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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1971 1
1974 1
1978 1
1979 1
1980 1
1982 1
1983 1
1985 2
1986 3
1987 1
1988 1
1989 4
1990 4
1991 3
1992 2
1993 1
1995 4
1996 1
1997 8
1998 2
1999 9
2000 5
2001 9
2002 7
2003 8
2004 14
2005 11
2006 16
2007 21
2008 25
2009 29
2010 29
2011 34
2012 21
2013 28
2014 27
2015 43
2016 30
2017 33
2018 32
2019 31
2020 27
2021 33
2022 21
2023 22
2024 37
2025 39
2026 32

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634 results

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Page 1
Showing results for loss of adipose fatty acid oxidation does not potentiate obesity at hermoneutrality. Cell Rep. 14(6):1308-16
Your search for Loss of Adipose Fatty Acid Oxidation Does Not Potentiate Obesity at Thermoneutrality. Cell Rep. 14(6):1308-16 retrieved no results
Understanding the Physiology of FGF21.
Fisher FM, Maratos-Flier E. Fisher FM, et al. Annu Rev Physiol. 2016;78:223-41. doi: 10.1146/annurev-physiol-021115-105339. Epub 2015 Nov 19. Annu Rev Physiol. 2016. PMID: 26654352 Review.
In the liver, FGF21 plays an important role in the regulation of fatty acid oxidation both in the fasted state and in mice consuming a high-fat, low-carbohydrate ketogenic diet. FGF21 also regulates fatty acid metabolism in mice consuming a diet …
In the liver, FGF21 plays an important role in the regulation of fatty acid oxidation both in the fasted state and in m …
Adipose Tissue Dysfunction in Polycystic Ovary Syndrome.
Bril F, Ezeh U, Amiri M, Hatoum S, Pace L, Chen YH, Bertrand F, Gower B, Azziz R. Bril F, et al. J Clin Endocrinol Metab. 2023 Dec 21;109(1):10-24. doi: 10.1210/clinem/dgad356. J Clin Endocrinol Metab. 2023. PMID: 37329216 Free PMC article.
PURPOSE: Polycystic ovary syndrome (PCOS) is a complex genetic trait and the most common endocrine disorder of women, clinically evident in 5% to 15% of reproductive-aged women globally, with associated cardiometabolic dysfunction. Adipose tissue (AT) dysfunction appears t …
PURPOSE: Polycystic ovary syndrome (PCOS) is a complex genetic trait and the most common endocrine disorder of women, clinically evident in …
Lipid metabolism in MASLD and MASH: From mechanism to the clinic.
Carli F, Della Pepa G, Sabatini S, Vidal Puig A, Gastaldelli A. Carli F, et al. JHEP Rep. 2024 Aug 9;6(12):101185. doi: 10.1016/j.jhepr.2024.101185. eCollection 2024 Dec. JHEP Rep. 2024. PMID: 39583092 Free PMC article. Review.
Metabolic dysfunction-associated steatotic liver disease/steatohepatitis (MASLD/MASH) is recognised as a metabolic disease characterised by excess intrahepatic lipid accumulation due to lipid overflow and synthesis, alongside impaired oxidation and/or export of these lipid …
Metabolic dysfunction-associated steatotic liver disease/steatohepatitis (MASLD/MASH) is recognised as a metabolic disease characterised by …
Energy Metabolism Changes and Dysregulated Lipid Metabolism in Postmenopausal Women.
Ko SH, Jung Y. Ko SH, et al. Nutrients. 2021 Dec 20;13(12):4556. doi: 10.3390/nu13124556. Nutrients. 2021. PMID: 34960109 Free PMC article. Review.
In particular, increased bone marrow-derived adipocytes due to estrogen loss contribute to increased visceral fat in postmenopausal women. Enhanced visceral fat lipolysis by adipose tissue lipoprotein lipase triggers the production of excessive free fatty …
In particular, increased bone marrow-derived adipocytes due to estrogen loss contribute to increased visceral fat in postmenopausal w …
Obesity and Cancer: A Translational Science Review.
Shen S, Brown KA, Green AK, Iyengar NM. Shen S, et al. JAMA. 2026 Apr 21;335(15):1341-1350. doi: 10.1001/jama.2026.1114. JAMA. 2026. PMID: 41801209 Review.
Excess energy, in the form of free fatty acids, is transferred to developing cancer cells and stimulates cancer development through genomic instability caused by oxidative stress and DNA damage. ...Weight loss may reduce cancer risk by attenuating adve …
Excess energy, in the form of free fatty acids, is transferred to developing cancer cells and stimulates cancer development th …
Mitochondrial control of fuel switching via carnitine biosynthesis.
