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Neuromuscular junction immaturity and muscle atrophy are hallmarks of the ColQ-deficient mouse, a model of congenital myasthenic syndrome with acetylcholinesterase deficiency.
Sigoillot SM, Bourgeois F, Karmouch J, Molgó J, Dobbertin A, Chevalier C, Houlgatte R, Léger J, Legay C. Sigoillot SM, et al. Among authors: legay c. FASEB J. 2016 Jun;30(6):2382-99. doi: 10.1096/fj.201500162. Epub 2016 Mar 18. FASEB J. 2016. PMID: 26993635
., Bourgeois, F., Karmouch, J., Molgo, J., Dobbertin, A., Chevalier, C., Houlgatte, R., Leger, J., Legay, C. Neuromuscular junction immaturity and muscle atrophy are hallmarks of the ColQ-deficient mouse, a model of congenital myasthenic syndrome with acetylc …
., Bourgeois, F., Karmouch, J., Molgo, J., Dobbertin, A., Chevalier, C., Houlgatte, R., Leger, J., Legay, C. Neuromuscu …
Why so many forms of acetylcholinesterase?
Legay C. Legay C. Microsc Res Tech. 2000 Apr 1;49(1):56-72. doi: 10.1002/(SICI)1097-0029(20000401)49:1<56::AID-JEMT7>3.0.CO;2-R. Microsc Res Tech. 2000. PMID: 10757879 Review.
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