Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My Custom Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
2003 1
2004 5
2006 1
2007 3
2009 2
2010 1
2011 2
2012 1
2013 1
2017 1
2019 1
2025 0

Publication date

Text availability

Article attribute

Article type

Additional filters

Article Language

Species

Sex

Age

Other

Search Results

17 results

Results by year

Filters applied: . Clear all
Page 1
Calcium channel function and regulation in beta 1- and beta 2-adrenoceptor transgenic mice.
Foerster K, Kaeferstein T, Groner F, Engelhardt S, Matthes J, Koch WJ, Lohse MJ, Herzig S. Foerster K, et al. Among authors: matthes j. Naunyn Schmiedebergs Arch Pharmacol. 2004 May;369(5):490-5. doi: 10.1007/s00210-004-0928-3. Epub 2004 Apr 9. Naunyn Schmiedebergs Arch Pharmacol. 2004. PMID: 15083268
Cardiac effects of catecholamines on the L-type calcium channel depend on beta-adrenoceptor subtype (beta(1)- vs. beta(2)-adrenoceptor). ...In order to examine the role of L-type calcium channels in altered cardiomyocyte calcium homeostas …
Cardiac effects of catecholamines on the L-type calcium channel depend on beta-adrenoceptor subtype (beta(1)- vs. beta( …
Single-channel gating and regulation of human L-type calcium channels in cardiomyocytes of transgenic mice.
Groner F, Rubio M, Schulte-Euler P, Matthes J, Khan IF, Bodi I, Koch SE, Schwartz A, Herzig S. Groner F, et al. Among authors: matthes j. Biochem Biophys Res Commun. 2004 Feb 13;314(3):878-84. doi: 10.1016/j.bbrc.2003.12.174. Biochem Biophys Res Commun. 2004. PMID: 14741718
Overexpression of human cardiac L-type Ca(2+) channel pores (hCa(v)1.2) in mice causes heart failure. Earlier studies showed Ca(v)1.2-mRNA increase by 2.8-fold, but whole-cell current density enhancement by </=1.5-fold only. ...Density of functional cha …
Overexpression of human cardiac L-type Ca(2+) channel pores (hCa(v)1.2) in mice causes heart failure. Earlier studies s …
Single-Channel Resolution of the Interaction between C-Terminal Ca(V)1.3 Isoforms and Calmodulin.
Kuzmenkina E, Novikova E, Jangsangthong W, Matthes J, Herzig S. Kuzmenkina E, et al. Among authors: matthes j. Biophys J. 2019 Mar 5;116(5):836-846. doi: 10.1016/j.bpj.2019.01.025. Epub 2019 Feb 1. Biophys J. 2019. PMID: 30773296 Free PMC article.
Voltage-dependent calcium (Ca(V)) 1.3 channels are involved in the control of cellular excitability and pacemaking in neuronal, cardiac, and sensory cells. Various proteins interact with the alternatively spliced channel C-terminus regulating gating of …
Voltage-dependent calcium (Ca(V)) 1.3 channels are involved in the control of cellular excitability and pacemaking in neuronal …
Pharmacoresistant Cav 2.3 (E-type/R-type) voltage-gated calcium channels influence heart rate dynamics and may contribute to cardiac impulse conduction.
Galetin T, Tevoufouet EE, Sandmeyer J, Matthes J, Nguemo F, Hescheler J, Weiergräber M, Schneider T. Galetin T, et al. Among authors: matthes j. Cell Biochem Funct. 2013 Jul;31(5):434-49. doi: 10.1002/cbf.2918. Epub 2012 Oct 22. Cell Biochem Funct. 2013. PMID: 23086800
Voltage-gated Ca(2+) channels regulate cardiac automaticity, rhythmicity and excitation-contraction coupling. Whereas L-type (Cav 1.2, Cav 1.3) and T-type (Cav 3.1, Cav 3.2) channels are widely accepted for their functional relevance in the heart, the …
Voltage-gated Ca(2+) channels regulate cardiac automaticity, rhythmicity and excitation-contraction coupling. Whereas L-type ( …
Galphai2- and Galphai3-specific regulation of voltage-dependent L-type calcium channels in cardiomyocytes.
Dizayee S, Kaestner S, Kuck F, Hein P, Klein C, Piekorz RP, Meszaros J, Matthes J, Bjrnbaumer L, Nürnberg B, Herzig S. Dizayee S, et al. Among authors: matthes j. PLoS One. 2011;6(9):e24979. doi: 10.1371/journal.pone.0024979. Epub 2011 Sep 26. PLoS One. 2011. PMID: 21966394 Free PMC article.
To test for isoform-specific functions of G(i) proteins, we examined their role in the regulation of cardiac L-type voltage-dependent calcium channels (L-VDCC). METHODS: Ventricular tissues and isolated myocytes were obtained from mice with targeted deletion …
