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Mechanisms of increased hippocampal excitability in the Mashl+/- mouse model of Na+ /K+ -ATPase dysfunction.
Hunanyan AS, Helseth AR, Abdelnour E, Kherallah B, Sachdev M, Chung L, Masoud M, Richardson J, Li Q, Nadler JV, Moore SD, Mikati MA. Hunanyan AS, et al. Epilepsia. 2018 Jul;59(7):1455-1468. doi: 10.1111/epi.14441. Epub 2018 Jun 11. Epilepsia. 2018. PMID: 29889309
OBJECTIVE: Na(+) /K(+) -ATPase dysfunction, primary (mutation) or secondary (energy crisis, neurodegenerative disease) increases neuronal excitability in the brain. ...SIGNIFICANCE: Our data indicate that, in our genetic model of A …
OBJECTIVE: Na(+) /K(+) -ATPase dysfunction, primary (mutation) or secondary (energy crisis, neurodegenerative di …
Knock-in mouse model of alternating hemiplegia of childhood: behavioral and electrophysiologic characterization.
Hunanyan AS, Fainberg NA, Linabarger M, Arehart E, Leonard AS, Adil SM, Helseth AR, Swearingen AK, Forbes SL, Rodriguiz RM, Rhodes T, Yao X, Kibbi N, Hochman DW, Wetsel WC, Hochgeschwender U, Mikati MA. Hunanyan AS, et al. Epilepsia. 2015 Jan;56(1):82-93. doi: 10.1111/epi.12878. Epub 2014 Dec 19. Epilepsia. 2015. PMID: 25523819 Free article.
OBJECTIVES: Mutations in the ATP1alpha3 subunit of the neuronal Na+/K+-ATPase are thought to be responsible for seizures, hemiplegias, and other symptoms of alternating hemiplegia of childhood (AHC). ...SIGNIFICANCE: Our model reproduces the major char …
OBJECTIVES: Mutations in the ATP1alpha3 subunit of the neuronal Na+/K+-ATPase are thought to be responsible for seizure …