Petasis NA - Search Results

1

COX-2 inhibition is neither necessary nor sufficient for celecoxib to suppress tumor cell proliferation and focus formation in vitro.

Chuang HC, Kardosh A, Gaffney KJ, Petasis NA, Schönthal AH. Chuang HC, et al. Among authors: petasis na. Mol Cancer. 2008 May 16;7:38. doi: 10.1186/1476-4598-7-38. Mol Cancer. 2008. PMID: 18485224 Free PMC article.

2

Dimethyl-celecoxib (DMC), a derivative of celecoxib that lacks cyclooxygenase-2-inhibitory function, potently mimics the anti-tumor effects of celecoxib on Burkitt's lymphoma in vitro and in vivo.

Kardosh A, Wang W, Uddin J, Petasis NA, Hofman FM, Chen TC, Schönthal AH. Kardosh A, et al. Among authors: petasis na. Cancer Biol Ther. 2005 May;4(5):571-82. doi: 10.4161/cbt.4.5.1699. Epub 2005 May 5. Cancer Biol Ther. 2005. PMID: 15846081

3

Multitarget inhibition of drug-resistant multiple myeloma cell lines by dimethyl-celecoxib (DMC), a non-COX-2 inhibitory analog of celecoxib.

Kardosh A, Soriano N, Liu YT, Uddin J, Petasis NA, Hofman FM, Chen TC, Schönthal AH. Kardosh A, et al. Among authors: petasis na. Blood. 2005 Dec 15;106(13):4330-8. doi: 10.1182/blood-2005-07-2819. Epub 2005 Aug 25. Blood. 2005. PMID: 16123214 Free article.

4

Dimethyl celecoxib as a novel non-cyclooxygenase 2 therapy in the treatment of non-small cell lung cancer.

Backhus LM, Petasis NA, Uddin J, Schönthal AH, Bart RD, Lin Y, Starnes VA, Bremner RM. Backhus LM, et al. Among authors: petasis na. J Thorac Cardiovasc Surg. 2005 Nov;130(5):1406-12. doi: 10.1016/j.jtcvs.2005.07.018. Epub 2005 Oct 13. J Thorac Cardiovasc Surg. 2005. PMID: 16256796 Free article.

5

Downregulation of survivin expression and concomitant induction of apoptosis by celecoxib and its non-cyclooxygenase-2-inhibitory analog, dimethyl-celecoxib (DMC), in tumor cells in vitro and in vivo.

Pyrko P, Soriano N, Kardosh A, Liu YT, Uddin J, Petasis NA, Hofman FM, Chen CS, Chen TC, Schönthal AH. Pyrko P, et al. Among authors: petasis na. Mol Cancer. 2006 May 18;5:19. doi: 10.1186/1476-4598-5-19. Mol Cancer. 2006. PMID: 16707021 Free PMC article.

6

Calcium-activated endoplasmic reticulum stress as a major component of tumor cell death induced by 2,5-dimethyl-celecoxib, a non-coxib analogue of celecoxib.

Pyrko P, Kardosh A, Liu YT, Soriano N, Xiong W, Chow RH, Uddin J, Petasis NA, Mircheff AK, Farley RA, Louie SG, Chen TC, Schönthal AH. Pyrko P, et al. Among authors: petasis na. Mol Cancer Ther. 2007 Apr;6(4):1262-75. doi: 10.1158/1535-7163.MCT-06-0629. Mol Cancer Ther. 2007. PMID: 17431104

7

Celecoxib analogs that lack COX-2 inhibitory function: preclinical development of novel anticancer drugs.

Schönthal AH, Chen TC, Hofman FM, Louie SG, Petasis NA. Schönthal AH, et al. Among authors: petasis na. Expert Opin Investig Drugs. 2008 Feb;17(2):197-208. doi: 10.1517/13543784.17.2.197. Expert Opin Investig Drugs. 2008. PMID: 18230053 Review.

8

Aggravated endoplasmic reticulum stress as a basis for enhanced glioblastoma cell killing by bortezomib in combination with celecoxib or its non-coxib analogue, 2,5-dimethyl-celecoxib.

Kardosh A, Golden EB, Pyrko P, Uddin J, Hofman FM, Chen TC, Louie SG, Petasis NA, Schönthal AH. Kardosh A, et al. Among authors: petasis na. Cancer Res. 2008 Feb 1;68(3):843-51. doi: 10.1158/0008-5472.CAN-07-5555. Cancer Res. 2008. PMID: 18245486

9

Green tea polyphenols block the anticancer effects of bortezomib and other boronic acid-based proteasome inhibitors.

Golden EB, Lam PY, Kardosh A, Gaffney KJ, Cadenas E, Louie SG, Petasis NA, Chen TC, Schönthal AH. Golden EB, et al. Among authors: petasis na. Blood. 2009 Jun 4;113(23):5927-37. doi: 10.1182/blood-2008-07-171389. Epub 2009 Feb 3. Blood. 2009. PMID: 19190249 Free article.

10

Enhanced killing of chemo-resistant breast cancer cells via controlled aggravation of ER stress.

Cho HY, Thomas S, Golden EB, Gaffney KJ, Hofman FM, Chen TC, Louie SG, Petasis NA, Schönthal AH. Cho HY, et al. Among authors: petasis na. Cancer Lett. 2009 Sep 8;282(1):87-97. doi: 10.1016/j.canlet.2009.03.007. Epub 2009 Apr 3. Cancer Lett. 2009. PMID: 19345476

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