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Measuring Mitochondrial Dysfunction Caused by Soluble α-Synuclein Oligomers.
Luth ES, Stavrovskaya IG. Luth ES, et al. Methods Mol Biol. 2019;1948:183-198. doi: 10.1007/978-1-4939-9124-2_14. Methods Mol Biol. 2019. PMID: 30771178
Defining the native state of α-synuclein.
Selkoe D, Dettmer U, Luth E, Kim N, Newman A, Bartels T. Selkoe D, et al. Neurodegener Dis. 2014;13(2-3):114-7. doi: 10.1159/000355516. Epub 2013 Oct 30. Neurodegener Dis. 2014. PMID: 24192542 Review.
N-alpha-acetylation of α-synuclein increases its helical folding propensity, GM1 binding specificity and resistance to aggregation.
Bartels T, Kim NC, Luth ES, Selkoe DJ. Bartels T, et al. Among authors: luth es. PLoS One. 2014 Jul 30;9(7):e103727. doi: 10.1371/journal.pone.0103727. eCollection 2014. PLoS One. 2014. PMID: 25075858 Free PMC article.
Soluble, prefibrillar α-synuclein oligomers promote complex I-dependent, Ca2+-induced mitochondrial dysfunction.
Luth ES, Stavrovskaya IG, Bartels T, Kristal BS, Selkoe DJ. Luth ES, et al. J Biol Chem. 2014 Aug 1;289(31):21490-507. doi: 10.1074/jbc.M113.545749. Epub 2014 Jun 18. J Biol Chem. 2014. PMID: 24942732 Free PMC article.
In vivo cross-linking reveals principally oligomeric forms of α-synuclein and β-synuclein in neurons and non-neural cells.
Dettmer U, Newman AJ, Luth ES, Bartels T, Selkoe D. Dettmer U, et al. Among authors: luth es. J Biol Chem. 2013 Mar 1;288(9):6371-85. doi: 10.1074/jbc.M112.403311. Epub 2013 Jan 14. J Biol Chem. 2013. PMID: 23319586 Free PMC article.
Purification of α-synuclein from human brain reveals an instability of endogenous multimers as the protein approaches purity.
Luth ES, Bartels T, Dettmer U, Kim NC, Selkoe DJ. Luth ES, et al. Biochemistry. 2015 Jan 20;54(2):279-92. doi: 10.1021/bi501188a. Epub 2014 Dec 23. Biochemistry. 2015. PMID: 25490121 Free PMC article.
Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation.
Dettmer U, Newman AJ, Soldner F, Luth ES, Kim NC, von Saucken VE, Sanderson JB, Jaenisch R, Bartels T, Selkoe D. Dettmer U, et al. Among authors: luth es. Nat Commun. 2015 Jun 16;6:7314. doi: 10.1038/ncomms8314. Nat Commun. 2015. PMID: 26076669 Free PMC article.
The AP2 clathrin adaptor protein complex regulates the abundance of GLR-1 glutamate receptors in the ventral nerve cord of Caenorhabditis elegans.
Garafalo SD, Luth ES, Moss BJ, Monteiro MI, Malkin E, Juo P. Garafalo SD, et al. Among authors: luth es. Mol Biol Cell. 2015 May 15;26(10):1887-900. doi: 10.1091/mbc.E14-06-1048. Epub 2015 Mar 18. Mol Biol Cell. 2015. PMID: 25788288 Free PMC article.
Biochemical and functional interaction of disrupted-in-schizophrenia 1 and amyloid precursor protein regulates neuronal migration during mammalian cortical development.
Young-Pearse TL, Suth S, Luth ES, Sawa A, Selkoe DJ. Young-Pearse TL, et al. Among authors: luth es. J Neurosci. 2010 Aug 4;30(31):10431-40. doi: 10.1523/JNEUROSCI.1445-10.2010. J Neurosci. 2010. PMID: 20685985 Free PMC article.
Bone marrow side population cells are enriched for progenitors capable of myogenic differentiation.
Luth ES, Jun SJ, Wessen MK, Liadaki K, Gussoni E, Kunkel LM. Luth ES, et al. J Cell Sci. 2008 May 1;121(Pt 9):1426-34. doi: 10.1242/jcs.021675. Epub 2008 Apr 8. J Cell Sci. 2008. PMID: 18397996 Free PMC article.
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