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Neuromuscular junction immaturity and muscle atrophy are hallmarks of the ColQ-deficient mouse, a model of congenital myasthenic syndrome with acetylcholinesterase deficiency.
Sigoillot SM, Bourgeois F, Karmouch J, Molgó J, Dobbertin A, Chevalier C, Houlgatte R, Léger J, Legay C. Sigoillot SM, et al. Among authors: legay c. FASEB J. 2016 Jun;30(6):2382-99. doi: 10.1096/fj.201500162. Epub 2016 Mar 18. FASEB J. 2016. PMID: 26993635
., Bourgeois, F., Karmouch, J., Molgo, J., Dobbertin, A., Chevalier, C., Houlgatte, R., Leger, J., Legay, C. Neuromuscular junction immaturity and muscle atrophy are hallmarks of the ColQ-deficient mouse, a model of congenital myasthenic syndrome with acetylc …
., Bourgeois, F., Karmouch, J., Molgo, J., Dobbertin, A., Chevalier, C., Houlgatte, R., Leger, J., Legay, C. Neuromuscu …
Cholinesterases regulation in the absence of ColQ.
Sigoillot SM, Bourgeois F, Legay C. Sigoillot SM, et al. Among authors: legay c. Chem Biol Interact. 2010 Sep 6;187(1-3):84-9. doi: 10.1016/j.cbi.2010.02.007. Epub 2010 Feb 11. Chem Biol Interact. 2010. PMID: 20153305
Developmental consequences of the ColQ/MuSK interactions.
Karmouch J, Dobbertin A, Sigoillot S, Legay C. Karmouch J, et al. Among authors: legay c. Chem Biol Interact. 2013 Mar 25;203(1):287-91. doi: 10.1016/j.cbi.2012.10.006. Epub 2012 Oct 23. Chem Biol Interact. 2013. PMID: 23089045 Review.
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