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Nonsteroidal anti-inflammatory drugs are associated with increased neuritic plaques.
Sonnen JA, Larson EB, Walker RL, Haneuse S, Crane PK, Gray SL, Breitner JC, Montine TJ. Sonnen JA, et al. Neurology. 2010 Sep 28;75(13):1203-10. doi: 10.1212/WNL.0b013e3181f52db1. Epub 2010 Sep 1. Neurology. 2010. PMID: 20811000 Free PMC article.
OBJECTIVES: Observational and experimental studies suggest that nonsteroidal anti-inflammatory drugs (NSAIDs) may protect against Alzheimer disease (AD); however, clinical trials and other observational studies, including the Adult Changes in Thought ( …
OBJECTIVES: Observational and experimental studies suggest that nonsteroidal anti-inflammatory drugs (NSAIDs) ma …
Memory and Brain Amyloid and Tau Effects of a Bioavailable Form of Curcumin in Non-Demented Adults: A Double-Blind, Placebo-Controlled 18-Month Trial.
Small GW, Siddarth P, Li Z, Miller KJ, Ercoli L, Emerson ND, Martinez J, Wong KP, Liu J, Merrill DA, Chen ST, Henning SM, Satyamurthy N, Huang SC, Heber D, Barrio JR. Small GW, et al. Am J Geriatr Psychiatry. 2018 Mar;26(3):266-277. doi: 10.1016/j.jagp.2017.10.010. Epub 2017 Oct 27. Am J Geriatr Psychiatry. 2018. PMID: 29246725 Free article. Clinical Trial.
OBJECTIVE: Because curcumin's anti-inflammatory properties may protect the brain from neurodegeneration, we studied its effect on memory in non-demented adults and explored its impact on brain amyloid and tau accumulation using 2-(1-{6-[(2-[F-18]fluoro …
OBJECTIVE: Because curcumin's anti-inflammatory properties may protect the brain from neurodegeneration, we studied its effect …
Peripheral administration of the soluble TNF inhibitor XPro1595 modifies brain immune cell profiles, decreases beta-amyloid plaque load, and rescues impaired long-term potentiation in 5xFAD mice.
MacPherson KP, Sompol P, Kannarkat GT, Chang J, Sniffen L, Wildner ME, Norris CM, Tansey MG. MacPherson KP, et al. Neurobiol Dis. 2017 Jun;102:81-95. doi: 10.1016/j.nbd.2017.02.010. Epub 2017 Feb 24. Neurobiol Dis. 2017. PMID: 28237313 Free PMC article.
Blocking entry of peripheral macrophages into the CNS was reported to increase amyloid burden in an AD mouse model. To assess inflammation in the 5xFAD (Tg) mouse model, we first quantified central and immune cell profiles in the deep cervical lymph nodes and spleen …
Blocking entry of peripheral macrophages into the CNS was reported to increase amyloid burden in an AD mouse model. To assess …
Aspirin Induces Lysosomal Biogenesis and Attenuates Amyloid Plaque Pathology in a Mouse Model of Alzheimer's Disease via PPARalpha.
Chandra S, Jana M, Pahan K. Chandra S, et al. J Neurosci. 2018 Jul 25;38(30):6682-6699. doi: 10.1523/JNEUROSCI.0054-18.2018. Epub 2018 Jul 2. J Neurosci. 2018. PMID: 29967008 Free PMC article.
Finally, oral administration of low-dose aspirin decreased amyloid plaque pathology in both male and female 5X familial Alzheimer's disease (5XFAD) mice in a PPARalpha-dependent fashion. This study reveals a new function of aspirin in stimulating lysosomal biogenesi …
Finally, oral administration of low-dose aspirin decreased amyloid plaque pathology in both male and female 5X familial Alzhei …
Alzheimer's disease and inflammation: a review of cellular and therapeutic mechanisms.
Halliday G, Robinson SR, Shepherd C, Kril J. Halliday G, et al. Clin Exp Pharmacol Physiol. 2000 Jan-Feb;27(1-2):1-8. doi: 10.1046/j.1440-1681.2000.03200.x. Clin Exp Pharmacol Physiol. 2000. PMID: 10696521 Review.
