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c-MYC: more than just a matter of life and death.
Pelengaris S, Khan M, Evan G. Pelengaris S, et al. Nat Rev Cancer. 2002 Oct;2(10):764-76. doi: 10.1038/nrc904. Nat Rev Cancer. 2002. PMID: 12360279 Review.
Oncogenic co-operation in beta-cell tumorigenesis.
Pelengaris S, Khan M. Pelengaris S, et al. Endocr Relat Cancer. 2001 Dec;8(4):307-14. doi: 10.1677/erc.0.0080307. Endocr Relat Cancer. 2001. PMID: 11733227 Review.
Action of Myc in vivo - proliferation and apoptosis.
Pelengaris S, Rudolph B, Littlewood T. Pelengaris S, et al. Curr Opin Genet Dev. 2000 Feb;10(1):100-5. doi: 10.1016/s0959-437x(99)00046-5. Curr Opin Genet Dev. 2000. PMID: 10679391 Review.
c-Myc and downstream targets in the pathogenesis and treatment of cancer.
Robson S, Pelengaris S, Khan M. Robson S, et al. Recent Pat Anticancer Drug Discov. 2006 Nov;1(3):305-26. doi: 10.2174/157489206778776934. Recent Pat Anticancer Drug Discov. 2006. PMID: 18221043 Review.
Brief inactivation of c-Myc is not sufficient for sustained regression of c-Myc-induced tumours of pancreatic islets and skin epidermis.
Pelengaris S, Abouna S, Cheung L, Ifandi V, Zervou S, Khan M. Pelengaris S, et al. BMC Biol. 2004 Dec 21;2:26. doi: 10.1186/1741-7007-2-26. BMC Biol. 2004. PMID: 15613240 Free PMC article.
Intriguingly, transient c-Myc inactivation proved sufficient for sustained osteosarcoma regression; the resulting osteocyte differentiation potentially explaining loss of c-Myc's oncogenic properties. ...
Intriguingly, transient c-Myc inactivation proved sufficient for sustained osteosarcoma regression; the resulting osteocyte differentiation …
The many faces of c-MYC.
Pelengaris S, Khan M. Pelengaris S, et al. Arch Biochem Biophys. 2003 Aug 15;416(2):129-36. doi: 10.1016/s0003-9861(03)00294-7. Arch Biochem Biophys. 2003. PMID: 12893289 Review.
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