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Page 1
Did you mean M. zhang[Author] (78,973 results)?
Autophagy induction promoted by m(6)A reader YTHDF3 through translation upregulation of FOXO3 mRNA.
Hao W, Dian M, Zhou Y, Zhong Q, Pang W, Li Z, Zhao Y, Ma J, Lin X, Luo R, Li Y, Jia J, Shen H, Huang S, Dai G, Wang J, Sun Y, Xiao D. Hao W, et al. Among authors: zhong q. Nat Commun. 2022 Oct 4;13(1):5845. doi: 10.1038/s41467-022-32963-0. Nat Commun. 2022. PMID: 36195598 Free PMC article.
Here, we show that the m(6)A reader YTHDF3 is essential for autophagy induction. m(6)A modification is up-regulated to promote autophagosome formation and lysosomal degradation upon nutrient deficiency. METTL3 depletion leads to a loss of functional m(6)A mod …
Here, we show that the m(6)A reader YTHDF3 is essential for autophagy induction. m(6)A modification is up-regulated to promote …
METTL3/METTL14 Transactivation and m(6)A-Dependent TGF-beta1 Translation in Activated Kupffer Cells.
Feng Y, Dong H, Sun B, Hu Y, Yang Y, Jia Y, Jia L, Zhong X, Zhao R. Feng Y, et al. Among authors: zhong x. Cell Mol Gastroenterol Hepatol. 2021;12(3):839-856. doi: 10.1016/j.jcmgh.2021.05.007. Epub 2021 May 13. Cell Mol Gastroenterol Hepatol. 2021. PMID: 33992834 Free PMC article.
RNA-sequencing and m(6)A-seq were employed to screen differentially expressed genes and responsive m(6)A peaks. ...CONCLUSIONS: NF-kappaB acts as transcription factor to transactivate METTL3/METTL14 genes upon LPS challenge, leading to global RNA m(6)A hyperm …
RNA-sequencing and m(6)A-seq were employed to screen differentially expressed genes and responsive m(6)A peaks. ...CONCLUSIONS …
PM2.5 induces pulmonary microvascular injury in COPD via METTL16-mediated m6A modification.
Guo X, Lin Y, Lin Y, Zhong Y, Yu H, Huang Y, Yang J, Cai Y, Liu F, Li Y, Zhang QQ, Dai J. Guo X, et al. Among authors: zhong y. Environ Pollut. 2022 Jun 15;303:119115. doi: 10.1016/j.envpol.2022.119115. Epub 2022 Mar 5. Environ Pollut. 2022. PMID: 35259473 Free article.
Moreover, microvascular injury in COPD was validated by measurements of plasma endothelial microparticles (EMPs) and serum VEGF in COPD patients. We then performed m(6)A sequencing, which confirmed that altered N(6)-methyladenosine (m(6)A) modification was induced b …
Moreover, microvascular injury in COPD was validated by measurements of plasma endothelial microparticles (EMPs) and serum VEGF in COPD pati …
EGFR promotes ALKBH5 nuclear retention to attenuate N6-methyladenosine and protect against ferroptosis in glioblastoma.
Lv D, Zhong C, Dixit D, Yang K, Wu Q, Godugu B, Prager BC, Zhao G, Wang X, Xie Q, Bao S, He C, Heiland DH, Rosenfeld MG, Rich JN. Lv D, et al. Among authors: zhong c. Mol Cell. 2023 Dec 7;83(23):4334-4351.e7. doi: 10.1016/j.molcel.2023.10.025. Epub 2023 Nov 17. Mol Cell. 2023. PMID: 37979586
Here, we show that epidermal growth factor receptor (EGFR) signaling repressed N(6)-methyladenosine (m(6)A) levels in glioblastoma stem cells (GSCs), whereas genetic or pharmacologic EGFR targeting elevated m(6)A levels. Activated EGFR induced non-receptor tyrosine …
Here, we show that epidermal growth factor receptor (EGFR) signaling repressed N(6)-methyladenosine (m(6)A) levels in glioblastoma st …
KIAA1429 promotes tumorigenesis and gefitinib resistance in lung adenocarcinoma by activating the JNK/ MAPK pathway in an m(6)A-dependent manner.
Lin X, Ye R, Li Z, Zhang B, Huang Y, Du J, Wang B, Meng H, Xian H, Yang X, Zhang X, Zhong Y, Huang Z. Lin X, et al. Among authors: zhong y. Drug Resist Updat. 2023 Jan;66:100908. doi: 10.1016/j.drup.2022.100908. Epub 2022 Dec 5. Drug Resist Updat. 2023. PMID: 36493511 Free article.
