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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1989 1
1990 2
1991 2
1992 2
1993 4
1994 4
1995 9
1996 9
1997 3
1998 14
1999 10
2000 11
2001 8
2002 13
2003 13
2004 25
2005 13
2006 9
2007 26
2008 20
2009 15
2010 15
2011 27
2012 30
2013 30
2014 26
2015 21
2016 16
2017 30
2018 21
2019 27
2020 10
2021 0
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418 results
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Page 1
Function of the myogenic regulatory factors Myf5, MyoD, Myogenin and MRF4 in skeletal muscle, satellite cells and regenerative myogenesis.
Zammit PS. Zammit PS. Semin Cell Dev Biol. 2017 Dec;72:19-32. doi: 10.1016/j.semcdb.2017.11.011. Epub 2017 Nov 15. Semin Cell Dev Biol. 2017. PMID: 29127046 Review.
While MYF5, MYOD, Myogenin and MRF4 have subtle roles in mature muscle, they again play a crucial role in directing satellite cell function to regenerate skeletal muscle: linking the genetic control of developmental and regenerative myogenesis. Here, I …
While MYF5, MYOD, Myogenin and MRF4 have subtle roles in mature muscle, they again play a crucial role in directing satellite …
Loss of MyoD and Myf5 in Skeletal Muscle Stem Cells Results in Altered Myogenic Programming and Failed Regeneration.
Yamamoto M, Legendre NP, Biswas AA, Lawton A, Yamamoto S, Tajbakhsh S, Kardon G, Goldhamer DJ. Yamamoto M, et al. Stem Cell Reports. 2018 Mar 13;10(3):956-969. doi: 10.1016/j.stemcr.2018.01.027. Epub 2018 Mar 1. Stem Cell Reports. 2018. PMID: 29478898 Free PMC article.
MyoD and Myf5 are fundamental regulators of skeletal muscle lineage determination in the embryo, and their expression is induced in satellite cells following muscle injury. ...We show that satellite cells lacking both MyoD and Myf5 (double knock …
MyoD and Myf5 are fundamental regulators of skeletal muscle lineage determination in the embryo, and their expression i …
Skeletal muscle hypertrophy and regeneration: interplay between the myogenic regulatory factors (MRFs) and insulin-like growth factors (IGFs) pathways.
Zanou N, Gailly P. Zanou N, et al. Cell Mol Life Sci. 2013 Nov;70(21):4117-30. doi: 10.1007/s00018-013-1330-4. Epub 2013 Apr 4. Cell Mol Life Sci. 2013. PMID: 23552962 Review.
Adult skeletal muscle can regenerate in response to muscle damage. This ability is conferred by the presence of myogenic stem cells called satellite cells. ...MGF is expressed first and is observed in satellite cells and in proliferating myoblasts wher …
Adult skeletal muscle can regenerate in response to muscle damage. This ability is conferred by the presence of myogeni …
Does skeletal muscle have an 'epi'-memory? The role of epigenetics in nutritional programming, metabolic disease, aging and exercise.
Sharples AP, Stewart CE, Seaborne RA. Sharples AP, et al. Aging Cell. 2016 Aug;15(4):603-16. doi: 10.1111/acel.12486. Epub 2016 Apr 22. Aging Cell. 2016. PMID: 27102569 Free PMC article. Review.
Skeletal muscle mass, quality and adaptability are fundamental in promoting muscle performance, maintaining metabolic function and supporting longevity and healthspan. ...Finally, we will outline the molecular and epigenetic mechanisms underlying skeletal
Skeletal muscle mass, quality and adaptability are fundamental in promoting muscle performance, maintaining metabolic f
VGLL3 operates via TEAD1, TEAD3 and TEAD4 to influence myogenesis in skeletal muscle.
Figeac N, Mohamed AD, Sun C, Schönfelder M, Matallanas D, Garcia-Munoz A, Missiaglia E, Collie-Duguid E, De Mello V, Pobbati AV, Pruller J, Jaka O, Harridge SDR, Hong W, Shipley J, Vargesson N, Zammit PS, Wackerhage H. Figeac N, et al. J Cell Sci. 2019 Jul 5;132(13):jcs225946. doi: 10.1242/jcs.225946. J Cell Sci. 2019. PMID: 31138678 Free PMC article.
VGLL proteins are transcriptional co-factors that bind TEAD family transcription factors to regulate events ranging from wing development in fly, to muscle fibre composition and immune function in mice. Here, we characterise Vgll3 in skeletal muscle. ...Howev …
VGLL proteins are transcriptional co-factors that bind TEAD family transcription factors to regulate events ranging from wing development in …
PRDM16 controls a brown fat/skeletal muscle switch.
Seale P, Bjork B, Yang W, Kajimura S, Chin S, Kuang S, Scimè A, Devarakonda S, Conroe HM, Erdjument-Bromage H, Tempst P, Rudnicki MA, Beier DR, Spiegelman BM. Seale P, et al. Nature. 2008 Aug 21;454(7207):961-7. doi: 10.1038/nature07182. Nature. 2008. PMID: 18719582 Free PMC article.
We also demonstrate that the transcriptional regulator PRDM16 (PRD1-BF1-RIZ1 homologous domain containing 16) controls a bidirectional cell fate switch between skeletal myoblasts and brown fat cells. Loss of PRDM16 from brown fat precursors causes a loss of brown fa …
We also demonstrate that the transcriptional regulator PRDM16 (PRD1-BF1-RIZ1 homologous domain containing 16) controls a bidirectional cell …
Maternal inflammation at midgestation impairs subsequent fetal myoblast function and skeletal muscle growth in rats, resulting in intrauterine growth restriction at term.
Cadaret CN, Posont RJ, Beede KA, Riley HE, Loy JD, Yates DT. Cadaret CN, et al. Transl Anim Sci. 2019 Mar;3(2):txz037. doi: 10.1093/tas/txz037. Epub 2019 Apr 3. Transl Anim Sci. 2019. PMID: 31032478 Free PMC article.

