Parris GE - Search Results

1

Cell-cell fusion and cancer: germ-cell cancer and teratomas.

Parris GE. Parris GE. Med Hypotheses. 2009 Feb;72(2):226. doi: 10.1016/j.mehy.2008.11.001. Epub 2008 Dec 11. Med Hypotheses. 2009. PMID: 19084344 No abstract available.

2

Chromosomal instability (CIN) leads to clone extinction, not cancer.

Parris GE. Parris GE. Med Hypotheses. 2008 Dec;71(6):983. doi: 10.1016/j.mehy.2008.07.046. Epub 2008 Sep 9. Med Hypotheses. 2008. PMID: 18783893 No abstract available.

3

2-deoxy-D-glucose as a potential drug against fusogenic viruses including HIV.

Parris GE. Parris GE. Med Hypotheses. 2008;70(4):776-82. doi: 10.1016/j.mehy.2007.08.021. Epub 2007 Oct 24. Med Hypotheses. 2008. PMID: 17920779

4

Anti-fusion drug candidates that may have beneficial effects against cancer, virus infections and autoimmune disorders.

Parris GE. Parris GE. Med Hypotheses. 2008;70(2):463. doi: 10.1016/j.mehy.2007.06.003. Epub 2007 Jul 25. Med Hypotheses. 2008. PMID: 17656030 No abstract available.

5

The cell clone ecology hypothesis and the cell fusion model of cancer progression and metastasis: history and experimental support.

Parris GE. Parris GE. Med Hypotheses. 2006;66(1):76-83. doi: 10.1016/j.mehy.2005.08.009. Epub 2005 Sep 19. Med Hypotheses. 2006. PMID: 16169667

6

Clinically significant cancer evolves from transient mutated and/or aneuploid neoplasia by cell fusion to form unstable syncytia that give rise to ecologically viable parasite species.

Parris GE. Parris GE. Med Hypotheses. 2005;65(5):846-50. doi: 10.1016/j.mehy.2005.05.036. Med Hypotheses. 2005. PMID: 16023792

7

The role of viruses in cell fusion and its importance to evolution, invasion and metastasis of cancer clones.

Parris GE. Parris GE. Med Hypotheses. 2005;64(5):1011-4. doi: 10.1016/j.mehy.2004.11.012. Med Hypotheses. 2005. PMID: 15780502

8

Some experimental observations consistent with cell fusion hypothesis of cancer evolution.

Parris G. Parris G. Med Hypotheses. 2005;65(5):993-4. doi: 10.1016/j.mehy.2005.06.004. Med Hypotheses. 2005. PMID: 16039068 No abstract available.

9

The cell clone ecology hypothesis and the cell fusion model of cancer progression and metastasis (II): three pathways for spontaneous cell-cell fusion and escape from the intercellular matrix.

Parris G. Parris G. Med Hypotheses. 2006;67(1):172-6. doi: 10.1016/j.mehy.2006.01.018. Epub 2006 Mar 3. Med Hypotheses. 2006. PMID: 16516400

Unsymmetrical chromosome segregation during mitosis of normal or mutated cells produces aneuploid cells and also contributes to the evolution of neoplasia. However, it has been pointed out (Parris GE. Med Hypotheses 2005;65:993-4 and 2006;66:76-83) that DNA damage a …

Unsymmetrical chromosome segregation during mitosis of normal or mutated cells produces aneuploid cells and also contributes to the evolutio …

10

Scope of medical implications of the Master Development Program hypothesis.

Parris GE. Parris GE. Med Hypotheses. 2010 May;74(5):953. doi: 10.1016/j.mehy.2009.12.001. Epub 2009 Dec 23. Med Hypotheses. 2010. PMID: 20031335 No abstract available.

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