Skip to main page content
Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

MyNCBI Filters
Text availability
Article attribute
Article type
Publication date

Search Results

178 results
Filters applied: . Clear all Results are displayed in a computed author sort order. Results by year timeline is unavailable
Page 1
Toxic Electrophiles Induce Expression of the Multidrug Efflux Pump MexEF-OprN in Pseudomonas aeruginosa through a Novel Transcriptional Regulator, CmrA.
Juarez P, Jeannot K, Plésiat P, Llanes C. Juarez P, et al. Among authors: Plesiat P. Antimicrob Agents Chemother. 2017 Jul 25;61(8):e00585-17. doi: 10.1128/AAC.00585-17. Print 2017 Aug. Antimicrob Agents Chemother. 2017. PMID: 28507116 Free PMC article.
Transcriptomic experiments demonstrated that CmrA positively regulates a small set of 11 genes, including PA14_38020 (homolog of PA2048), which is required for the MexS/T-dependent activation of mexEF-oprN PA2048 codes for a protein sharing conserved domains with the quinol monooxygenase YgiN from Escherichia coli Interestingly, exposure of strain PA14 to toxic electrophilic molecules (glyoxal, methylglyoxal, and cinnamaldehyde) strongly activates the CmrA pathway and upregulates MexEF-OprN and, thus, increases the resistance of P. aeruginosa to the pump substrates. ...
Transcriptomic experiments demonstrated that CmrA positively regulates a small set of 11 genes, including PA14_38020 (homolog of PA2048), wh …
[Pseudomonas aeruginosa infection in cystic fibrosis: predominance of a single strain and the influence of antibiotic therapy and the environment].
Michel-Briand Y, Plesiat P, Godard C, Noir A, Estavoyer JM. Michel-Briand Y, et al. Among authors: Plesiat P. Bull Acad Natl Med. 1992 Nov;176(8):1159-69; discussion 1170-1. Bull Acad Natl Med. 1992. PMID: 1300222 French.
Swabs were taken from the environments of infected patients and were tested for P. aeruginosa: this bacteria was found in three sites, and two of these contained an isolate with the same pulsotype as the strain responsible for the infection, whereas no P. aeruginosa was detected in the environment of an uninfected patient. ...
Swabs were taken from the environments of infected patients and were tested for P. aeruginosa: this bacteria was found in three sites …
Cloning, sequencing, and expression of the Pseudomonas testosteroni gene encoding 3-oxosteroid delta 1-dehydrogenase.
Plesiat P, Grandguillot M, Harayama S, Vragar S, Michel-Briand Y. Plesiat P, et al. J Bacteriol. 1991 Nov;173(22):7219-27. doi: 10.1128/jb.173.22.7219-7227.1991. J Bacteriol. 1991. PMID: 1657885 Free PMC article.
The induced level was about 40 times higher than the induced level in P. testosteroni. Delta 1-Dehydrogenase synthesized in E. coli was localized in the inner membrane fraction. ...
The induced level was about 40 times higher than the induced level in P. testosteroni. Delta 1-Dehydrogenase synthesized in E. coli w …
Prevalence and profiles of plasmids in Pseudomonas aeruginosa.
Plesiat P, Alkhalaf B, Michel-Briand Y. Plesiat P, et al. Eur J Clin Microbiol Infect Dis. 1988 Apr;7(2):261-4. doi: 10.1007/bf01963098. Eur J Clin Microbiol Infect Dis. 1988. PMID: 3134228
[Pseudomonas aeruginosa septicemia. Host-related risk factors in 82 episodes].
Roche O, Beuhorry-Sassus F, Boillot A, Dupont MJ, Plésiat P, Talon D, Cahn JY, Michel-Briand Y. Roche O, et al. Among authors: Plesiat P. Presse Med. 1995 Sep 2-9;24(25):1164-6. Presse Med. 1995. PMID: 7567833 French.
With univariate analysis body temperature below 38,5 degrees C was significant (p = 0.007) for death at day 2 and appropriate antibiotic treatment after diagnosis was significant (p < 0.001) for absence of death on day 2. For multivariate analysis, chemotherapy and shock syndrome were significant (p = 0.005 and 0.09 respectively) for death at day 2 and appropriate antibiotic treatment was significant (p = 0.005) for absence of death on day 2. ...
With univariate analysis body temperature below 38,5 degrees C was significant (p = 0.007) for death at day 2 and appropriate antibio …
OprK and OprM define two genetically distinct multidrug efflux systems in Pseudomonas aeruginosa.
Hamzehpour MM, Pechere JC, Plesiat P, Köhler T. Hamzehpour MM, et al. Among authors: Plesiat P. Antimicrob Agents Chemother. 1995 Nov;39(11):2392-6. doi: 10.1128/aac.39.11.2392. Antimicrob Agents Chemother. 1995. PMID: 8585714 Free PMC article.
These experiments demonstrated the existence of two genetically distinct efflux systems in P. aeruginosa. The identities of the operons encoding the two efflux systems and their physiological roles are discussed....
These experiments demonstrated the existence of two genetically distinct efflux systems in P. aeruginosa. The identities of the opero …
Multidrug efflux in intrinsic resistance to trimethoprim and sulfamethoxazole in Pseudomonas aeruginosa.
Köhler T, Kok M, Michea-Hamzehpour M, Plesiat P, Gotoh N, Nishino T, Curty LK, Pechere JC. Köhler T, et al. Among authors: Plesiat P. Antimicrob Agents Chemother. 1996 Oct;40(10):2288-90. Antimicrob Agents Chemother. 1996. PMID: 9036831 Free PMC article.
These results demonstrate that the mexABoprM multidrug efflux system is mainly responsible for the intrinsic resistance of P. aeruginosa to TMP and SMX....
These results demonstrate that the mexABoprM multidrug efflux system is mainly responsible for the intrinsic resistance of P. aerugin …
178 results
Jump to page
Feedback