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Serotonergic stimulation of corticotropin-releasing hormone and pro-opiomelanocortin gene expression.
Jørgensen H, Knigge U, Kjaer A, Møller M, Warberg J. Jørgensen H, et al. Among authors: kjaer a. J Neuroendocrinol. 2002 Oct;14(10):788-95. doi: 10.1046/j.1365-2826.2002.00839.x. J Neuroendocrinol. 2002. PMID: 12372003
Central administration of 5-HT agonists specific to 5-HT1A, 5-HT1B, 5-HT2A or 5-HT2C receptors increased CRH mRNA in the PVN by 15-50%, POMC mRNA in the anterior pituitary by 15-27% and ACTH secretion three- to five-fold, whereas a specific 5-HT3 agonist had no effect. Sys …
Central administration of 5-HT agonists specific to 5-HT1A, 5-HT1B, 5-HT2A or 5-HT2C receptors increased CRH mRNA in the PVN by 15-50%, POMC …
Histamine- and stress-induced secretion of ACTH and beta-endorphin: involvement of corticotropin-releasing hormone and vasopressin.
Kjaer A, Knigge U, Bach FW, Warberg J. Kjaer A, et al. Neuroendocrinology. 1992 Sep;56(3):419-28. doi: 10.1159/000126258. Neuroendocrinology. 1992. PMID: 1331840
Intracerebroventricular infusion of HA (34-540 nmol) increased the plasma levels of ACTH and beta-END immunoreactivity (beta-ENDir) in a dose-dependent manner. ...The posterior pituitary extract caused a dose-dependent rise in plasma beta-ENDir which might be due to …
Intracerebroventricular infusion of HA (34-540 nmol) increased the plasma levels of ACTH and beta-END immunoreactivity (beta-ENDir) in a
Histamine H1 and H2 receptor activation stimulates ACTH and beta-endorphin secretion by increasing corticotropin-releasing hormone in the hypophyseal portal blood.
Kjaer A, Knigge U, Plotsky PM, Bach FW, Warberg J. Kjaer A, et al. Neuroendocrinology. 1992 Dec;56(6):851-5. doi: 10.1159/000126316. Neuroendocrinology. 1992. PMID: 1369594

Pretreatment with a specific CRH antiserum (abCRH) inhibited the responses by 50 and 70%, respectively (p < 0.01). Intracerebroventricular administration of 2-TEA or 4-MeHA increased the beta-END immunoreactivity in peripheral plasma 3- or 2-fold, respectively (p < 0

Pretreatment with a specific CRH antiserum (abCRH) inhibited the responses by 50 and 70%, respectively (p < 0.01). Intracerebroven

Mediation of the stress-induced prolactin release by hypothalamic histaminergic neurons and the possible involvement of vasopressin in this response.
Kjaer A, Knigge U, Olsen L, Vilhardt H, Warberg J. Kjaer A, et al. Endocrinology. 1991 Jan;128(1):103-10. doi: 10.1210/endo-128-1-103. Endocrinology. 1991. PMID: 1986913
In contrast, pretreatment with a specific oxytocin (OT) antagonist (50 nmol) had no effect on the HA- or stress-induced PRL release. ...The AVP antiserum or the AVP antagonist almost prevented the PRL release induced by iv infusion of 800 pmol AVP or a posterior pit …
In contrast, pretreatment with a specific oxytocin (OT) antagonist (50 nmol) had no effect on the HA- or stress-induced PRL release. …
The prolactin releasing effect of histamine is unrelated to its vascular action.
Kjaer A, Knigge U, Warberg J. Kjaer A, et al. Acta Endocrinol (Copenh). 1990 Jan;122(1):49-54. doi: 10.1530/acta.0.1220049. Acta Endocrinol (Copenh). 1990. PMID: 2305605
We investigated in male rats whether the prolactin releasing effect of systemically infused histamine might be attributed to a vasodilatatory action of the compound. ...Since the H1 receptor seemed to be the predominant mediator of the histamine-induced hypotension and PRL …
We investigated in male rats whether the prolactin releasing effect of systemically infused histamine might be attributed to a vasodi …
Involvement of oxytocin in histamine- and stress-induced ACTH and prolactin secretion.
Kjaer A, Knigge U, Warberg J. Kjaer A, et al. Neuroendocrinology. 1995 Jun;61(6):704-13. doi: 10.1159/000126898. Neuroendocrinology. 1995. PMID: 7659194
The neurohypophysial peptide oxytocin (OT) is released in response to different stressors and has been suggested as a 'stress hormone'. In addition, we have recently shown that centrally administered histamine (HA), which is a mediator of stress-induced release of A …
The neurohypophysial peptide oxytocin (OT) is released in response to different stressors and has been suggested as a 'stress hormone …
Dehydration stimulates hypothalamic gene expression of histamine synthesis enzyme: importance for neuroendocrine regulation of vasopressin and oxytocin secretion.
Kjaer A, Larsen PJ, Knigge U, Warberg J. Kjaer A, et al. Endocrinology. 1995 May;136(5):2189-97. doi: 10.1210/endo.136.5.7720668. Endocrinology. 1995. PMID: 7720668
Because histamine (HA), which serves as a central neurotransmitter, releases AVP and OT from the neurohypophysis when administered centrally, we investigated the possible role of HA in dehydration-induced AVP and OT secretion. ...
Because histamine (HA), which serves as a central neurotransmitter, releases AVP and OT from the neurohypophysis when administered ce …
Histamine- and stress-induced prolactin secretion: importance of vasopressin V1- and V2-receptors.
Kjaer A, Knigge U, Warberg J. Kjaer A, et al. Eur J Endocrinol. 1994 Oct;131(4):391-7. doi: 10.1530/eje.0.1310391. Eur J Endocrinol. 1994. PMID: 7921229
Pretreatment with the selective V1-receptor antagonists [1-(p-t-butyl-beta-mercapto-beta, beta-cyclopentamethylene propionic acid)-2-(O-methyl)tyrosine-8-D-arginine]vasopressin or [1-(beta-mercapto-beta, beta-cyclopentamethylene propionic acid)-2-(O-methyl)tyrosine-8-arginine]vas …
Pretreatment with the selective V1-receptor antagonists [1-(p-t-butyl-beta-mercapto-beta, beta-cyclopentamethylene propionic acid)-2-(O-meth …
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