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Genome-wide meta-analysis identifies six novel loci associated with habitual coffee consumption.
Coffee and Caffeine Genetics Consortium, Cornelis MC, Byrne EM, Esko T, Nalls MA, Ganna A, Paynter N, Monda KL, Amin N, Fischer K, Renstrom F, Ngwa JS, Huikari V, Cavadino A, Nolte IM, Teumer A, Yu K, Marques-Vidal P, Rawal R, Manichaikul A, Wojczynski MK, Vink JM, Zhao JH, Burlutsky G, Lahti J, Mikkilä V, Lemaitre RN, Eriksson J, Musani SK, Tanaka T, Geller F, Luan J, Hui J, Mägi R, Dimitriou M, Garcia ME, Ho WK, Wright MJ, Rose LM, Magnusson PK, Pedersen NL, Couper D, Oostra BA, Hofman A, Ikram MA, Tiemeier HW, Uitterlinden AG, van Rooij FJ, Barroso I, Johansson I, Xue L, Kaakinen M, Milani L, Power C, Snieder H, Stolk RP, Baumeister SE, Biffar R, Gu F, Bastardot F, Kutalik Z, Jacobs DR Jr, Forouhi NG, Mihailov E, Lind L, Lindgren C, Michaëlsson K, Morris A, Jensen M, Khaw KT, Luben RN, Wang JJ, Männistö S, Perälä MM, Kähönen M, Lehtimäki T, Viikari J, Mozaffarian D, Mukamal K, Psaty BM, Döring A, Heath AC, Montgomery GW, Dahmen N, Carithers T, Tucker KL, Ferrucci L, Boyd HA, Melbye M, Treur JL, Mellström D, Hottenga JJ, Prokopenko I, Tönjes A, Deloukas P, Kanoni S, Lorentzon M, Houston DK, Liu Y, Danesh J, Rasheed A, Mason MA, Zonderman AB, Franke L, Kristal BS; International Parkinson’s Disease Genomics Consortium (IPDGC); North American Brain Expression Consortium (NABEC); UK Brain Expression Consortium (UKBEC), Karjalainen J, Reed DR, Westra HJ, Evans MK, Saleheen D, Harris TB, Dedoussis G, Curhan G, Stumvoll M, Beilby J, Pasquale LR, Feenstra B, Bandinelli S, Ordovas JM, Chan AT, Peters U, Ohlsson C, Gieger C, Martin NG, Waldenberger M, Siscovick DS, Raitakari O, Eriksson JG, Mitchell P, Hunter DJ, Kraft P, Rimm EB, Boomsma DI, Borecki IB, Loos RJ, Wareham NJ, Vollenweider P, Caporaso N, Grabe HJ, Neuhouser ML, Wolffenbuttel BH, Hu FB, Hyppönen E, Järvelin MR, Cupples LA, Franks PW, Ridker PM, van Duijn CM, Heiss G, Metspalu A, North KE, Ingelsson E, Nettleton JA, van Dam RM, Chasman DI. Coffee and Caffeine Genetics Consortium, et al. Mol Psychiatry. 2015 May;20(5):647-656. doi: 10.1038/mp.2014.107. Epub 2014 Oct 7. Mol Psychiatry. 2015. PMID: 25288136 Free PMC article. Review.
Two map to GCKR and MLXIPL genes related to metabolic traits but lacking known roles in coffee consumption. Enhancer and promoter histone marks populate the regions of many confirmed loci and several potential regulatory SNPs are highly correlated with the lead SNP of each …
Two map to GCKR and MLXIPL genes related to metabolic traits but lacking known roles in coffee consumption. Enhancer and promoter his …
Adipocyte ACLY Facilitates Dietary Carbohydrate Handling to Maintain Metabolic Homeostasis in Females.
