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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
2011 2
2012 1
2013 1
2014 5
2015 5
2017 4
2018 2
2019 2
2021 1
2022 2
2023 1
2024 1

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Sequencing the next generation of glioblastomas.
Jovčevska I. Jovčevska I. Crit Rev Clin Lab Sci. 2018 Jun;55(4):264-282. doi: 10.1080/10408363.2018.1462759. Epub 2018 Apr 18. Crit Rev Clin Lab Sci. 2018. PMID: 29668342 Review.
The most aggressive brain malignancy, glioblastoma, accounts for 60-70% of all gliomas and is uniformly fatal. According to the molecular signature, glioblastoma is divided into four subtypes (proneural, neural, classical, and mesenchymal), each with its own …
The most aggressive brain malignancy, glioblastoma, accounts for 60-70% of all gliomas and is uniformly fatal. According to the molec …
The adaptive transition of glioblastoma stem cells and its implications on treatments.
Wang Z, Zhang H, Xu S, Liu Z, Cheng Q. Wang Z, et al. Signal Transduct Target Ther. 2021 Mar 23;6(1):124. doi: 10.1038/s41392-021-00491-w. Signal Transduct Target Ther. 2021. PMID: 33753720 Free PMC article. Review.
Previous studies reported two subtypes, proneural and mesenchymal, of glioblastoma stem cells manifesting different sensitivity to radiotherapy or chemotherapy. ...In this review, we summarized the basic characteristics, adaptive transition and implications of gl
Previous studies reported two subtypes, proneural and mesenchymal, of glioblastoma stem cells manifesting different sensitivit …
Biomarkers of glioblastoma multiforme.
Nicolaidis S. Nicolaidis S. Metabolism. 2015 Mar;64(3 Suppl 1):S22-7. doi: 10.1016/j.metabol.2014.10.031. Epub 2014 Oct 30. Metabolism. 2015. PMID: 25468141 Review.
Glioblastoma multiforme (GBM) is the most common and lethal primary malignancy of the central nervous system. ...Aberrant gene expression and copy number alterations make it possible to identify four subtypes: classical, mesenchymal, proneural, and neural. More and
Glioblastoma multiforme (GBM) is the most common and lethal primary malignancy of the central nervous system. ...Aberrant gene expres
KCa3.1 Channels and Glioblastoma: In Vitro Studies.
Klumpp L, Sezgin EC, Skardelly M, Eckert F, Huber SM. Klumpp L, et al. Curr Neuropharmacol. 2018;16(5):627-635. doi: 10.2174/1570159X15666170808115821. Curr Neuropharmacol. 2018. PMID: 28786347 Free PMC article. Review.
METHOD: PubMed was searched in order to summarize our current knowledge on the molecular signaling upstream and downstream and the effector functions of KCa3.1 channel activity in tumor cells in general and in glioblastoma cells in particular. In addition, KCa3.1 expressio …
METHOD: PubMed was searched in order to summarize our current knowledge on the molecular signaling upstream and downstream and the effector …
Brain Tumors of Glial Origin.
Waker CA, Lober RM. Waker CA, et al. Adv Exp Med Biol. 2019;1190:281-297. doi: 10.1007/978-981-32-9636-7_18. Adv Exp Med Biol. 2019. PMID: 31760651 Review.
Isocitrate dehydrogenase (IDH) mutation is an early event in the formation of some diffuse gliomas, and is the best understood mechanism of their epigenetic dysregulation. Glioblastoma may evolve from lower-grade lesions with IDH mutations, or arise independently from copy …
Isocitrate dehydrogenase (IDH) mutation is an early event in the formation of some diffuse gliomas, and is the best understood mechanism of …
Established and emerging variants of glioblastoma multiforme: review of morphological and molecular features.
Karsy M, Gelbman M, Shah P, Balumbu O, Moy F, Arslan E. Karsy M, et al. Folia Neuropathol. 2012;50(4):301-21. doi: 10.5114/fn.2012.32361. Folia Neuropathol. 2012. PMID: 23319187 Free article. Review.
Since the recent publication of the World Health Organization brain tumour classification guidelines in 2007, a significant expansion in the molecular understanding of glioblastoma multiforme (GBM) and its pathological as well as genomic variants has been evident. The purp …
Since the recent publication of the World Health Organization brain tumour classification guidelines in 2007, a significant expansion in the …
Current Insights into Mesenchymal Signatures in Glioblastoma.
Matsumoto Y, Ichikawa T, Kurozumi K, Date I. Matsumoto Y, et al. Acta Med Okayama. 2022 Oct;76(5):489-502. doi: 10.18926/AMO/64024. Acta Med Okayama. 2022. PMID: 36352795 Free article. Review.
Glioblastoma (GBM) is a fatal primary malignant brain tumor in adults. ...GBM-intrinsic transcriptional profiling has suggested the presence of at least three subtypes of GBM: the proneural, classic, and mesenchymal subtypes. The mesenchymal subtype is the most aggr
Glioblastoma (GBM) is a fatal primary malignant brain tumor in adults. ...GBM-intrinsic transcriptional profiling has suggested the p
A Comprehensive Review of Genomics and Noncoding RNA in Gliomas.
Hassan A, Mosley J, Singh S, Zinn PO. Hassan A, et al. Top Magn Reson Imaging. 2017 Feb;26(1):3-14. doi: 10.1097/RMR.0000000000000111. Top Magn Reson Imaging. 2017. PMID: 28079712 Review.
Glioblastoma (GBM) is the most malignant primary adult brain tumor. In spite of our greater understanding of the biology of GBMs, clinical outcome of GBM patients remains poor, as their median survival with best available treatment is 12 to 18 months. Recent efforts of The
Glioblastoma (GBM) is the most malignant primary adult brain tumor. In spite of our greater understanding of the biology of GBMs, cli
Isocitrate dehydrogenase status and molecular subclasses of glioma and glioblastoma.
Agnihotri S, Aldape KD, Zadeh G. Agnihotri S, et al. Neurosurg Focus. 2014 Dec;37(6):E13. doi: 10.3171/2014.9.FOCUS14505. Neurosurg Focus. 2014. PMID: 25434382 Review.
Diffuse gliomas and secondary glioblastomas (GBMs) that develop from low-grade gliomas are a common and incurable class of brain tumor. Mutations in the metabolic enzyme glioblastomas (IDH1) represent a distinguishing feature of low-grade gliomas and secondary GBMs. …
Diffuse gliomas and secondary glioblastomas (GBMs) that develop from low-grade gliomas are a common and incurable class of brain tumo …
Brain neoplasms and coagulation.
Magnus N, D'Asti E, Garnier D, Meehan B, Rak J. Magnus N, et al. Semin Thromb Hemost. 2013 Nov;39(8):881-95. doi: 10.1055/s-0033-1357483. Epub 2013 Oct 9. Semin Thromb Hemost. 2013. PMID: 24108471 Review.
Chronic activation of the coagulation system is particularly obvious in glioblastoma multiforme (GBM), where intratumoral vasoocclusive thrombosis may contribute to hypoxia, pseudopalisading necrosis, and angiogenesis. ...Discovery of distinct oncogenic networks led to rec …
Chronic activation of the coagulation system is particularly obvious in glioblastoma multiforme (GBM), where intratumoral vasoocclusi …
24 results