Skip to main page content
Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

My NCBI Filters
Text availability
Article attribute
Article type
Publication date

Search Results

709 results
Filters applied: . Clear all Results are displayed in a computed author sort order. Results by year timeline is unavailable
Page 1
Gi3 does not contribute to the inhibition of adenylate cyclase when stimulation of an alpha 2-adrenergic receptor causes activation of both Gi2 and Gi3.
McClue SJ, Selzer E, Freissmuth M, Milligan G. McClue SJ, et al. Among authors: milligan g. Biochem J. 1992 Jun 1;284 ( Pt 2)(Pt 2):565-8. doi: 10.1042/bj2840565. Biochem J. 1992. PMID: 1318036 Free PMC article.
Agonist occupancy of the alpha 2-C10 adrenergic receptor in a stable clone (1C) of Rat 1 fibroblasts produced by transfection of cells with genomic DNA encoding this receptor causes the activation of both of the pertussis-toxin-sensitive G-proteins Gi2 and Gi3 [Milligan
Agonist occupancy of the alpha 2-C10 adrenergic receptor in a stable clone (1C) of Rat 1 fibroblasts produced by transfection of cells with …
Alpha 2-C10 adrenergic receptors expressed in rat 1 fibroblasts can regulate both adenylylcyclase and phospholipase D-mediated hydrolysis of phosphatidylcholine by interacting with pertussis toxin-sensitive guanine nucleotide-binding proteins.
MacNulty EE, McClue SJ, Carr IC, Jess T, Wakelam MJ, Milligan G. MacNulty EE, et al. Among authors: milligan g. J Biol Chem. 1992 Feb 5;267(4):2149-56. J Biol Chem. 1992. PMID: 1346392
We have demonstrated recently in cells of one of these clones (1C) that the alpha 2-C10 receptor interacts directly with two distinct pertussis toxin-sensitive G-proteins, Gi2 and Gi3 (Milligan, G., Carr, C., Gould, G. W., Mullaney, I., and Lavan, B.E. …
We have demonstrated recently in cells of one of these clones (1C) that the alpha 2-C10 receptor interacts directly with two distinct pertus …
Concurrent down-regulation of IP prostanoid receptors and the alpha-subunit of the stimulatory guanine-nucleotide-binding protein (Gs) during prolonged exposure of neuroblastoma x glioma cells to prostanoid agonists. Quantification and functional implications.
Adie EJ, Mullaney I, McKenzie FR, Milligan G. Adie EJ, et al. Among authors: milligan g. Biochem J. 1992 Jul 15;285 ( Pt 2)(Pt 2):529-36. doi: 10.1042/bj2850529. Biochem J. 1992. PMID: 1379045 Free PMC article.
The remaining receptors were capable of interacting with the stimulatory G-protein (Gs) of the adenylate cyclase cascade, as saturation analysis of the binding of [3H]PGE1 indicated that they had a similar affinity for the 3H-labelled ligand, and because the specific bindi …
The remaining receptors were capable of interacting with the stimulatory G-protein (Gs) of the adenylate cyclase cascade, as saturati …
Agonist control of G-protein levels.
Milligan G, Green A. Milligan G, et al. Trends Pharmacol Sci. 1991 Jun;12(6):207-9. doi: 10.1016/0165-6147(91)90551-3. Trends Pharmacol Sci. 1991. PMID: 1646508 Review. No abstract available.
Cholera toxin impairment of opioid-mediated inhibition of adenylate cyclase in neuroblastoma x glioma hybrid cells is due to a toxin-induced decrease in opioid receptor levels.
McKenzie FR, Milligan G. McKenzie FR, et al. Among authors: milligan g. Biochem J. 1991 Apr 1;275 ( Pt 1)(Pt 1):175-81. doi: 10.1042/bj2750175. Biochem J. 1991. PMID: 1673334 Free PMC article.
Furthermore, levels of another pertussis toxin-sensitive G-protein (Go) expressed by these cells was also not lowered by cholera toxin treatment. However, as previously noted in other cells [Milligan, Unson & Wakelam (1989) Biochem. J. 262, 643-649], marked down …
Furthermore, levels of another pertussis toxin-sensitive G-protein (Go) expressed by these cells was also not lowered by cholera toxi …
709 results
You have reached the last page of results. A maximum of 10,000 results are available.
Jump to page