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c-Rel in Epidermal Homeostasis: A Spotlight on c-Rel in Cell Cycle Regulation.
Lorenz VN, Schön MP, Seitz CS. Lorenz VN, et al. J Invest Dermatol. 2016 Jun;136(6):1090-1096. doi: 10.1016/j.jid.2016.02.003. Epub 2016 Mar 29. J Invest Dermatol. 2016. PMID: 27032306 Free article. Review.
While differentiation takes place in the suprabasal layers, proliferation, including mitosis, is usually restricted to the basal layer. Only recently identified as an important regulator of epidermal homeostasis, c-Rel, an NF-kappaB transcription factor subunit, aff …
While differentiation takes place in the suprabasal layers, proliferation, including mitosis, is usually restricted to the basal layer. Only …
The c-Rel subunit of NF-kappaB regulates epidermal homeostasis and promotes skin fibrosis in mice.
Fullard N, Moles A, O'Reilly S, van Laar JM, Faini D, Diboll J, Reynolds NJ, Mann DA, Reichelt J, Oakley F. Fullard N, et al. Am J Pathol. 2013 Jun;182(6):2109-20. doi: 10.1016/j.ajpath.2013.02.016. Epub 2013 Apr 4. Am J Pathol. 2013. PMID: 23562440 Free PMC article.
We show that the primary defect in both Nfkb1(-/-) and c-Rel(-/-) mice is within keratinocytes that display reduced proliferation both in vitro and in vivo. ...Taken together, our data reveal a role for p50 and c-Rel in regulating epidermal proliferati …
We show that the primary defect in both Nfkb1(-/-) and c-Rel(-/-) mice is within keratinocytes that display reduced proliferat …
The c-Rel subunit of NF-kappaB is a crucial regulator of phenotype and motility of HaCaT keratinocytes.
Lorenz VN, Schön MP, Seitz CS. Lorenz VN, et al. Arch Dermatol Res. 2015 Aug;307(6):523-30. doi: 10.1007/s00403-015-1562-2. Epub 2015 Apr 5. Arch Dermatol Res. 2015. PMID: 25842167
To further study the impact of c-Rel on essential keratinocyte features such as migration and epithelial morphology, c-Rel was down-regulated in HaCaT keratinocytes by a siRNA approach. This inhibition of c-Rel impaired the keratinocyte-t …
To further study the impact of c-Rel on essential keratinocyte features such as migration and epithelial morphology, c-Rel
Cutting edge: NF-kappaB p65 and c-Rel control epidermal development and immune homeostasis in the skin.
Grinberg-Bleyer Y, Dainichi T, Oh H, Heise N, Klein U, Schmid RM, Hayden MS, Ghosh S. Grinberg-Bleyer Y, et al. J Immunol. 2015 Mar 15;194(6):2472-6. doi: 10.4049/jimmunol.1402608. Epub 2015 Feb 13. J Immunol. 2015. PMID: 25681334 Free PMC article.
However, the role of NF-kappaB in psoriasis remains controversial. We analyzed the function of canonical NF-kappaB in the epidermis using CRE-mediated deletion of p65 and c-Rel in keratinocytes. In contrast to animals lacking p65 or c-Rel alone, mice l …
However, the role of NF-kappaB in psoriasis remains controversial. We analyzed the function of canonical NF-kappaB in the epidermis u …
Intersection of the p63 and NF-κB pathways in epithelial homeostasis and disease.
King KE, George AL, Sakakibara N, Mahmood K, Moses MA, Weinberg WC. King KE, et al. Mol Carcinog. 2019 Sep;58(9):1571-1580. doi: 10.1002/mc.23081. Epub 2019 Jul 8. Mol Carcinog. 2019. PMID: 31286584 Review.
Overexpression of deltaNp63alpha, a member of the p53/p63/p73 family of transcription factors, is a molecular attribute of human squamous cancers of the head and neck, lung and skin. The TP63 gene plays important roles in epidermal morphogenesis and homeostasis, regulating …
Overexpression of deltaNp63alpha, a member of the p53/p63/p73 family of transcription factors, is a molecular attribute of human squamous ca …
The transcription factors c-rel and RelA control epidermal development and homeostasis in embryonic and adult skin via distinct mechanisms.