At necropsy on dGA 20, average fetal mass and upper hindlimb cross-sectional areas were reduced (P < 0.05) in MI-IUGR fetuses compared with controls. MyoD(+) and myf5(+) myoblasts were less abundant (P < 0.05), and myogenin(+) myoblasts were more abunda

At necropsy on dGA 20, average fetal mass and upper hindlimb cross-sectional areas were reduced (P < 0.05) in MI-IUGR fetuses compared wi

Rapamycin-induced autophagy decreases Myf5 and MyoD proteins in C2C12 myoblast cells.
Yoo YM, Jung EM, Jeung EB. Yoo YM, et al. Toxicol In Vitro. 2019 Aug;58:132-141. doi: 10.1016/j.tiv.2019.03.025. Epub 2019 Mar 21. Toxicol In Vitro. 2019. PMID: 30905858
Rapamycin is an immunosuppressant that inhibits the mammalian or mechanistic target of rapamycin (mTOR) protein kinase and extends lifespan in organisms including mice. Myf5 and MyoD act as muscle-specific transcriptional factors for skeletal muscle di …
Rapamycin is an immunosuppressant that inhibits the mammalian or mechanistic target of rapamycin (mTOR) protein kinase and extends lifespan …
MLL1 promotes myogenesis by epigenetically regulating Myf5.
Cai S, Zhu Q, Guo C, Yuan R, Zhang X, Nie Y, Chen L, Fang Y, Chen K, Zhang J, Mo D, Chen Y. Cai S, et al. Cell Prolif. 2020 Feb;53(2):e12744. doi: 10.1111/cpr.12744. Epub 2019 Dec 15. Cell Prolif. 2020. PMID: 31840352 Free PMC article.
Mechanically, MLL1 transcriptionally regulated Myf5 by mediating H3K4me3 on its promoter. In vivo data implied that MLL1 was required for Pax7-positive satellite cell proliferation and muscle repair. CONCLUSION: MLL1 facilitates proliferation of myoblasts and …
Mechanically, MLL1 transcriptionally regulated Myf5 by mediating H3K4me3 on its promoter. In vivo data implied that MLL1 was required …
Direct Reprogramming of Mouse Fibroblasts into Functional Skeletal Muscle Progenitors.
Bar-Nur O, Gerli MFM, Di Stefano B, Almada AE, Galvin A, Coffey A, Huebner AJ, Feige P, Verheul C, Cheung P, Payzin-Dogru D, Paisant S, Anselmo A, Sadreyev RI, Ott HC, Tajbakhsh S, Rudnicki MA, Wagers AJ, Hochedlinger K. Bar-Nur O, et al. Stem Cell Reports. 2018 May 8;10(5):1505-1521. doi: 10.1016/j.stemcr.2018.04.009. Stem Cell Reports. 2018. PMID: 29742392 Free PMC article.
Skeletal muscle harbors quiescent stem cells termed satellite cells and proliferative progenitors termed myoblasts, which play pivotal roles during muscle regeneration. ...Here, we show that ectopic expression of the myogenic transcription factor MyoD,
Skeletal muscle harbors quiescent stem cells termed satellite cells and proliferative progenitors termed myoblasts, whi
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