Fernandez S, Viola JM, Torres A, Wallace M, Trefely S, Zhao S, Affronti HC, Gengatharan JM, Guertin DA, Snyder NW, Metallo CM, Wellen KE. Fernandez S, et al. Cell Rep. 2019 May 28;27(9):2772-2784.e6. doi: 10.1016/j.celrep.2019.04.112. Cell Rep. 2019. PMID: 31141698 Free PMC article.
Is C771G polymorphism of MLX interacting protein-like (MLXIPL) gene a novel genetic risk factor for non-alcoholic fatty liver disease?
Seifi M, Ghasemi A, Namipashaki A, Samadikuchaksaraei A. Seifi M, et al. Cell Mol Biol (Noisy-le-grand). 2014;60(3):37-42. Cell Mol Biol (Noisy-le-grand). 2014. PMID: 26177557
Since, no study has been reported on the association between MLXIPL gene and non-alcoholic fatty liver disease (NAFLD), we aimed to identify a connection between this genetic variation and NAFLD. ...C771G polymorphism in the MLXIPL gene potentially plays a si …
Since, no study has been reported on the association between MLXIPL gene and non-alcoholic fatty liver disease (NAFLD), we aim …
ChREBP expression in the liver, adipose tissue and differentiated preadipocytes in human obesity.
Hurtado del Pozo C, Vesperinas-García G, Rubio MÁ, Corripio-Sánchez R, Torres-García AJ, Obregon MJ, Calvo RM. Hurtado del Pozo C, et al. Biochim Biophys Acta. 2011 Dec;1811(12):1194-200. doi: 10.1016/j.bbalip.2011.07.016. Epub 2011 Jul 30. Biochim Biophys Acta. 2011. PMID: 21840420 Free article.
We have studied ChREBP mRNA and protein expression levels in the liver and the omental (OM) and subcutaneous (SC) adipose tissues from obese and lean subjects, as well as in human differentiated preadipocytes. ...We found opposing results in terms of ChREBP regulation in t …
We have studied ChREBP mRNA and protein expression levels in the liver and the omental (OM) and subcutaneous (SC) adipose tissues fro …
A Slow-Digesting Carbohydrate Diet during Rat Pregnancy Protects Offspring from Non-Alcoholic Fatty Liver Disease Risk through the Modulation of the Carbohydrate-Response Element and Sterol Regulatory Element Binding Proteins.
Salto R, Manzano M, Girón MD, Cano A, Castro A, Vílchez JD, Cabrera E, López-Pedrosa JM. Salto R, et al. Nutrients. 2019 Apr 14;11(4):844. doi: 10.3390/nu11040844. Nutrients. 2019. PMID: 31013988 Free PMC article.
However, the effects of these diets on metabolic programming in the livers of offspring, and the liver metabolism contributions to adipogenesis, remain to be addressed. ...In conclusion, an HF-RD diet during pregnancy translates to changes in liver signaling …
However, the effects of these diets on metabolic programming in the livers of offspring, and the liver metabolism contribution …
High prevalence of an anti-hypertriglyceridemic variant of the MLXIPL gene in Central Asia.
Nakayama K, Yanagisawa Y, Ogawa A, Ishizuka Y, Munkhtulga L, Charupoonphol P, Supannnatas S, Kuartei S, Chimedregzen U, Koda Y, Ishida T, Kagawa Y, Iwamoto S. Nakayama K, et al. J Hum Genet. 2011 Dec;56(12):828-33. doi: 10.1038/jhg.2011.109. Epub 2011 Sep 22. J Hum Genet. 2011. PMID: 21938000
MLXIPL is a transcription factor integral to the regulation of glycolysis and lipogenesis in the liver. Common variants of the MLXIPL gene (MLXIPL) are known to influence plasma triglyceride levels in people of European descent. As MLXIPL has a
MLXIPL is a transcription factor integral to the regulation of glycolysis and lipogenesis in the liver. Common variants of the
Isoflavones as a smart curer for non-alcoholic fatty liver disease and pathological adiposity via ChREBP and Wnt signaling.