Auger C, Nishida H, Yuan B, Silva GM, Fujimoto M, Li M, Katoh D, Wang D, Granath-Panelo M, Shin J, Witte R, Yook JS, Verkerke ARP, Banks AS, Hui S, Sun L, Kajimura S. Auger C, et al. Science. 2026 Feb 12;391(6786):eady5532. doi: 10.1126/science.ady5532. Epub 2026 Feb 12. Science. 2026. PMID: 41505503
Environmental adaptation often involves a shift in energy utilization toward mitochondrial fatty acid oxidation, which requires carnitine. Besides dietary sources of animal origin, carnitine biosynthesis from trimethyllysine (TML) is essential, particularly f …
Environmental adaptation often involves a shift in energy utilization toward mitochondrial fatty acid oxidation, which …
Targeting activin receptor-like kinase 7 ameliorates adiposity and associated metabolic disorders.
Zhao M, Okunishi K, Bu Y, Kikuchi O, Wang H, Kitamura T, Izumi T. Zhao M, et al. JCI Insight. 2023 Feb 22;8(4):e161229. doi: 10.1172/jci.insight.161229. JCI Insight. 2023. PMID: 36626233 Free PMC article.
In the present study, treatment with a neutralizing mAb against ALK7 caused a substantial loss of adipose mass and improved glucose intolerance and insulin resistance in both genetic and diet-induced mouse obesity models. The enhanced lipolysis increased f …
In the present study, treatment with a neutralizing mAb against ALK7 caused a substantial loss of adipose mass and improved gl …
Intermittent fasting protects beta-cell identity and function in a type-2 diabetes model.
Patel S, Yan Z, Remedi MS. Patel S, et al. Metabolism. 2024 Apr;153:155813. doi: 10.1016/j.metabol.2024.155813. Epub 2024 Feb 1. Metabolism. 2024. PMID: 38307325 Free PMC article.
T2DM is characterized by hyperglycemia, insulin secretion deficiency and insulin resistance. Chronic hyperglycemia induces beta-cell dysfunction, loss of beta-cell mass/identity and beta-cell dedifferentiation. Intermittent fasting (IF) a commonly used …
T2DM is characterized by hyperglycemia, insulin secretion deficiency and insulin resistance. Chronic hyperglycemia induces beta-cell …
Angiocrine polyamine production regulates adiposity.
Monelli E, Villacampa P, Zabala-Letona A, Martinez-Romero A, Llena J, Beiroa D, Gouveia L, Chivite I, Zagmutt S, Gama-Perez P, Osorio-Conles O, Muixi L, Martinez-Gonzalez A, Castillo SD, Martín-Martín N, Castel P, Valcarcel-Jimenez L, Garcia-Gonzalez I, Villena JA, Fernandez-Ruiz S, Serra D, Herrero L, Benedito R, Garcia-Roves P, Vidal J, Cohen P, Nogueiras R, Claret M, Carracedo A, Graupera M. Monelli E, et al. Nat Metab. 2022 Mar;4(3):327-343. doi: 10.1038/s42255-022-00544-6. Epub 2022 Mar 14. Nat Metab. 2022. PMID: 35288722
Here, we show that local production of polyamines in ECs stimulates adipocyte lipolysis and regulates WAT homeostasis in mice. We promote enhanced cell-autonomous angiogenesis by deleting Pten in the murine endothelium. Endothelial Pten loss leads to a WAT-selective …
Here, we show that local production of polyamines in ECs stimulates adipocyte lipolysis and regulates WAT homeostasis in mice. We promote en …
Brown fat ATP-citrate lyase links carbohydrate availability to thermogenesis and guards against metabolic stress.
Korobkina ED, Calejman CM, Haley JA, Kelly ME, Li H, Gaughan M, Chen Q, Pepper HL, Ahmad H, Boucher A, Fluharty SM, Lin TY, Lotun A, Peura J, Trefely S, Green CR, Vo P, Semenkovich CF, Pitarresi JR, Spinelli JB, Aydemir O, Metallo CM, Lynes MD, Jang C, Snyder NW, Wellen KE, Guertin DA. Korobkina ED, et al. Nat Metab. 2024 Nov;6(11):2187-2202. doi: 10.1038/s42255-024-01143-3. Epub 2024 Oct 14. Nat Metab. 2024. PMID: 39402290 Free PMC article.
Brown adipose tissue (BAT) engages futile fatty acid synthesis-oxidation cycling, the purpose of which has remained elusive. ...ACLY's role in preventing BAT stress becomes critical when mice are weaned onto a carbohydrate-plentiful diet, while removin …
Brown adipose tissue (BAT) engages futile fatty acid synthesis-oxidation cycling, the purpose of which has remai …
634 results