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BCR-ABL gene mutations in relation to clinical resistance of Philadelphia-chromosome-positive leukaemia to STI571: a prospective study.
von Bubnoff N, Schneller F, Peschel C, Duyster J. von Bubnoff N, et al. Among authors: peschel c. Lancet. 2002 Feb 9;359(9305):487-91. doi: 10.1016/S0140-6736(02)07679-1. Lancet. 2002. PMID: 11853795
The Bcr-Abl mutations T315I and Y253H do not confer a growth advantage in the absence of imatinib.
Miething C, Feihl S, Mugler C, Grundler R, von Bubnoff N, Lordick F, Peschel C, Duyster J. Miething C, et al. Among authors: peschel c. Leukemia. 2006 Apr;20(4):650-7. doi: 10.1038/sj.leu.2404151. Leukemia. 2006. PMID: 16482207
Mechanisms of apoptosis-induction by rottlerin: therapeutic implications for B-CLL.
Ringshausen I, Oelsner M, Weick K, Bogner C, Peschel C, Decker T. Ringshausen I, et al. Among authors: peschel c. Leukemia. 2006 Mar;20(3):514-20. doi: 10.1038/sj.leu.2404113. Leukemia. 2006. PMID: 16437144
Phosphorylation of tyrosine 393 in the kinase domain of Bcr-Abl influences the sensitivity towards imatinib in vivo.
Miething C, Mugler C, Grundler R, Hoepfl J, Bai RY, Peschel C, Duyster J. Miething C, et al. Among authors: peschel c. Leukemia. 2003 Sep;17(9):1695-9. doi: 10.1038/sj.leu.2403040. Leukemia. 2003. PMID: 12970766
Grb4/Nckbeta acts as a nuclear repressor of v-Abl-induced transcription from c-jun/c-fos promoter elements.
Jahn T, Seipel P, Coutinho S, Miething C, Peschel C, Duyster J. Jahn T, et al. Among authors: peschel c. J Biol Chem. 2001 Nov 16;276(46):43419-27. doi: 10.1074/jbc.M107030200. Epub 2001 Aug 20. J Biol Chem. 2001. PMID: 11514578
Nucleophosmin-anaplastic lymphoma kinase associated with anaplastic large-cell lymphoma activates the phosphatidylinositol 3-kinase/Akt antiapoptotic signaling pathway.
Bai RY, Ouyang T, Miething C, Morris SW, Peschel C, Duyster J. Bai RY, et al. Among authors: peschel c. Blood. 2000 Dec 15;96(13):4319-27. Blood. 2000. PMID: 11110708
Nucleophosmin-anaplastic lymphoma kinase of large-cell anaplastic lymphoma is a constitutively active tyrosine kinase that utilizes phospholipase C-gamma to mediate its mitogenicity.
Bai RY, Dieter P, Peschel C, Morris SW, Duyster J. Bai RY, et al. Among authors: peschel c. Mol Cell Biol. 1998 Dec;18(12):6951-61. doi: 10.1128/mcb.18.12.6951. Mol Cell Biol. 1998. PMID: 9819383 Free PMC article.
Characterization of Ggrb4, an adapter protein interacting with Bcr-Abl.
Coutinho S, Jahn T, Lewitzky M, Feller S, Hutzler P, Peschel C, Duyster J. Coutinho S, et al. Among authors: peschel c. Blood. 2000 Jul 15;96(2):618-24. Blood. 2000. PMID: 10887126
Bcr-Abl kinase promotes cell cycle entry of primary myeloid CML cells in the absence of growth factors.
Jonuleit T, Peschel C, Schwab R, van der Kuip H, Buchdunger E, Fischer T, Huber C, Aulitzky WE. Jonuleit T, et al. Among authors: peschel c. Br J Haematol. 1998 Feb;100(2):295-303. doi: 10.1046/j.1365-2141.1998.00564.x. Br J Haematol. 1998. PMID: 9488616
Primary proliferating immature myeloid cells from CML patients are not resistant to induction of apoptosis by DNA damage and growth factor withdrawal.
Albrecht T, Schwab R, Henkes M, Peschel C, Huber C, Aulitzky WE. Albrecht T, et al. Among authors: peschel c. Br J Haematol. 1996 Dec;95(3):501-7. doi: 10.1046/j.1365-2141.1996.d01-1934.x. Br J Haematol. 1996. PMID: 8943891
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