In this study, we evaluated the effects of vir-like m(6)A methyltransferase-associated protein (KIAA1429) depletion on proliferation, migration, invasion, and drug resistance of LUAD cells, and identified m(6)A-dependent downstream genes influenced by KIAA1429. ...W …
In this study, we evaluated the effects of vir-like m(6)A methyltransferase-associated protein (KIAA1429) depletion on proliferation, …
The RNA N6-methyladenosine modification landscape of human fetal tissues.
Xiao S, Cao S, Huang Q, Xia L, Deng M, Yang M, Jia G, Liu X, Shi J, Wang W, Li Y, Liu S, Zhu H, Tan K, Luo Q, Zhong M, He C, Xia L. Xiao S, et al. Among authors: zhong m. Nat Cell Biol. 2019 May;21(5):651-661. doi: 10.1038/s41556-019-0315-4. Epub 2019 Apr 29. Nat Cell Biol. 2019. PMID: 31036937
Here, to explore the global landscape of m(6)A in human tissues, we generated 21 whole-transcriptome m(6)A methylomes across major fetal tissues using m(6)A sequencing. These data reveal dynamic m(6)A methylation, identify large numbers of tissue diffe …
Here, to explore the global landscape of m(6)A in human tissues, we generated 21 whole-transcriptome m(6)A methylomes across m …
m(6)A epitranscriptomic modification regulates neural progenitor-to-glial cell transition in the retina.
Xin Y, He Q, Liang H, Zhang K, Guo J, Zhong Q, Chen D, Li J, Liu Y, Chen S. Xin Y, et al. Among authors: zhong q. Elife. 2022 Dec 2;11:e79994. doi: 10.7554/eLife.79994. Elife. 2022. PMID: 36459087 Free PMC article.
Overexpression of m(6)A-regulated RPC transcripts in late RPCs partially recapitulates the Mettl3-deficient retinal phenotype. ...However, it is unclear whether m(6)A is involved in how glial cells in the retina develop. To address this question, Xin et al. studied …
Overexpression of m(6)A-regulated RPC transcripts in late RPCs partially recapitulates the Mettl3-deficient retinal phenotype. ...How …
Emerging role of m(6) A RNA methylation in nutritional physiology and metabolism.
Wu J, Frazier K, Zhang J, Gan Z, Wang T, Zhong X. Wu J, et al. Among authors: zhong x. Obes Rev. 2020 Jan;21(1):e12942. doi: 10.1111/obr.12942. Epub 2019 Sep 2. Obes Rev. 2020. PMID: 31475777 Free PMC article. Review.
Recent evidence indicates that m(6) A methylation regulates physiology and metabolism, and m(6) A has been increasingly implicated in a variety of human diseases, including obesity, diabetes, metabolic syndrome and cancer. Conversely, nutrition and diet can modulate …
Recent evidence indicates that m(6) A methylation regulates physiology and metabolism, and m(6) A has been increasingly implic …
The regulatory role of N6 -methyladenosine modification in the interaction between host and microbes.
Zhuo R, Xu M, Wang X, Zhou B, Wu X, Leone V, Chang EB, Zhong X. Zhuo R, et al. Among authors: zhong x. Wiley Interdiscip Rev RNA. 2022 Nov;13(6):e1725. doi: 10.1002/wrna.1725. Epub 2022 Mar 17. Wiley Interdiscip Rev RNA. 2022. PMID: 35301791 Review.
N(6) -methyladenosine (m(6) A) is the most prevalent posttranscriptional modification in eukaryotic mRNAs. ...In this review, we summarize the regulatory mechanisms of m(6) A modification on viruses and commensal microbiota, highlight the roles of m(6) A meth …
N(6) -methyladenosine (m(6) A) is the most prevalent posttranscriptional modification in eukaryotic mRNAs. ...In this review, we summ …
Brucella-driven host N-glycome remodeling controls infection.
Cabello AL, Wells K, Peng W, Feng HQ, Wang J, Meyer DF, Noroy C, Zhao ES, Zhang H, Li X, Chang H, Gomez G, Mao Y, Patrick KL, Watson RO, Russell WK, Yu A, Zhong J, Guo F, Li M, Zhou M, Qian X, Kobayashi KS, Song J, Panthee S, Mechref Y, Ficht TA, Qin QM, de Figueiredo P. Cabello AL, et al. Among authors: zhong j. Cell Host Microbe. 2024 Apr 10;32(4):588-605.e9. doi: 10.1016/j.chom.2024.03.003. Epub 2024 Mar 25. Cell Host Microbe. 2024. PMID: 38531364
4,493 results