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Development of lupus-like autoimmune diseases by disruption of the PD-1 gene encoding an ITIM motif-carrying immunoreceptor.
Nishimura H, Nose M, Hiai H, Minato N, Honjo T. Nishimura H, et al. Among authors: nose m. Immunity. 1999 Aug;11(2):141-51. doi: 10.1016/s1074-7613(00)80089-8. Immunity. 1999. PMID: 10485649
Enhanced expression of the soluble form of E-selectin attenuates progression of lupus nephritis and vasculitis in MRL/lpr mice.
Nakatani K, Yoshimoto S, Asai O, Sakan H, Terada M, Saito Y, Nose M, Iwano M, Konishi N. Nakatani K, et al. Among authors: nose m. Immun Inflamm Dis. 2013 Oct;1(1):37-46. doi: 10.1002/iid3.6. Epub 2013 Oct 30. Immun Inflamm Dis. 2013. PMID: 25400916 Free PMC article.
Association between E-selectin expression and histopathological modification of glomerular lesions by non-nephritogenic IgM antibodies in experimental lupus nephritis.
Nakatani K, Asai O, Sakan H, Yoshimoto S, Terada M, Nose M, Iwano M, Konishi N. Nakatani K, et al. Among authors: nose m. Mod Rheumatol. 2014 Sep;24(5):808-15. doi: 10.3109/14397595.2013.868070. Epub 2013 Dec 29. Mod Rheumatol. 2014. PMID: 24372297
Dcir deficiency causes development of autoimmune diseases in mice due to excess expansion of dendritic cells.
Fujikado N, Saijo S, Yonezawa T, Shimamori K, Ishii A, Sugai S, Kotaki H, Sudo K, Nose M, Iwakura Y. Fujikado N, et al. Among authors: nose m. Nat Med. 2008 Feb;14(2):176-80. doi: 10.1038/nm1697. Epub 2008 Jan 20. Nat Med. 2008. PMID: 18204462
A bacterial artificial chromosome transgene with polymorphic Cd72 inhibits the development of glomerulonephritis and vasculitis in MRL-Faslpr lupus mice.
Oishi H, Tsubaki T, Miyazaki T, Ono M, Nose M, Takahashi S. Oishi H, et al. Among authors: nose m. J Immunol. 2013 Mar 1;190(5):2129-37. doi: 10.4049/jimmunol.1202196. Epub 2013 Jan 30. J Immunol. 2013. PMID: 23365086 Free PMC article.
Autosomal loci associated with a sex-related difference in the development of autoimmune phenotypes in a lupus model.
Misu N, Zhang M, Mori S, Miyazaki T, Furukawa H, Sasaki T, Nose M, Ono M. Misu N, et al. Among authors: nose m. Eur J Immunol. 2007 Oct;37(10):2787-96. doi: 10.1002/eji.200637016. Eur J Immunol. 2007. PMID: 17823981
Nrf2 deficiency improves autoimmune nephritis caused by the fas mutation lpr.
Morito N, Yoh K, Hirayama A, Itoh K, Nose M, Koyama A, Yamamoto M, Takahashi S. Morito N, et al. Among authors: nose m. Kidney Int. 2004 May;65(5):1703-13. doi: 10.1111/j.1523-1755.2004.00565.x. Kidney Int. 2004. PMID: 15086909
Bone marrow cell transfer of autoimmune diseases in a MRL strain of mice with a deficit in functional Fas ligand: dissociation of arteritis from glomerulonephritis.
Ito MR, Ono M, Itoh J, Nose M. Ito MR, et al. Among authors: nose m. Pathol Int. 2003 Aug;53(8):518-24. doi: 10.1046/j.1440-1827.2003.01516.x. Pathol Int. 2003. PMID: 12895230
Suppression of experimental lupus nephritis by aberrant expression of the soluble E-selectin gene.
Takahashi S, Araki K, Araki M, Ito MR, Nakatani K, Fujii H, Izui S, Vassalli P, Nose M. Takahashi S, et al. Among authors: nose m. Pathol Int. 2002 Mar;52(3):175-80. doi: 10.1046/j.1440-1827.2002.01337.x. Pathol Int. 2002. PMID: 11972860
Constitutive OX40/OX40 ligand interaction induces autoimmune-like diseases.
Murata K, Nose M, Ndhlovu LC, Sato T, Sugamura K, Ishii N. Murata K, et al. Among authors: nose m. J Immunol. 2002 Oct 15;169(8):4628-36. doi: 10.4049/jimmunol.169.8.4628. J Immunol. 2002. PMID: 12370402
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