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Year Number of Results
1994 2
1995 1
1996 1
1997 1
1999 1
2003 2
2005 1
2006 1
2007 1
2021 0
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Page 1
Learning in Aplysia: looking at synaptic plasticity from both sides.
Roberts AC, Glanzman DL. Roberts AC, et al. Trends Neurosci. 2003 Dec;26(12):662-70. doi: 10.1016/j.tins.2003.09.014. Trends Neurosci. 2003. PMID: 14624850 Review.
The cellular mechanisms of learning in Aplysia: of blind men and elephants.
Glanzman DL. Glanzman DL. Biol Bull. 2006 Jun;210(3):271-9. doi: 10.2307/4134563. Biol Bull. 2006. PMID: 16801500 Review.
The cellular basis of classical conditioning in Aplysia californica--it's less simple than you think.
Glanzman DL. Glanzman DL. Trends Neurosci. 1995 Jan;18(1):30-6. doi: 10.1016/0166-2236(95)93947-v. Trends Neurosci. 1995. PMID: 7535488 Review.
Postsynaptic regulation of the development and long-term plasticity of Aplysia sensorimotor synapses in cell culture.
Glanzman DL. Glanzman DL. J Neurobiol. 1994 Jun;25(6):666-93. doi: 10.1002/neu.480250608. J Neurobiol. 1994. PMID: 8071666 Review.
Hebbian induction of long-term potentiation of Aplysia sensorimotor synapses: partial requirement for activation of an NMDA-related receptor.
Lin XY, Glanzman DL. Lin XY, et al. Proc Biol Sci. 1994 Mar 22;255(1344):215-21. doi: 10.1098/rspb.1994.0031. Proc Biol Sci. 1994. PMID: 7912832
Differential classical conditioning of the gill-withdrawal reflex in Aplysia recruits both NMDA receptor-dependent enhancement and NMDA receptor-dependent depression of the reflex.
Jami SA, Wright WG, Glanzman DL. Jami SA, et al. J Neurosci. 2007 Mar 21;27(12):3064-8. doi: 10.1523/JNEUROSCI.2581-06.2007. J Neurosci. 2007. PMID: 17376967 Free PMC article.
Prolonged habituation of the gill-withdrawal reflex in Aplysia depends on protein synthesis, protein phosphatase activity, and postsynaptic glutamate receptors.
Ezzeddine Y, Glanzman DL. Ezzeddine Y, et al. J Neurosci. 2003 Oct 22;23(29):9585-94. doi: 10.1523/JNEUROSCI.23-29-09585.2003. J Neurosci. 2003. PMID: 14573538 Free PMC article.
Synaptic facilitation and behavioral dishabituation in Aplysia: dependence on release of Ca2+ from postsynaptic intracellular stores, postsynaptic exocytosis, and modulation of postsynaptic AMPA receptor efficacy.
Li Q, Roberts AC, Glanzman DL. Li Q, et al. J Neurosci. 2005 Jun 8;25(23):5623-37. doi: 10.1523/JNEUROSCI.5305-04.2005. J Neurosci. 2005. PMID: 15944390 Free PMC article.
Mediation of classical conditioning in Aplysia californica by long-term potentiation of sensorimotor synapses.
Murphy GG, Glanzman DL. Murphy GG, et al. Science. 1997 Oct 17;278(5337):467-71. doi: 10.1126/science.278.5337.467. Science. 1997. PMID: 9334306 Free article.
Cellular analog of differential classical conditioning in Aplysia: disruption by the NMDA receptor antagonist DL-2-amino-5-phosphonovalerate.
Murphy GG, Glanzman DL. Murphy GG, et al. J Neurosci. 1999 Dec 1;19(23):10595-602. doi: 10.1523/JNEUROSCI.19-23-10595.1999. J Neurosci. 1999. PMID: 10575055 Free PMC article.
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