Skip to main page content
Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

My NCBI Filters
Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1994 2
1995 1
1996 1
1997 1
1999 1
2003 2
2005 1
2006 1
2007 1
2021 0
Text availability
Article attribute
Article type
Publication date

Search Results

11 results
Results by year
Filters applied: . Clear all
Page 1
Learning in Aplysia: looking at synaptic plasticity from both sides.
Roberts AC, Glanzman DL. Roberts AC, et al. Trends Neurosci. 2003 Dec;26(12):662-70. doi: 10.1016/j.tins.2003.09.014. Trends Neurosci. 2003. PMID: 14624850 Review.
The cellular mechanisms of learning in Aplysia: of blind men and elephants.
Glanzman DL. Glanzman DL. Biol Bull. 2006 Jun;210(3):271-9. doi: 10.2307/4134563. Biol Bull. 2006. PMID: 16801500 Review.
The cellular basis of classical conditioning in Aplysia californica--it's less simple than you think.
Glanzman DL. Glanzman DL. Trends Neurosci. 1995 Jan;18(1):30-6. doi: 10.1016/0166-2236(95)93947-v. Trends Neurosci. 1995. PMID: 7535488 Review.
Postsynaptic regulation of the development and long-term plasticity of Aplysia sensorimotor synapses in cell culture.
Glanzman DL. Glanzman DL. J Neurobiol. 1994 Jun;25(6):666-93. doi: 10.1002/neu.480250608. J Neurobiol. 1994. PMID: 8071666 Review.
Hebbian induction of long-term potentiation of Aplysia sensorimotor synapses: partial requirement for activation of an NMDA-related receptor.
Lin XY, Glanzman DL. Lin XY, et al. Proc Biol Sci. 1994 Mar 22;255(1344):215-21. doi: 10.1098/rspb.1994.0031. Proc Biol Sci. 1994. PMID: 7912832
Differential classical conditioning of the gill-withdrawal reflex in Aplysia recruits both NMDA receptor-dependent enhancement and NMDA receptor-dependent depression of the reflex.
Jami SA, Wright WG, Glanzman DL. Jami SA, et al. J Neurosci. 2007 Mar 21;27(12):3064-8. doi: 10.1523/JNEUROSCI.2581-06.2007. J Neurosci. 2007. PMID: 17376967 Free PMC article.
Prolonged habituation of the gill-withdrawal reflex in Aplysia depends on protein synthesis, protein phosphatase activity, and postsynaptic glutamate receptors.
Ezzeddine Y, Glanzman DL. Ezzeddine Y, et al. J Neurosci. 2003 Oct 22;23(29):9585-94. doi: 10.1523/JNEUROSCI.23-29-09585.2003. J Neurosci. 2003. PMID: 14573538 Free PMC article.
Synaptic facilitation and behavioral dishabituation in Aplysia: dependence on release of Ca2+ from postsynaptic intracellular stores, postsynaptic exocytosis, and modulation of postsynaptic AMPA receptor efficacy.
Li Q, Roberts AC, Glanzman DL. Li Q, et al. J Neurosci. 2005 Jun 8;25(23):5623-37. doi: 10.1523/JNEUROSCI.5305-04.2005. J Neurosci. 2005. PMID: 15944390 Free PMC article.
Mediation of classical conditioning in Aplysia californica by long-term potentiation of sensorimotor synapses.
Murphy GG, Glanzman DL. Murphy GG, et al. Science. 1997 Oct 17;278(5337):467-71. doi: 10.1126/science.278.5337.467. Science. 1997. PMID: 9334306 Free article.
Cellular analog of differential classical conditioning in Aplysia: disruption by the NMDA receptor antagonist DL-2-amino-5-phosphonovalerate.
Murphy GG, Glanzman DL. Murphy GG, et al. J Neurosci. 1999 Dec 1;19(23):10595-602. doi: 10.1523/JNEUROSCI.19-23-10595.1999. J Neurosci. 1999. PMID: 10575055 Free PMC article.
11 results
You have reached the last page of results. A maximum of 10,000 results are available.
Jump to page
Feedback