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1825 3
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1829 1
1830 1
1832 1
1834 2
1841 2
1843 4
1844 1
1845 3
1846 9
1847 6
1848 2
1849 3
1850 4
1851 3
1852 4
1853 5
1854 5
1855 8
1856 2
1857 5
1858 2
1859 2
1860 2
1861 2
1863 2
1864 5
1866 9
1867 3
1868 3
1869 3
1870 4
1871 2
1872 10
1873 4
1874 8
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1878 8
1879 3
1880 7
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1882 11
1883 10
1884 7
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1893 5
1894 11
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1897 5
1898 10
1899 4
1900 4
1901 9
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1903 3
1904 5
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1907 7
1908 24
1909 28
1910 32
1911 24
1912 26
1913 39
1914 26
1915 25
1916 21
1917 15
1918 24
1919 16
1920 27
1921 13
1922 29
1923 27
1924 9
1925 16
1926 16
1927 33
1928 25
1929 16
1930 24
1931 22
1932 21
1933 19
1934 16
1935 11
1936 12
1937 16
1938 15
1939 14
1940 9
1941 3
1942 5
1943 4
1944 14
1945 46
1946 117
1947 136
1948 172
1949 136
1950 221
1951 255
1952 235
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1954 239
1955 270
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1974 1002
1975 3437
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877,404 results

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Page 1
Left Ventricular Diastolic Function: Understanding Pathophysiology, Diagnosis, and Prognosis With Echocardiography.
Nagueh SF. Nagueh SF. JACC Cardiovasc Imaging. 2020 Jan;13(1 Pt 2):228-244. doi: 10.1016/j.jcmg.2018.10.038. Epub 2019 Apr 12. JACC Cardiovasc Imaging. 2020. PMID: 30982669 Free article. Review.
Left ventricular diastolic function plays an important role in determining left ventricular filling and stroke volume. ...Using echocardiography, it is possible to diagnose the presence of diastolic dysfunction and the pathophysiologic mechanisms involved as they af
Left ventricular diastolic function plays an important role in determining left ventricular filling and stroke volume. ...Usin
Left ventricular hypertrophy and hypertension.
Yildiz M, Oktay AA, Stewart MH, Milani RV, Ventura HO, Lavie CJ. Yildiz M, et al. Prog Cardiovasc Dis. 2020 Jan-Feb;63(1):10-21. doi: 10.1016/j.pcad.2019.11.009. Epub 2019 Nov 21. Prog Cardiovasc Dis. 2020. PMID: 31759953 Review.
Hypertension (HTN) is a major modifiable risk factor for cardiovascular disease (CVD) morbidity and mortality. The left ventricle (LV) is a primary target for HTN end-organ damage. In addition to being a marker of HTN, LV geometrical changes: concentric remodeling, concent …
Hypertension (HTN) is a major modifiable risk factor for cardiovascular disease (CVD) morbidity and mortality. The left ventricle (LV …
Left atrium as key player and essential biomarker in heart failure.
Inoue K, Smiseth OA. Inoue K, et al. J Cardiol. 2025 Jan;85(1):8-16. doi: 10.1016/j.jjcc.2024.07.006. Epub 2024 Jul 29. J Cardiol. 2025. PMID: 39084316 Free article. Review.
This article reviews roles of the left atrium as regulator of left ventricular filling, as compensatory reserve in left ventricular dysfunction and as diagnostic marker in patients with cardiovascular disorders. ...Recommendations for how to apply left
This article reviews roles of the left atrium as regulator of left ventricular filling, as compensatory reserve in left
Left ventricular hypertrabeculation/noncompaction.
Stöllberger C, Finsterer J. Stöllberger C, et al. J Am Soc Echocardiogr. 2004 Jan;17(1):91-100. doi: 10.1016/S0894-7317(03)00514-5. J Am Soc Echocardiogr. 2004. PMID: 14712196 Review.
In normal human hearts the left ventricle (LV) has up to 3 prominent trabeculations and is, thus, less trabeculated than the right ventricle. ...
In normal human hearts the left ventricle (LV) has up to 3 prominent trabeculations and is, thus, less trabeculated than the right ve …
Prognostic Implications of Left Ventricular Hypertrophy.
Stewart MH, Lavie CJ, Shah S, Englert J, Gilliland Y, Qamruddin S, Dinshaw H, Cash M, Ventura H, Milani R. Stewart MH, et al. Prog Cardiovasc Dis. 2018 Nov-Dec;61(5-6):446-455. doi: 10.1016/j.pcad.2018.11.002. Epub 2018 Nov 6. Prog Cardiovasc Dis. 2018. PMID: 30408469 Review.
Left ventricular hypertrophy (LVH) was one of the earliest studied echocardiographic characteristics of the left ventricle. ...
Left ventricular hypertrophy (LVH) was one of the earliest studied echocardiographic characteristics of the left ventricle. ..
Four left ventricular apical abnormalities.
Conti CR. Conti CR. Clin Cardiol. 2006 Feb;29(2):50-1. doi: 10.1002/clc.4960290202. Clin Cardiol. 2006. PMID: 16506637 Free PMC article. Review.
This editorial reviews the clinical presentation, electrocardiographic manifestations, appearance of the left ventricle by imaging techniques, and the state of the coronary arteries in four left ventricular apical abnormalities (Table I). Three of these conditions, …
This editorial reviews the clinical presentation, electrocardiographic manifestations, appearance of the left ventricle by imaging te …
Implications of left ventricular remodeling in hypertrophic cardiomyopathy.
Maron BJ, Spirito P. Maron BJ, et al. Am J Cardiol. 1998 Jun 1;81(11):1339-44. doi: 10.1016/s0002-9149(98)00164-7. Am J Cardiol. 1998. PMID: 9631972 Review.
Left ventricular remodeling occurs spontaneously among patients with hypertrophic cardiomyopathy in several ways: (1) wall thickening in children; (2) wall thinning associated with cavity enlargement in midlife; and possibly (3) a very gradual wall thinning process occurri
Left ventricular remodeling occurs spontaneously among patients with hypertrophic cardiomyopathy in several ways: (1) wall thickening
From Left Ventricular Hypertrophy to Dysfunction and Failure.
Lazzeroni D, Rimoldi O, Camici PG. Lazzeroni D, et al. Circ J. 2016;80(3):555-64. doi: 10.1253/circj.CJ-16-0062. Epub 2016 Feb 5. Circ J. 2016. PMID: 26853555 Free article. Review.
Left ventricular hypertrophy (LVH) is growth in left ventricular mass caused by increased cardiomyocyte size. ...
Left ventricular hypertrophy (LVH) is growth in left ventricular mass caused by increased cardiomyocyte size. ...
Left ventricular remodelling after acute myocardial infarction--solved and unsolved issues.
Bassand JP. Bassand JP. Eur Heart J. 1995 Aug;16 Suppl I:58-63. doi: 10.1093/eurheartj/16.suppl_i.58. Eur Heart J. 1995. PMID: 8829958 Review.
Left ventricular remodelling after acute myocardial infarction, through long-term left ventricular chamber dilatation and increased wall stress can result in alteration of the contractile properties of the non-infarct zone, impairment of the systolic and diastolic p
Left ventricular remodelling after acute myocardial infarction, through long-term left ventricular chamber dilatation and incr
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