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Genetic alterations in sporadic and Crohn's-associated adenocarcinomas of the small intestine.
Rashid A, Hamilton SR. Rashid A, et al. Gastroenterology. 1997 Jul;113(1):127-35. doi: 10.1016/s0016-5085(97)70087-8. Gastroenterology. 1997. PMID: 9207270
CONCLUSIONS: Accumulation of ras and p53 alterations occurs during the adenoma/dysplasia-carcinoma sequence in small intestinal carcinogenesis, but a ras-independent pathway may also exist. ...
CONCLUSIONS: Accumulation of ras and p53 alterations occurs during the adenoma/dysplasia-carcinoma sequence in small intestinal carcinogenes …
Genetic alterations in hepatocellular carcinomas: association between loss of chromosome 4q and p53 gene mutations.
Rashid A, Wang JS, Qian GS, Lu BX, Hamilton SR, Groopman JD. Rashid A, et al. Br J Cancer. 1999 Apr;80(1-2):59-66. doi: 10.1038/sj.bjc.6690321. Br J Cancer. 1999. PMID: 10389978 Free PMC article.
All seven HCCs with a p53 mutation from Qidong and three of five from Shanghai had the aflatoxin-associated point mutation with a G to T transversion at codon 249, position 3. ...These data indicate a possible interaction between p53 gene mutation and 4q loss …
All seven HCCs with a p53 mutation from Qidong and three of five from Shanghai had the aflatoxin-associated point mutation with a
Phenotypic and molecular characteristics of hyperplastic polyposis.
Rashid A, Houlihan PS, Booker S, Petersen GM, Giardiello FM, Hamilton SR. Rashid A, et al. Gastroenterology. 2000 Aug;119(2):323-32. doi: 10.1053/gast.2000.9361. Gastroenterology. 2000. PMID: 10930367
Patients with a large HP differed from those with multiple HPs in having a high frequency of right-sided HP (63% vs. 22%, P = 0.01) and of right-sided colon cancer (100% vs. 8%, P = 0.003). Hyperplastic polyposis was associated with a family history of colore …
Patients with a large HP differed from those with multiple HPs in having a high frequency of right-sided HP (63% vs. 22%, P = …
Genetic epidemiology of mutated K-ras proto-oncogene, altered suppressor genes, and microsatellite instability in colorectal adenomas.
Rashid A, Zahurak M, Goodman SN, Hamilton SR. Rashid A, et al. Gut. 1999 Jun;44(6):826-33. doi: 10.1136/gut.44.6.826. Gut. 1999. PMID: 10323885 Free PMC article.
Patients 65 years of age and older had a decreased probability of K-ras mutations (26% versus 45%). Overexpression of p53 gene product was present in only 6% of adenomas but was more frequent in villous or tubulovillous adenomas (19% versus 3%). ...RER positive adenomas we …
Patients 65 years of age and older had a decreased probability of K-ras mutations (26% versus 45%). Overexpression of p53 gene produc …
Accumulated clonal genetic alterations in familial and sporadic colorectal carcinomas with widespread instability in microsatellite sequences.
Fujiwara T, Stolker JM, Watanabe T, Rashid A, Longo P, Eshleman JR, Booker S, Lynch HT, Jass JR, Green JS, Kim H, Jen J, Vogelstein B, Hamilton SR. Fujiwara T, et al. Among authors: rashid a. Am J Pathol. 1998 Oct;153(4):1063-78. doi: 10.1016/S0002-9440(10)65651-9. Am J Pathol. 1998. PMID: 9777938 Free PMC article.
A subset of hereditary and sporadic colorectal carcinomas is defined by microsatellite instability (MSI), but the spectra of gene mutations have not been characterized extensively. ...
A subset of hereditary and sporadic colorectal carcinomas is defined by microsatellite instability (MSI), but the spectra of gene mut
Dysplasia and dysregulation of proliferation in foveolar and surface epithelia of fundic gland polyps from patients with familial adenomatous polyposis.
Wu TT, Kornacki S, Rashid A, Yardley JH, Hamilton SR. Wu TT, et al. Among authors: rashid a. Am J Surg Pathol. 1998 Mar;22(3):293-8. doi: 10.1097/00000478-199803000-00003. Am J Surg Pathol. 1998. PMID: 9500770
Genetic alterations and epithelial dysplasia in juvenile polyposis syndrome and sporadic juvenile polyps.
Wu TT, Rezai B, Rashid A, Luce MC, Cayouette MC, Kim C, Sani N, Mishra L, Moskaluk CA, Yardley JH, Hamilton SR. Wu TT, et al. Among authors: rashid a. Am J Pathol. 1997 Mar;150(3):939-47. Am J Pathol. 1997. PMID: 9060832 Free PMC article.
Our results indicate that the multiple genetic alterations involved in usual colorectal neoplasia also play a role in neoplastic transformation of juvenile polyps, predominantly in juvenile polyposis syndrome....
Our results indicate that the multiple genetic alterations involved in usual colorectal neoplasia also play a role in neoplastic tran …
Elevated expression of fatty acid synthase and fatty acid synthetic activity in colorectal neoplasia.
Rashid A, Pizer ES, Moga M, Milgraum LZ, Zahurak M, Pasternack GR, Kuhajda FP, Hamilton SR. Rashid A, et al. Am J Pathol. 1997 Jan;150(1):201-8. Am J Pathol. 1997. PMID: 9006336 Free PMC article.
Our study shows that FAS is expressed in all colorectal neoplasms and there is a concomitant increase in fatty acid synthesis. FAS may therefore represent a potential therapeutic target....
Our study shows that FAS is expressed in all colorectal neoplasms and there is a concomitant increase in fatty acid synthesis. FAS ma …
The effects of sulindac on colorectal proliferation and apoptosis in familial adenomatous polyposis.
Pasricha PJ, Bedi A, O'Connor K, Rashid A, Akhtar AJ, Zahurak ML, Piantadosi S, Hamilton SR, Giardiello FM. Pasricha PJ, et al. Among authors: rashid a. Gastroenterology. 1995 Sep;109(3):994-8. doi: 10.1016/0016-5085(95)90411-5. Gastroenterology. 1995. PMID: 7657130 Clinical Trial.
METHODS: Cell proliferation was studied via immunohistochemistry for cell nuclear antigen in a group of 22 patients randomized to either sulindac (150 mg twice a day) or placebo in a previously published trial. ...RESULTS: Although sulindac caused a si …
METHODS: Cell proliferation was studied via immunohistochemistry for cell nuclear antigen in a group of 22 patients randomized to eit …
Hypermethylation of the hMLH1 gene promoter is associated with microsatellite instability in early human gastric neoplasia.
Fleisher AS, Esteller M, Tamura G, Rashid A, Stine OC, Yin J, Zou TT, Abraham JM, Kong D, Nishizuka S, James SP, Wilson KT, Herman JG, Meltzer SJ. Fleisher AS, et al. Among authors: rashid a. Oncogene. 2001 Jan 18;20(3):329-35. doi: 10.1038/sj.onc.1204104. Oncogene. 2001. PMID: 11313962
A significant portion of gastric cancers exhibit defective DNA mismatch repair, manifested as microsatellite instability (MSI). ...We therefore investigated hMLH1 hypermethylation, hMLH1 expression and MSI in a group of early gastric cancers and gastric adenomas. ..
A significant portion of gastric cancers exhibit defective DNA mismatch repair, manifested as microsatellite instability (MSI). ...We
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