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Effect of aldosterone on BK channel expression in mammalian cortical collecting duct.
Estilo G, Liu W, Pastor-Soler N, Mitchell P, Carattino MD, Kleyman TR, Satlin LM. Estilo G, et al. Among authors: liu w. Am J Physiol Renal Physiol. 2008 Sep;295(3):F780-8. doi: 10.1152/ajprenal.00002.2008. Epub 2008 Jun 25. Am J Physiol Renal Physiol. 2008. PMID: 18579708 Free PMC article.
Dietary K(+) loading for 10-14 days leads to an increase in BK channel mRNA abundance, enhanced flow-stimulated K(+) secretion in microperfused CCDs, and a redistribution of immunodetectable channels from an intracellular pool to the apical membrane (Najjar F, Zhou H, Morimoto T, …
Dietary K(+) loading for 10-14 days leads to an increase in BK channel mRNA abundance, enhanced flow-stimulated K(+) secretion in microperfu …
Role of NKCC in BK channel-mediated net K⁺ secretion in the CCD.
Liu W, Schreck C, Coleman RA, Wade JB, Hernandez Y, Zavilowitz B, Warth R, Kleyman TR, Satlin LM. Liu W, et al. Am J Physiol Renal Physiol. 2011 Nov;301(5):F1088-97. doi: 10.1152/ajprenal.00347.2011. Epub 2011 Aug 3. Am J Physiol Renal Physiol. 2011. PMID: 21816753 Free PMC article.
Angiotensin II type 2 receptor regulates ROMK-like K⁺ channel activity in the renal cortical collecting duct during high dietary K⁺ adaptation.
Wei Y, Liao Y, Zavilowitz B, Ren J, Liu W, Chan P, Rohatgi R, Estilo G, Jackson EK, Wang WH, Satlin LM. Wei Y, et al. Among authors: liu w. Am J Physiol Renal Physiol. 2014 Oct 1;307(7):F833-43. doi: 10.1152/ajprenal.00141.2014. Epub 2014 Aug 6. Am J Physiol Renal Physiol. 2014. PMID: 25100281 Free PMC article.
Mechanoregulation of intracellular Ca2+ concentration is attenuated in collecting duct of monocilium-impaired orpk mice.
Liu W, Murcia NS, Duan Y, Weinbaum S, Yoder BK, Schwiebert E, Satlin LM. Liu W, et al. Am J Physiol Renal Physiol. 2005 Nov;289(5):F978-88. doi: 10.1152/ajprenal.00260.2004. Epub 2005 Jun 21. Am J Physiol Renal Physiol. 2005. PMID: 15972389 Free article.
New findings implicate defects in structure/function of primary cilia as central to the development of polycystic kidney disease. Our group (Liu W, Xu S, Woda C, Kim P, Weinbaum S, and Satlin LM, Am J Physiol Renal Physiol 285: F998-F1012, 2003) recently reported th …
New findings implicate defects in structure/function of primary cilia as central to the development of polycystic kidney disease. Our group …
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