):738-42. Am J Gastroenterol. 1991. PMID: 2038997
There have been reports that delayed gastric emptying is a contributing factor in some patients. To determine whether delayed gastric emptying plays a role in the genesis of GER, we correlated gastric-emptying measures obtained by scintigraphic techniques with the d …
There have been reports that delayed gastric emptying is a contributing factor in some patients. To determine whether delayed gastric …
Excessive production of reactive oxygen metabolites by inflamed colon: analysis by chemiluminescence probe.
Keshavarzian A, Sedghi S, Kanofsky J, List T, Robinson C, Ibrahim C, Winship D. Keshavarzian A, et al. Gastroenterology. 1992 Jul;103(1):177-85. doi: 10.1016/0016-5085(92)91111-g. Gastroenterology. 1992. PMID: 1612325
Addition of catalase, an H2O2 scavenger, or azide, a myeloperoxidase inhibitor, into the media significantly decreased chemiluminescence from inflamed mucosal scrapings. Indomethacin, an antioxidant cyclo-oxygenase inhibitor, also decreased chemiluminescence, but MK-866, …
Addition of catalase, an H2O2 scavenger, or azide, a myeloperoxidase inhibitor, into the media significantly decreased chemiluminesce …
Beneficial effect of a bile acid resin binder on enteral feeding induced diarrhea.
DeMeo M, Kolli S, Keshavarzian A, Borton M, Al-Hosni M, Dyavanapalli M, Shiau A, Tu N, Frommel T, Zarling E, Goris G, Shawaryn G, Mobarhan S. DeMeo M, et al. Among authors: keshavarzian a. Am J Gastroenterol. 1998 Jun;93(6):967-71. doi: 10.1111/j.1572-0241.1998.00289.x. Am J Gastroenterol. 1998. PMID: 9647030 Clinical Trial.
Subsequent enteral feeding then causes a relative luminal excess of bile acids, which leads to choleretic diarrhea. Hence, diarrhea induced by enteral feeding should improve with the use of a bile acid binding agent, such as Colestid Granules. METHODS: We evaluated …
Subsequent enteral feeding then causes a relative luminal excess of bile acids, which leads to choleretic diarrhea. Hence, diarrhea i …
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