To test for isoform-specific functions of G(i) proteins, we examined their role in the regulation of cardiac L-type voltage-dependent …
Beta-adrenergic regulation of the heart expressing the Ser1700A/Thr1704A mutated Cav1.2 channel.
Poomvanicha M, Matthes J, Domes K, Patrucco E, Angermeier E, Laugwitz KL, Schneider T, Hofmann F. Poomvanicha M, et al. Among authors: matthes j. J Mol Cell Cardiol. 2017 Oct;111:10-16. doi: 10.1016/j.yjmcc.2017.07.119. Epub 2017 Aug 1. J Mol Cell Cardiol. 2017. PMID: 28778765
Beta-adrenergic stimulation of the heart increases I(Ca). PKA dependent phosphorylation of several amino acids (among them Ser 1700 and Thr 1704 in the carboxy-terminus of the Cav1.2 alpha(1)c subunit) has been implicated as decisive for the beta-adrenergic up-regulation o …
Beta-adrenergic stimulation of the heart increases I(Ca). PKA dependent phosphorylation of several amino acids (among them Ser 1700 a …
Cardioprotection specific for the G protein Gi2 in chronic adrenergic signaling through beta 2-adrenoceptors.
Foerster K, Groner F, Matthes J, Koch WJ, Birnbaumer L, Herzig S. Foerster K, et al. Among authors: matthes j. Proc Natl Acad Sci U S A. 2003 Nov 25;100(24):14475-80. doi: 10.1073/pnas.1936026100. Epub 2003 Nov 11. Proc Natl Acad Sci U S A. 2003. PMID: 14612574 Free PMC article.
Two subtypes of beta-adrenoceptors, beta 1 and beta 2, mediate cardiac catecholamine effects. These two types differ qualitatively, e.g., regarding G protein coupling and calcium channel stimulation. ...Single calcium-channel activity was strong …
Two subtypes of beta-adrenoceptors, beta 1 and beta 2, mediate cardiac catecholamine effects. These two types differ qualitatively, e …
Mechanism of Ca(v)1.2 channel modulation by the amino terminus of cardiac beta2-subunits.
Herzig S, Khan IF, Gründemann D, Matthes J, Ludwig A, Michels G, Hoppe UC, Chaudhuri D, Schwartz A, Yue DT, Hullin R. Herzig S, et al. Among authors: matthes j. FASEB J. 2007 May;21(7):1527-38. doi: 10.1096/fj.06-7377com. Epub 2007 Feb 8. FASEB J. 2007. PMID: 17289923
L-type calcium channels are composed of a pore, alpha1c (Ca(V)1.2), and accessory beta- and alpha2delta-subunits. ...The data show that the length of amino termini is a relevant physiological mechanism for channel closure and inactivation, and that natural al …
L-type calcium channels are composed of a pore, alpha1c (Ca(V)1.2), and accessory beta- and alpha2delta-subunits. ...The data …
Effect of MANT-nucleotides on L-type calcium currents in murine cardiomyocytes.
Hübner M, Dizayee S, Matthes J, Seifert R, Herzig S. Hübner M, et al. Among authors: matthes j. Naunyn Schmiedebergs Arch Pharmacol. 2011 Jun;383(6):573-83. doi: 10.1007/s00210-011-0626-x. Epub 2011 Apr 12. Naunyn Schmiedebergs Arch Pharmacol. 2011. PMID: 21484439
Membranous adenylyl cyclases play a major role in G-protein-coupled receptor signalling and regulate various cellular responses, such as cardiac contraction. Cardiac apoptosis and development of cardiac dysfunction is prevented in mice lacking AC 5, a predomi …
Membranous adenylyl cyclases play a major role in G-protein-coupled receptor signalling and regulate various cellular responses, such as …
Transgenic simulation of human heart failure-like L-type Ca2+-channels: implications for fibrosis and heart rate in mice.
Beetz N, Hein L, Meszaros J, Gilsbach R, Barreto F, Meissner M, Hoppe UC, Schwartz A, Herzig S, Matthes J. Beetz N, et al. Among authors: matthes j. Cardiovasc Res. 2009 Dec 1;84(3):396-406. doi: 10.1093/cvr/cvp251. Epub 2009 Jul 20. Cardiovasc Res. 2009. PMID: 19620129 Free PMC article.
AIMS: Cardiac L-type Ca(2+)-currents show distinct alterations in chronic heart failure, including increased single-channel activity and blunted adrenergic stimulation, but minor changes of whole-cell currents. ...Overt cardiac hypertrophy was not obse …
AIMS: Cardiac L-type Ca(2+)-currents show distinct alterations in chronic heart failure, including increased single-channel …
17 results