Kim MH, Kang KS. Kim MH, et al. Prev Med. 2012 May;54 Suppl:S57-63. doi: 10.1016/j.ypmed.2011.12.018. Epub 2011 Dec 28. Prev Med. 2012. PMID: 22227283 Review.
OBJECTIVE: Non-alcoholic fatty liver disease (NAFLD) and pathological adiposity has emerged as an important modern disease. Along with this, the requirement for alternative and natural medicine for preventing NAFLD and adiposity has been increasing rapidly and considerably …
OBJECTIVE: Non-alcoholic fatty liver disease (NAFLD) and pathological adiposity has emerged as an important modern disease. Along wit …
Targeted sequencing of candidate genes of dyslipidemia in Punjabi Sikhs: Population-specific rare variants in GCKR promote ectopic fat deposition.
Sanghera DK, Hopkins R, Malone-Perez MW, Bejar C, Tan C, Mussa H, Whitby P, Fowler B, Rao CV, Fung KA, Lightfoot S, Frazer JK. Sanghera DK, et al. PLoS One. 2019 Aug 1;14(8):e0211661. doi: 10.1371/journal.pone.0211661. eCollection 2019. PLoS One. 2019. PMID: 31369557 Free PMC article.
Gene-centric analysis revealed burden of variants for increasing HTG risk in GCKR (p = 2.1x10-5), LPL (p = 1.6x10-3) and MLXIPL (p = 1.6x10-2) genes. ...Liver histology of the GCKRmut showed severe fatty metamorphosis which correlated with ~7 fold increase in the mR …
Gene-centric analysis revealed burden of variants for increasing HTG risk in GCKR (p = 2.1x10-5), LPL (p = 1.6x10-3) and MLXIPL (p = …
De novo lipogenesis in human fat and liver is linked to ChREBP-betaand metabolic health.
Eissing L, Scherer T, Tödter K, Knippschild U, Greve JW, Buurman WA, Pinnschmidt HO, Rensen SS, Wolf AM, Bartelt A, Heeren J, Buettner C, Scheja L. Eissing L, et al. Nat Commun. 2013;4:1528. doi: 10.1038/ncomms2537. Nat Commun. 2013. PMID: 23443556 Free PMC article.
How de novo lipogenesis in white adipose tissue versus liver is altered in human obesity and insulin resistance is poorly understood. ...Notably, lipogenic gene expression in both white adipose tissue and liver was strongly linked to the expression of carbohydrate-r …
How de novo lipogenesis in white adipose tissue versus liver is altered in human obesity and insulin resistance is poorly understood. …
Six new loci associated with blood low-density lipoprotein cholesterol, high-density lipoprotein cholesterol or triglycerides in humans.
Kathiresan S, Melander O, Guiducci C, Surti A, Burtt NP, Rieder MJ, Cooper GM, Roos C, Voight BF, Havulinna AS, Wahlstrand B, Hedner T, Corella D, Tai ES, Ordovas JM, Berglund G, Vartiainen E, Jousilahti P, Hedblad B, Taskinen MR, Newton-Cheh C, Salomaa V, Peltonen L, Groop L, Altshuler DM, Orho-Melander M. Kathiresan S, et al. Nat Genet. 2008 Feb;40(2):189-97. doi: 10.1038/ng.75. Epub 2008 Jan 13. Nat Genet. 2008. PMID: 18193044 Free PMC article.
Of the six newly identified chromosomal regions, two were associated with LDL cholesterol (1p13 near CELSR2, PSRC1 and SORT1 and 19p13 near CILP2 and PBX4), one with HDL cholesterol (1q42 in GALNT2) and five with triglycerides (7q11 near TBL2 and MLXIPL, 8q24 near TRIB1, 1 …
Of the six newly identified chromosomal regions, two were associated with LDL cholesterol (1p13 near CELSR2, PSRC1 and SORT1 and 19p13 near …
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