There is a considerable body of in vitro evidence indicating that the inflammatory response of microglial cells is reduced by non-steroidal anti-inflammatory drugs (NSAID). ...Cyclo-oxygenase 2 enzyme is constitutively expressed in neuron …
There is a considerable body of in vitro evidence indicating that the inflammatory response of microglial cells is reduced by non
Immunological aspects of alzheimer's disease: therapeutic implications.
Hoozemans JJ, Rozemuller AJ, Veerhuis R, Eikelenboom P. Hoozemans JJ, et al. BioDrugs. 2001;15(5):325-37. doi: 10.2165/00063030-200115050-00004. BioDrugs. 2001. PMID: 11437695 Review.
Several lines of evidence indicate that an inflammatory process contributes to the pathology of AD. First, inflammatory proteins have been identified as being associated with neuritic plaques and in glial cells surrounding these plaques. …
Several lines of evidence indicate that an inflammatory process contributes to the pathology of AD. First, inflammatory protei …
Interactions between APP secretases and inflammatory mediators.
Sastre M, Walter J, Gentleman SM. Sastre M, et al. J Neuroinflammation. 2008 Jun 18;5:25. doi: 10.1186/1742-2094-5-25. J Neuroinflammation. 2008. PMID: 18564425 Free PMC article. Review.
There is now a large body of evidence linking inflammation to Alzheimer's disease (AD). This association manifests itself neuropathologically in the presence of activated microglia and astrocytes around neuritic plaques and increased levels of infla
There is now a large body of evidence linking inflammation to Alzheimer's disease (AD). This association manifests itself neuropathol …
Amyloid-beta vaccination, but not nitro-nonsteroidal anti-inflammatory drug treatment, increases vascular amyloid and microhemorrhage while both reduce parenchymal amyloid.
Wilcock DM, Jantzen PT, Li Q, Morgan D, Gordon MN. Wilcock DM, et al. Neuroscience. 2007 Feb 9;144(3):950-60. doi: 10.1016/j.neuroscience.2006.10.020. Epub 2006 Nov 28. Neuroscience. 2007. PMID: 17137722 Free PMC article.
We found that all treatments reduced amyloid deposition, both compact and diffuse, to the same extent while only vaccinated animals, with or without nonsteroidal anti-inflammatory drug (NSAID) treatment, showed increased microglial activation …
We found that all treatments reduced amyloid deposition, both compact and diffuse, to the same extent while only vaccinated animals, …
Alzheimer's disease: etiologies, pathophysiology, cognitive reserve, and treatment opportunities.
Cummings JL, Vinters HV, Cole GM, Khachaturian ZS. Cummings JL, et al. Neurology. 1998 Jul;51(1 Suppl 1):S2-17; discussion S65-7. doi: 10.1212/wnl.51.1_suppl_1.s2. Neurology. 1998. PMID: 9674758 Review.
All mutations known to cause AD increase the production of beta-amyloid peptide. This protein is derived from amyloid precursor protein and, when aggregated in a beta-pleated sheet configuration, is neurotoxic and forms the core of neuritic plaques
All mutations known to cause AD increase the production of beta-amyloid peptide. This protein is derived from amyloid p …
Assessment of diets containing curcumin, epigallocatechin-3-gallate, docosahexaenoic acid and alpha-lipoic acid on amyloid load and inflammation in a male transgenic mouse model of Alzheimer's disease: Are combinations more effective?
Sharman MJ, Gyengesi E, Liang H, Chatterjee P, Karl T, Li QX, Wenk MR, Halliwell B, Martins RN, Münch G. Sharman MJ, et al. Neurobiol Dis. 2019 Apr;124:505-519. doi: 10.1016/j.nbd.2018.11.026. Epub 2019 Jan 2. Neurobiol Dis. 2019. PMID: 30610916
A curcumin only diet was shown to lower amyloid plaque load (p = .028), but when combined with EGCG, DHA and ALA did not result in further decreases in amyloid plaque load. ...In the amygdala associated cued test, all mice demonstrated the abili …
A curcumin only diet was shown to lower amyloid plaque load (p = .028), but when combined with EGCG, DHA and ALA did not resul …
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