Gugasyan R, Voss A, Varigos G, Thomas T, Grumont RJ, Kaur P, Grigoriadis G, Gerondakis S. Gugasyan R, et al. Mol Cell Biol. 2004 Jul;24(13):5733-45. doi: 10.1128/MCB.24.13.5733-5745.2004. Mol Cell Biol. 2004. PMID: 15199130 Free PMC article.
Using mice lacking RelA and c-rel, which survive throughout embryogenesis on a tumor necrosis factor alpha (TNF-alpha)-deficient background (rela(-/-) c-rel(-/-) tnfalpha(-/-)), we show that c-rel and RelA are required for normal epidermal development. …
Using mice lacking RelA and c-rel, which survive throughout embryogenesis on a tumor necrosis factor alpha (TNF-alpha)-deficient back …
Expression of NF-kappaB in epidermis and the relationship between NF-kappaB activation and inhibition of keratinocyte growth.
Takao J, Yudate T, Das A, Shikano S, Bonkobara M, Ariizumi K, Cruz PD. Takao J, et al. Br J Dermatol. 2003 Apr;148(4):680-8. doi: 10.1046/j.1365-2133.2003.05285.x. Br J Dermatol. 2003. PMID: 12752124
The involvement of NF-kappaB was assessed using sodium salicylate, which inhibits NF-kappaB activation. RESULTS: The NF-kappaB subunits, p50, p65, RelB, and c-Rel (but not p52), were detected in keratinocytes and in normal epidermis at mRNA and protein levels …
The involvement of NF-kappaB was assessed using sodium salicylate, which inhibits NF-kappaB activation. RESULTS: The NF-kappaB subunits, p50 …
Human Kruppel-like factor5/KLF5: synergy with NF-kappaB/Rel factors and expression in human skin and hair follicles.
Sur I, Undén AB, Toftgård R. Sur I, et al. Eur J Cell Biol. 2002 Jun;81(6):323-34. doi: 10.1078/0171-9335-00257. Eur J Cell Biol. 2002. PMID: 12113473 Free article.
Overexpression of KLF5 leads to an increase in the TPA response from VLTRE, a TPA-inducible enhancer element that shows keratinocyte specificity with respect to Rel/NF-kappaB binding. The KLF5-mediated transcriptional increase is not observed in the presence of over …
Overexpression of KLF5 leads to an increase in the TPA response from VLTRE, a TPA-inducible enhancer element that shows keratinocyte
Persistent IKKα phosphorylation induced apoptosis in UVB and Poly I:C co-treated HaCaT cells plausibly through pro-apoptotic p73 and abrogation of IκBα.
Otkur W, Wang F, Liu W, Hayashi T, Tashiro SI, Onodera S, Ikejima T. Otkur W, et al. Mol Immunol. 2018 Dec;104:69-78. doi: 10.1016/j.molimm.2018.10.007. Epub 2018 Nov 13. Mol Immunol. 2018. PMID: 30445257
In this study, we silenced main transcriptional factors in NF-kappaB family, RelA, RelB and c-Rel, but to our surprise the results show that none of them participate in apoptosis induction in UVB-Poly I:C-treated HaCaT cells. ...The findings partially clarify the possible …
In this study, we silenced main transcriptional factors in NF-kappaB family, RelA, RelB and c-Rel, but to our surprise the results sh …
Squamous cell carcinomas and increased apoptosis in skin with inhibited Rel/nuclear factor-kappaB signaling.
van Hogerlinden M, Rozell BL, Ahrlund-Richter L, Toftgård R. van Hogerlinden M, et al. Cancer Res. 1999 Jul 15;59(14):3299-303. Cancer Res. 1999. PMID: 10416581 Free article.
The Rel/nuclear factor-kappaB (Rel/NF-kappaB) transcription factors have been implicated previously in control of apoptosis, cell proliferation, and oncogenesis. ...Presence of hyperplasia and hair follicle degeneration demonstrate an important role for Rel/N …
The Rel/nuclear factor-kappaB (Rel/NF-kappaB) transcription factors have been implicated previously in control of apoptosis, c …
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