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Fetal cord blood's potential for bone marrow transplantation.
Ende N, Rameshwar P, Ende M. Ende N, et al. Life Sci. 1989;44(25):1987-90. doi: 10.1016/0024-3205(89)90412-8. Life Sci. 1989. PMID: 2661947
Substance P (SP) mediates production of stem cell factor and interleukin-1 in bone marrow stroma: potential autoregulatory role for these cytokines in SP receptor expression and induction.
Rameshwar P, Gascón P. Rameshwar P, et al. Blood. 1995 Jul 15;86(2):482-90. Blood. 1995. PMID: 7541664
Substance P (SP) is a neuropeptide widely distributed in the nervous system. Its release within the bone marrow (BM) can mediate bidirectional neurohematopoietic communication via specific receptors: neurokinin-1R (NK-1R), NK-2R, or NK-3R. ...
Substance P (SP) is a neuropeptide widely distributed in the nervous system. Its release within the bone marrow (BM) can mediate bidi …
Hemorrhagic shock abolishes the myelopoietic response to turpentine-induced soft tissue injury.
Raff G, Livingston DH, Wang MT, Rameshwar P. Raff G, et al. J Surg Res. 1995 Jul;59(1):75-9. doi: 10.1006/jsre.1995.1134. J Surg Res. 1995. PMID: 7630140
TURP induced a significant increase in CFU-GM on Day 1 compared to that in control (47 +/- 22 vs 21 +/- 11; P < 0.05). SHOCK completely abolished this response to TURP. ...

TURP induced a significant increase in CFU-GM on Day 1 compared to that in control (47 +/- 22 vs 21 +/- 11; P < 0.05). SHOCK compl

In vitro stimulatory effect of substance P on hematopoiesis.
Rameshwar P, Ganea D, Gascón P. Rameshwar P, et al. Blood. 1993 Jan 15;81(2):391-8. Blood. 1993. PMID: 7678516
The neuropeptide Substance P (SP) is widely distributed in the peripheral nervous system. Its biologic effects have been extensively studied in the immune system. ...
The neuropeptide Substance P (SP) is widely distributed in the peripheral nervous system. Its biologic effects have been extensively …
Induction of negative hematopoietic regulators by neurokinin-A in bone marrow stroma.
Rameshwar P, Gascón P. Rameshwar P, et al. Blood. 1996 Jul 1;88(1):98-106. Blood. 1996. PMID: 8704207
Substance P (SP) and neurokinin-A (NK-A) are derived from the preprotachykinin l gene. Although SP and NK-A can bind to either NK-1, NK-2, or NK-3 receptors (R), they have preferences for NK-1R and NK-2R, respectively. ...
Substance P (SP) and neurokinin-A (NK-A) are derived from the preprotachykinin l gene. Although SP and NK-A can bind to either NK-1, …
Receptor induction regulates the synergistic effects of substance P with IL-1 and platelet-derived growth factor on the proliferation of bone marrow fibroblasts.
Rameshwar P, Poddar A, Zhu G, Gascón P. Rameshwar P, et al. J Immunol. 1997 Apr 1;158(7):3417-24. J Immunol. 1997. PMID: 9120302
The neuropeptide substance P (SP) stimulates CFU in bone marrow (BM) cultures. Although the methylcellulose matrix used in these assays does not provide an appropriate substratum to support adherent-dependent cells, we have observed that cultures containing optimal SP (10( …
The neuropeptide substance P (SP) stimulates CFU in bone marrow (BM) cultures. Although the methylcellulose matrix used in these assa …
Hematopoietic regulation mediated by interactions among the neurokinins and cytokines.
Rameshwar P, Poddar A, Gascón P. Rameshwar P, et al. Leuk Lymphoma. 1997 Dec;28(1-2):1-10. doi: 10.3109/10428199709058325. Leuk Lymphoma. 1997. PMID: 9498698 Review.
This review summarizes the current data regarding the mechanisms by which two mammalian neurokinins (tachykinins), substance P (SP) and neurokinin-A (NK-A) are involved in hematopoiesis. ...
This review summarizes the current data regarding the mechanisms by which two mammalian neurokinins (tachykinins), substance P (SP) a …
Systemic transforming growth factor-beta in patients with bone marrow fibrosis--pathophysiological implications.
Rameshwar P, Chang VT, Thacker UF, Gascón P. Rameshwar P, et al. Am J Hematol. 1998 Oct;59(2):133-42. doi: 10.1002/(sici)1096-8652(199810)59:2<133::aid-ajh6>3.0.co;2-z. Am J Hematol. 1998. PMID: 9766798
Compared with nonfibrosis sera, there was significant TGF-beta elevation in BM fibrosis sera (P < 0.0001). Most (>80%) of the TGF-beta is active and belongs to the-beta1 isoform. ...

Compared with nonfibrosis sera, there was significant TGF-beta elevation in BM fibrosis sera (P < 0.0001). Most (>80%) of the T

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